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Cellular and Molecular Life Sciences, ISSN 1420-682X, 2/2014, Volume 71, Issue 4, pp. 549 - 574
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 01/2018, Volume 128, Issue 1, pp. 97 - 107
Journal Article
Journal Article
Circulation Research, ISSN 0009-7330, 06/2010, Volume 106, Issue 11, pp. 1675 - 1680
Fibrosis is one of the largest groups of diseases for which there is no therapy but is believed to occur because of a persistent tissue repair program. During... 
TGF | Endothelin | Scarring | PDGF | Fibrosis | CARDIAC & CARDIOVASCULAR SYSTEMS | scarring | GENE-REGULATION | FIBROTIC PHENOTYPE | MATRIX CONTRACTION | endothelin | GROWTH-FACTOR-BETA | MYOCARDIAL FIBROSIS | IN-VIVO | PERIPHERAL VASCULAR DISEASE | fibrosis | SMOOTH-MUSCLE-CELLS | DERMAL FIBROBLASTS | TGF beta | MICE LACKING SMAD3 | HEMATOLOGY | MYOFIBROBLAST DIFFERENTIATION
Journal Article
Chest, ISSN 0012-3692, 05/2017, Volume 151, Issue 5, pp. 971 - 981
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 10/2017, Volume 127, Issue 10, pp. 3770 - 3783
The master cytokine TGF-beta mediates tissue fibrosis associated with inflammation and tissue injury. TGF-beta induces fibroblast activation and... 
GENETIC MANIPULATION | MEDICINE, RESEARCH & EXPERIMENTAL | HYPERTROPHY | GROWTH-FACTOR-BETA | HEART-DISEASE | TGF-BETA | SMAD3 | MYOCARDIAL-INFARCTION | IN-VIVO | MYOFIBROBLAST DIFFERENTIATION | PROTECTS | Receptors, Transforming Growth Factor beta - genetics | Heart Diseases - metabolism | Male | Smad3 Protein - metabolism | Myofibroblasts - metabolism | Smad3 Protein - genetics | Gene Deletion | Myocardium - metabolism | Smad2 Protein - genetics | Protein-Serine-Threonine Kinases - metabolism | Myofibroblasts - pathology | Signal Transduction | Protein-Serine-Threonine Kinases - genetics | Smad2 Protein - metabolism | Mice, Transgenic | Myocardium - pathology | Organ Specificity | Myocytes, Cardiac - pathology | Animals | Transforming Growth Factor beta - genetics | Receptors, Transforming Growth Factor beta - metabolism | Fibrosis | Myocytes, Cardiac - metabolism | Mice | Transforming Growth Factor beta - metabolism | Heart Diseases - genetics | Heart Diseases - pathology | Transcription factors | Phosphorylation | Heart attacks | Disease | Homeostasis | Smad3 protein | Gene deletion | Kinases | Proteins | Clonal deletion | Smad2 protein | Rodents | Fibroblasts | Extracellular matrix | Heart diseases | Growth factors | Heart failure | Cytokines | Cardiomyocytes | Gene expression | Pressure | Latency | Adenoviruses | Genetic engineering | Apoptosis
Journal Article
Journal Article