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2001, 1, ISBN 185317890X, xv, 528
In this volume, an international team of experts in chronic myeloid leukemia share their expertise. In particular, they contribute their insights on the latest... 
Treatment | Leukemia, Myeloid | Acute myeloid leukemia | therapy | Clinical & internal medicine | Acute myelocytic leukaemia | Hematology | Oncology | Acute myelocytic leukemia
Book
1999, Basic and clinical oncology, ISBN 9780824799014, Volume 16., xi, 442
Book
2007, Contemporary hematology, ISBN 9781588296214, xiv, 426
Acute Myelogenous Leukemia is a timely compilation of new concepts in the molecular pathogenesis and molecular therapy of acute myelogenous leukemia (AML). The... 
Acute myeloid leukemia | Leukemia, Myelocytic, Acute | Clinical & internal medicine | Hematology | Oncology | Cancer Research | Internal Medicine | Medicine & Public Health
Book
2008, ISBN 3540723021, x, 294
Better therapy of acute leukemias depends ultimately on better understanding of the distinction between leukemic and normal progenitor cells. Thus, several... 
Acute leukemia -- Treatment
Book
Annals of Hematology, ISSN 0939-5555, 4/2018, Volume 97, Issue 4, pp. 597 - 604
Data are limited in developing countries regarding the clinicopathologic features and response to therapy of chronic myeloid leukemia (CML) in the era of... 
Imatinib | Survey | CML | Medicine & Public Health | Hematology | Oncology | Management | Tunisia | Clinical practice | SURVIVAL | INTERFERON | CHRONIC-PHASE | RECOMMENDATIONS | HEMATOLOGY | EUROPEAN-LEUKEMIANET | Prognosis | Follow-Up Studies | Humans | Leukemia, Myeloid, Chronic-Phase - pathology | Middle Aged | Child, Preschool | Male | Antineoplastic Agents - therapeutic use | Practice Patterns, Physicians | Protein Kinase Inhibitors - adverse effects | Splenomegaly - pathology | Leukemia, Myeloid, Accelerated Phase - epidemiology | Young Adult | Imatinib Mesylate - therapeutic use | Leukemia, Myeloid, Accelerated Phase - pathology | Antineoplastic Agents - adverse effects | Imatinib Mesylate - adverse effects | Leukemia, Myeloid, Accelerated Phase - diagnosis | Splenomegaly - etiology | Aged, 80 and over | Adult | Female | Retrospective Studies | Child | Leukemia, Myeloid, Accelerated Phase - drug therapy | Leukemia, Myeloid, Chronic-Phase - drug therapy | Splenomegaly - prevention & control | Leukemia, Myeloid, Chronic-Phase - diagnosis | Leukemia, Myeloid, Chronic-Phase - epidemiology | Tumor Burden - drug effects | Protein Kinase Inhibitors - therapeutic use | Adolescent | Survival Analysis | Aged | Tunisia - epidemiology | Antimitotic agents | Chronic myeloid leukemia | Antineoplastic agents | Epidemiology | Patient outcomes | Analysis | Multivariate analysis | Medical prognosis | Leukemia | Developing countries--LDCs | Index Medicus | Tumor Burden/drug effects | Leukemia, Myeloid, Chronic-Phase/epidemiology | Splenomegaly/prevention & control | Leukemia, Myeloid, Accelerated Phase/epidemiology | Antineoplastic Agents/therapeutic use | Splenomegaly/pathology | Life Sciences | Leukemia, Myeloid, Chronic-Phase/pathology | Antineoplastic Agents/adverse effects | Tunisia/epidemiology | Leukemia, Myeloid, Accelerated Phase/pathology | Leukemia, Myeloid, Chronic-Phase/diagnosis | Leukemia, Myeloid, Chronic-Phase/drug therapy | Splenomegaly/etiology | Human health and pathology | Protein Kinase Inhibitors/adverse effects | Leukemia, Myeloid, Accelerated Phase/drug therapy | Imatinib Mesylate/therapeutic use | Leukemia, Myeloid, Accelerated Phase/diagnosis | Protein Kinase Inhibitors/therapeutic use | Imatinib Mesylate/adverse effects
Journal Article
1975, ISBN 088416022X, 126
Book
07/2013, ISBN 1780842759
Book
The New England Journal of Medicine, ISSN 0028-4793, 06/2016, Volume 374, Issue 23, pp. 2209 - 2221
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 08/2012, Volume 367, Issue 9, pp. 805 - 816
Patients with acute myeloid leukemia who received marrow grafts from KIR2DS1-expressing donors mismatched at HLA-C had a better outcome than those with no... 
SURVIVAL | ALLOREACTIVITY | MEDICINE, GENERAL & INTERNAL | IMMUNOGLOBULIN-LIKE RECEPTORS | NK CELLS | RECOGNITION | ACUTE MYELOGENOUS LEUKEMIA | HAPLOTYPES | MYELOID-LEUKEMIA | STEM-CELL TRANSPLANTATION | KIR-LIGAND INCOMPATIBILITY | HLA-C Antigens - metabolism | Receptors, KIR - physiology | Receptors, KIR - genetics | Humans | Kaplan-Meier Estimate | Proportional Hazards Models | Hematopoietic Stem Cell Transplantation | Genotype | Secondary Prevention | Leukemia, Myeloid, Acute - prevention & control | Transplantation, Homologous | Leukemia, Myeloid, Acute - mortality | Killer Cells, Natural - immunology | Unrelated Donors | Aged | Retrospective Studies | HLA-C Antigens - genetics | Leukemia, Myeloid, Acute - therapy | Leukemia, Myeloid, Acute - genetics | Treatment outcome | Killer cells | Usage | Transplantation | Health aspects | Hematopoietic stem cells | Bone grafts | Haplotypes | Transplants & implants | Linkage disequilibrium | Stem cell transplantation | Cytotoxicity | Health Insurance Portability & Accountability Act 1996-US | Killer cell immunoglobulin-like receptors | Bone marrow | Natural killer cells | Genotypes | Immune system | Antigens | Potassium channels (inwardly-rectifying) | Myeloid leukemia | Mortality | Hemopoiesis | Heterozygosity | Studies | Hypotheses | Genotyping | Cell lines | Ligands | Histocompatibility antigen HLA | Acute myeloid leukemia | Index Medicus | Abridged Index Medicus
Journal Article
Molecular Therapy, ISSN 1525-0016, 09/2017, Volume 25, Issue 9, pp. 2202 - 2213
The successful immunotherapy of acute myeloid leukemia (AML) has been hampered because most potential antigenic targets are shared with normal hematopoietic... 
AML | CLL-1 | CAR | MEDICINE, RESEARCH & EXPERIMENTAL | STEM-CELLS | SUICIDE GENE | PERSISTENCE | BISPECIFIC ANTIBODY | THERAPY | IMMUNOTHERAPY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | IN-VIVO | EXPANSION | GENETICS & HEREDITY | SAFETY SWITCH | LYMPHOCYTES | Humans | Middle Aged | Male | Leukemia, Myeloid, Acute - immunology | Recombinant Fusion Proteins | Young Adult | T-Lymphocytes - metabolism | Myeloid Cells - immunology | Inflammation Mediators - metabolism | Aged, 80 and over | Adult | Female | Child | Leukemia, Myeloid, Acute - therapy | Disease Models, Animal | Gene Expression | Cytokines - metabolism | Immunotherapy, Adoptive | Receptors, Antigen, T-Cell - metabolism | Xenograft Model Antitumor Assays | Animals | Leukemia, Myeloid, Acute - diagnosis | Adolescent | Cell Line, Tumor | Myeloid Cells - metabolism | T-Cell Antigen Receptor Specificity | T-Lymphocytes - immunology | Aged | Mice | Receptors, Antigen, T-Cell - genetics | Lectins, C-Type - antagonists & inhibitors | Cytotoxicity, Immunologic | Leukemia, Myeloid, Acute - genetics | Leukemia | Stem cell transplantation | Cytotoxicity | Leukocytes (mononuclear) | Lymphocytes T | Suicide | Cancer therapies | Hematopoietic stem cells | Lymphocytes | Immunotherapy | Xenografts | Peripheral blood mononuclear cells | Conflicts of interest | Tumor necrosis factor-TNF | Suicide genes | Antigens | Myeloid cells | Chronic lymphatic leukemia | Cell survival | Statistical analysis | Cytokines | Colonies | Myeloid leukemia | Caspase | Patients | Progenitor cells | Hemopoiesis | Chemotherapy | Cell lines | Stem cells | Caspase-9 | Software | Acute myeloid leukemia | Index Medicus | Original
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2016, Volume 11, Issue 12, pp. e0166891 - e0166891
Background Approximately fifty percent of patients with acute myeloid leukemia can be cured with current therapeutic strategies which include, standard dose... 
GEMTUZUMAB OZOGAMICIN | PHARMACOKINETICS | IMMUNOTHERAPY | MULTIDISCIPLINARY SCIENCES | IMMUNOGLOBULIN | DEATH | ON-TARGET | CASPASE-9 | INDUCED KILLER-CELLS | SAFETY SWITCH | LYMPHOCYTES | Recombinant Fusion Proteins - immunology | Cyclophosphamide - analogs & derivatives | Caspase 9 - genetics | Humans | Sialic Acid Binding Ig-like Lectin 3 - immunology | Leukemia, Myeloid, Acute - immunology | Cellular Reprogramming | Biphenyl Compounds - pharmacology | Lysosomal-Associated Membrane Protein 1 - genetics | Nitrophenols - pharmacology | Myeloid Cells - immunology | T-Lymphocytes - drug effects | Lysosomal-Associated Membrane Protein 1 - immunology | Tumor Necrosis Factor-alpha - immunology | Myeloid Cells - drug effects | Receptors, Antigen, T-Cell - immunology | Leukemia, Myeloid, Acute - therapy | Sialic Acid Binding Ig-like Lectin 3 - genetics | Leukemia, Myeloid, Acute - pathology | Clinical Trials as Topic | Sulfonamides - pharmacology | Piperazines - pharmacology | Caspase 9 - immunology | Bridged Bicyclo Compounds, Heterocyclic - pharmacology | T-Lymphocytes - cytology | B7-H1 Antigen - pharmacology | Cyclophosphamide - pharmacology | Interferon-gamma - immunology | Sialic Acid Binding Ig-like Lectin 3 - antagonists & inhibitors | Cell Line, Tumor | Recombinant Fusion Proteins - genetics | Cell Engineering | T-Lymphocytes - immunology | Cell Proliferation - drug effects | Receptors, Antigen, T-Cell - genetics | Myeloid Cells - pathology | Primary Cell Culture | Genetic Vectors | Tumor Necrosis Factor-alpha - biosynthesis | Interferon-gamma - biosynthesis | Cytotoxicity, Immunologic | Leukemia, Myeloid, Acute - genetics | Usage | Transplantation | Research | T cells | Immunotherapy | Stem cells | Transplants & implants | Bcl-2 protein | Leukemia | Mental health | Oncology | Standard error | Lymphocytes T | Suicide | Hematopoietic stem cells | Infusion | Immunology | Granulocytes | Lymphocytes | Drug dosages | Genetic modification | Suicide genes | Antigens | Cytokines | Risk groups | Hematology | Myeloid leukemia | Safety measures | Risk reduction | T cell receptors | Tumor necrosis factor-α | Patients | Hemopoiesis | Studies | Chemotherapy | Inhibitors | γ-Interferon | Degranulation | Antitumor activity | Ligands | Interferon | Gene therapy | Acute myeloid leukemia | Cancer | Apoptosis | Index Medicus
Journal Article