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Biochimie, ISSN 0300-9084, 12/2018, Volume 155, pp. 67 - 82
Exosome-mediated intercellular communication has been increasingly recognized as having a broad impact on Alzheimer's disease (AD) pathogenesis. Still, limited... 
Microglia activation | Neuron-microglia communication | Neuroinflammation | Inflammatory-associated miRNAs | Exosomes | Alzheimer's disease
Journal Article
BIOCHIMIE, ISSN 0300-9084, 12/2018, Volume 155, pp. 67 - 82
Exosome-mediated intercellular communication has been increasingly recognized as having a broad impact on Alzheimer's disease (AD) pathogenesis. Still, limited... 
CLEARANCE | ALZHEIMERS-DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | Neuroinflammation | NECROSIS-FACTOR-ALPHA | AMYLOID PRECURSOR PROTEIN | Exosomes | Microglia activation | Neuron-microglia communication | Inflammatory-associated miRNAs | NEURONAL FUNCTION | INFLAMMATION | A-BETA | ACCUMULATION | Alzheimer's disease | EXPRESSION | BRAIN
Journal Article
Frontiers in cellular neuroscience, ISSN 1662-5102, 2018, Volume 12, p. 136
It is becoming evident that microglia, the resident immune cells of the central nervous system (CNS), are active contributors in neurological disorders.... 
Journal Article
Journal Article
Journal of Neuroinflammation, ISSN 1742-2094, 07/2012, Volume 9, Issue 1, pp. 165 - 165
Background: In physiological conditions, it is postulated that neurons control microglial reactivity through a series of inhibitory mechanisms, involving... 
C/EBPβ | Neuron-microglia communication | Reactive microglia | Neuroinflammation | CD200R1 | In vitro | MULTIPLE-SCLEROSIS LESIONS | TRANSCRIPTIONAL ACTIVATION | C/EBP beta | NECROSIS-FACTOR-ALPHA | NEUROSCIENCES | BINDING-PROTEIN-BETA | IN-VITRO | INTERFERON-GAMMA | GENE-EXPRESSION | CENTRAL-NERVOUS-SYSTEM | INFLAMMATORY CYTOKINES | NF-KAPPA-B | Microglia - metabolism | CCAAT-Enhancer-Binding Protein-beta | Coculture Techniques | Mice, Inbred C57BL | Antigens, Surface - biosynthesis | Antigens, Surface - genetics | Cells, Cultured | Gene Expression Regulation | Mice, 129 Strain | Orexin Receptors | Receptors, Cell Surface - antagonists & inhibitors | Mice, Knockout | Animals | CCAAT-Enhancer-Binding Proteins - biosynthesis | Mice | Receptors, Cell Surface - biosynthesis | CCAAT-Enhancer-Binding Proteins - genetics | Protein Binding - physiology | Receptors, Cell Surface - genetics | Animal experimentation | Chromatin | Nervous system diseases | RNA | Neurons | Gene expression | Mitogens | Protein binding | Studies | Polymerase chain reaction | Flow cytometry | Genotype & phenotype | Ischemia | Neurodegeneration | Rodents | Regulation | Laboratory animals | Experiments | Binding sites | Manuscripts | Neurosciences | Nerve tissue | Neurobiologia | Cultiu de teixits | Neuroglia | Neurociències | Neurobiology | Tissue culture | Teixit nerviós | Neuròglia
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 10/2008, Volume 107, Issue 1, pp. 73 - 85
Glial cells and neurons are in constant reciprocal signalling both under physiological and neuropathological conditions. Microglial activation is often... 
mitogen‐activated protein kinases | fibroblast growth factor‐2 | neuron‐microglia crosstalk | fibroblast growth factor receptor | cathepsins | neuroprotection | heat‐shock protein induction | Neuron-microglia crosstalk | Neuroprotection | Heat-shock protein induction | Cathepsins | Fibroblast growth factor receptor | Fibroblast growth factor-2 | Mitogen-activated protein kinases | CEREBELLAR GRANULE NEURONS | KYNURENINE PATHWAY | PROTEIN-KINASE | ALZHEIMERS-DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | fibroblast growth factor-2 | neuro-protection | FIBROBLAST-GROWTH-FACTOR | NEUROSCIENCES | neuron-microglia crosstalk | heat-shock protein induction | TUMOR-NECROSIS-FACTOR | QUINOLINIC ACID | LYSOSOMAL MEMBRANE PERMEABILIZATION | mitogen-activated protein kinases | RAT-BRAIN | OXIDATIVE TOXICITY | Brain Diseases - metabolism | Microglia - metabolism | Nerve Degeneration - physiopathology | Cell Communication - physiology | JNK Mitogen-Activated Protein Kinases - metabolism | Cytoprotection - physiology | Extracellular Signal-Regulated MAP Kinases - metabolism | Brain Diseases - physiopathology | Nerve Degeneration - metabolism | Up-Regulation - physiology | Fibroblast Growth Factor 2 - metabolism | Neurons - metabolism | Quinolinic Acid - toxicity | Neurons - drug effects | Organ Culture Techniques | Calcium-Binding Proteins - metabolism | Microglia - drug effects | Mice, Inbred C57BL | Cells, Cultured | Neurotoxins - toxicity | HSP70 Heat-Shock Proteins - metabolism | Animals | Cell Death - physiology | Cathepsins - metabolism | Quinolinic Acid - metabolism | Neurotoxins - metabolism | Signal Transduction - physiology | Mice | Mice, Inbred BALB C | Physiological aspects | Neurosciences | Fibroblast growth factors | Neurons | Neurology | Signal transduction | Biochemistry | Cellular biology | Transgenic animals | Rodents | Index Medicus
Journal Article
Journal Article
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