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Oncogene, ISSN 0950-9232, 04/2004, Volume 23, Issue 16, pp. 2881 - 2890
Journal Article
2005, 1. Aufl., Neuroscience intelligence unit, ISBN 9780306478505, 102
Cell-Cycle Mechanisms and Neuronal Cell Death examines the role of cell cycle activation in the molecular mechanisms leading to neuronal degeneration. Leading... 
Nervous system | Surgery | Degeneration | Pathophysiology | Neurons | Cell cycle | Apoptosis | Neurosciences | Biomedicine | Pharmacology/Toxicology | Neurobiology | Cell Biology
Book
Nature, ISSN 0028-0836, 11/2012, Volume 491, Issue 7422, pp. 109 - 113
Journal Article
Advances in experimental medicine and biology, ISSN 0065-2598, 2018, Volume 1091, pp. 129 - 160
Aluminum (Al), an abundant element in the earth's crust, is well-known for its neurotoxicity. Nonetheless, its causal role in neurodegenerative diseases,... 
Aluminum | Cell death | Necroptosis | Neural cells | Autophagy | Apoptosis | INDUCED NECROPTOSIS | INDUCED APOPTOSIS | ALZHEIMERS-DISEASE | CANCER DRUG-RESISTANCE | TAU | NEURODEGENERATION | NEUROSCIENCES | DAMAGE | CHRONIC EXPOSURE | NECROSIS | CULTURED ASTROCYTES | TOXICOLOGY | Animals | Neurons | Cell Death | Rats | Mice | Necrosis
Journal Article
Nature Medicine, ISSN 1078-8956, 10/2016, Volume 22, Issue 10, pp. 1101 - 1107
Journal Article
Journal Article
BRAIN PATHOLOGY, ISSN 1015-6305, 04/2004, Volume 14, Issue 2, pp. 215 - 222
Neuronal and glial cell death and traumatic axonal injury contribute to the overall pathology of traumatic brain injury (TBI) in both humans and animals. In... 
DIFFUSE AXONAL INJURY | CORTICAL IMPACT INJURY | FOCAL CEREBRAL-ISCHEMIA | TUMOR-SUPPRESSOR GENE | SELECTIVE NEURONAL DEATH | PATHOLOGY | NEUROSCIENCES | CLINICAL NEUROLOGY | PROTEIN-KINASE INHIBITION | SPINAL-CORD-INJURY | HUMAN HEAD-INJURY | CENTRAL-NERVOUS-SYSTEM | CYTOCHROME-C RELEASE
Journal Article
Autophagy, ISSN 1554-8627, 12/2014, Volume 10, Issue 12, pp. 2208 - 2222
Dysregulation of autophagy contributes to neuronal cell death in several neurodegenerative and lysosomal storage diseases. Markers of autophagy are also... 
β; AIF1/IBA1, allograft inflammatory factor 1 | SPTAN1, spectrin, α, non-erythrocytic 1 | LAMP1, lysosomal-associated membrane protein 1 | LC3, microtubule associated protein 1 light chain 3 | ACTB, actin | APC, adenomatous polyposis coli | ATG7, autophagy-related 7 | ULK1, unc-51 like autophagy activating kinase 1 | AIFM1, apoptosis-inducing factor, mitochondrion-associated, 1 | ATG5, autophagy-related 5 | autophagy flux | CASP3, caspase 3 | GFP, green fluorescent protein | autophagy | traumatic brain injury | CSPG4, chondroitin sulfate proteoglycan 4 | CAPS12, caspase 12 | neuronal cell death | ATG12, autophagy-related 12 | SQSTM1, sequestosome 1 | CD68, CD68 molecule | CCI, controlled cortical impact | RBFOX3, RNA binding protein, fox-1 homolog (C. elegans) 3 | LAMP2, lysosomal-associated membrane protein 2 | TBI, traumatic brain injury | lysosome | CTSD, cathepsin D | Traumatic brain injury | Autophagy | Neuronal cell death | Autophagy flux | Lysosome | NEURODEGENERATIVE DISEASE | UBIQUITIN | LYSOSOMAL STORAGE DISORDERS | CEREBRAL-CORTEX | RAT MODEL | CELL BIOLOGY | PATHWAY | DEGRADATION | MICE | EXPRESSION | PARKINSONS-DISEASE | Sequestosome-1 Protein | Heat-Shock Proteins - metabolism | Mice, Inbred C57BL | Phagosomes - metabolism | Neurons - cytology | Autophagy - physiology | Brain Injuries - metabolism | Apoptosis Regulatory Proteins - metabolism | Brain - metabolism | Animals | Cell Death - physiology | Neurons - metabolism | Adaptor Proteins, Signal Transducing - metabolism | Brain Injuries - pathology | Disease Models, Animal | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2018, Volume 13, Issue 5, pp. e0196387 - e0196387
Statins inhibit HMG-CoA reductase, the rate-limiting enzyme in the cholesterol biosynthesis pathway (CBP), and are used for the prevention of cardiovascular... 
Male | Mouse Embryonic Stem Cells - cytology | Neural Stem Cells - cytology | RNA, Messenger - metabolism | Simvastatin - pharmacology | Autophagy - drug effects | Mouse Embryonic Stem Cells - drug effects | Mouse Embryonic Stem Cells - metabolism | Female | Sterol Regulatory Element Binding Protein 2 - genetics | Cell Death - drug effects | Pravastatin - pharmacology | Cell Survival - drug effects | RNA, Messenger - genetics | Cells, Cultured | Neural Stem Cells - drug effects | Transcriptome - drug effects | Animals | Biosynthetic Pathways - drug effects | Hydroxymethylglutaryl-CoA Reductase Inhibitors - pharmacology | Cell Differentiation - drug effects | Mice | Polyisoprenyl Phosphates - pharmacology | Sesquiterpenes - pharmacology | Cell Cycle - drug effects | Cholesterol - biosynthesis | Neural Stem Cells - metabolism | Pharmacology, Experimental | Stem cells | Physiological aspects | Dosage and administration | Developmental neurology | Research | Pharmacokinetics | Health aspects | Statins | Cholesterol | Cell proliferation | Brain | Medical services | Clinical trials | Activation | Genomes | Biosynthesis | Pravastatin | Autophagy | Cell fate | Transcription activation | Cell cycle | G1 phase | Medical research | Cell survival | Simvastatin | Cortex | Poly(ADP-ribose) polymerase | Pre-eclampsia | Gene expression | Survival | Progenitor cells | Pregnancy | Cell death | Cells (biology) | Neural stem cells | Hydroxymethylglutaryl-CoA reductase | Cardiovascular diseases | Differentiation | Phagocytosis | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2006, Volume 103, Issue 48, pp. 18314 - 18319
Apoptosis-inducing factor (AIF), a mitochondrial oxidoreductase, is released into the cytoplasm to induce cell death in response to poly(ADP-ribose) (PAR)... 
Mitochondria | Fractionation | Neurons | Cell death | N methyl D aspartate receptors | Antibodies | HeLa cells | Histones | Polymers | Condensation | Poly(ADP-ribose) polymerase | Excitotoxicity | TRANSLOCATION | polymerase | MULTIDISCIPLINARY SCIENCES | FOCAL CEREBRAL-ISCHEMIA | MITOCHONDRIA | NEURONAL CULTURES | NEUROTOXICITY | OXYGEN-GLUCOSE DEPRIVATION | IN-VITRO | NMDA