Catalogue
Articles
RSS feedX
Search Filters
Format
Subjects
Library Location
Language
Publication Date2014, 2014, ISBN 3319037285, vi, 150 pages
"The expression "near-death experience" is associated in the popular understanding with access to knowledge about our transition between the states of life and...
Death | Religion and Psychology | Neuropsychology | Near-death experiences | methods | Psychological aspects | Neurosciences | Religious Studies | Psychology | Popular Science in Psychology | Neurobiology
Death | Religion and Psychology | Neuropsychology | Near-death experiences | methods | Psychological aspects | Neurosciences | Religious Studies | Psychology | Popular Science in Psychology | Neurobiology
Book
Details 
The FEBS Journal, ISSN 1742-464X, 01/2010, Volume 277, Issue 1, pp. 22 - 29
Extracellular signal‐regulated kinase (ERK) is a versatile protein kinase that regulates many cellular functions. Growing evidence suggests that ERK1/2 plays a...
sustained ERK | cell survival | neurodegenerative disease | Akt/PKB | neuronal death | apoptosis | transient ERK | caspase | ERK | ischemia | Sustained ERK | Cell survival | Ischemia | Caspase | Neurodegenerative disease | Neuronal death | Transient ERK | Apoptosis | ACTIVATED PROTEIN-KINASE | INDUCED APOPTOSIS | PKB | MAP KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | FOCAL CEREBRAL-ISCHEMIA | Akt | PLASMA-MEMBRANE | NERVE GROWTH-FACTOR | PERSISTENT ACTIVATION | CORTICAL-NEURONS | SIGNAL-REGULATED KINASE | OXIDATIVE TOXICITY | Neurons - pathology | Nerve Degeneration - enzymology | Neurodegenerative Diseases - pathology | Mitogen-Activated Protein Kinase 1 - physiology | Neurodegenerative Diseases - etiology | Humans | Ischemia - enzymology | Mitogen-Activated Protein Kinase 3 - physiology | Nerve Degeneration - pathology | MAP Kinase Signaling System | Animals | Cell Death - physiology | Neurons - enzymology | Models, Neurological | Extracellular Signal-Regulated MAP Kinases - physiology | Nerve Degeneration - etiology | Ischemia - pathology | Neurodegenerative Diseases - enzymology | Nervous system diseases | Neurosciences | Biochemistry | Cell death | Neurons | Signal transduction | Cellular biology | Kinases
sustained ERK | cell survival | neurodegenerative disease | Akt/PKB | neuronal death | apoptosis | transient ERK | caspase | ERK | ischemia | Sustained ERK | Cell survival | Ischemia | Caspase | Neurodegenerative disease | Neuronal death | Transient ERK | Apoptosis | ACTIVATED PROTEIN-KINASE | INDUCED APOPTOSIS | PKB | MAP KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | FOCAL CEREBRAL-ISCHEMIA | Akt | PLASMA-MEMBRANE | NERVE GROWTH-FACTOR | PERSISTENT ACTIVATION | CORTICAL-NEURONS | SIGNAL-REGULATED KINASE | OXIDATIVE TOXICITY | Neurons - pathology | Nerve Degeneration - enzymology | Neurodegenerative Diseases - pathology | Mitogen-Activated Protein Kinase 1 - physiology | Neurodegenerative Diseases - etiology | Humans | Ischemia - enzymology | Mitogen-Activated Protein Kinase 3 - physiology | Nerve Degeneration - pathology | MAP Kinase Signaling System | Animals | Cell Death - physiology | Neurons - enzymology | Models, Neurological | Extracellular Signal-Regulated MAP Kinases - physiology | Nerve Degeneration - etiology | Ischemia - pathology | Neurodegenerative Diseases - enzymology | Nervous system diseases | Neurosciences | Biochemistry | Cell death | Neurons | Signal transduction | Cellular biology | Kinases
Journal Article
Details
Digital rights
Loading RightsMolecular and Cellular Neuroscience, ISSN 1044-7431, 09/2015, Volume 68, pp. 203 - 211
Astrocytes, the main element of the homeostatic system in the brain, are affected in various neurological conditions including Alzheimer's disease (AD). A...
Astrogliosis | Glia | Alzheimer's disease | Apoptosis | OXIDATIVE STRESS | ACTIVATION | APOPTOTIC CELL-DEATH | ALZHEIMERS-DISEASE | FOXO TRANSCRIPTION FACTORS | MILD COGNITIVE IMPAIRMENT | NEUROSCIENCES | MATTER LESIONS | NEURONAL DEATH | A-BETA | CULTURED ASTROCYTES | RNA, Small Interfering - genetics | Amyloid beta-Peptides - pharmacology | Caspase 3 - metabolism | Carbocyanines - metabolism | Ki-67 Antigen - metabolism | Male | Green Fluorescent Proteins - genetics | Membrane Potential, Mitochondrial - drug effects | Dose-Response Relationship, Drug | Transfection | Bcl-2-Like Protein 11 | Forkhead Transcription Factors - metabolism | Membrane Proteins - metabolism | Cell Death - drug effects | Animals, Newborn | Proto-Oncogene Proteins - metabolism | Astrocytes - drug effects | Green Fluorescent Proteins - metabolism | Cells, Cultured | Rats | Rats, Sprague-Dawley | Apoptosis Regulatory Proteins - metabolism | Animals | Signal Transduction - drug effects | Benzimidazoles - metabolism | Forkhead Box Protein O3 | RNA, Small Interfering - metabolism
Astrogliosis | Glia | Alzheimer's disease | Apoptosis | OXIDATIVE STRESS | ACTIVATION | APOPTOTIC CELL-DEATH | ALZHEIMERS-DISEASE | FOXO TRANSCRIPTION FACTORS | MILD COGNITIVE IMPAIRMENT | NEUROSCIENCES | MATTER LESIONS | NEURONAL DEATH | A-BETA | CULTURED ASTROCYTES | RNA, Small Interfering - genetics | Amyloid beta-Peptides - pharmacology | Caspase 3 - metabolism | Carbocyanines - metabolism | Ki-67 Antigen - metabolism | Male | Green Fluorescent Proteins - genetics | Membrane Potential, Mitochondrial - drug effects | Dose-Response Relationship, Drug | Transfection | Bcl-2-Like Protein 11 | Forkhead Transcription Factors - metabolism | Membrane Proteins - metabolism | Cell Death - drug effects | Animals, Newborn | Proto-Oncogene Proteins - metabolism | Astrocytes - drug effects | Green Fluorescent Proteins - metabolism | Cells, Cultured | Rats | Rats, Sprague-Dawley | Apoptosis Regulatory Proteins - metabolism | Animals | Signal Transduction - drug effects | Benzimidazoles - metabolism | Forkhead Box Protein O3 | RNA, Small Interfering - metabolism
Journal Article
Details
Digital rights
Loading RightsInternational Journal of Developmental Neuroscience, ISSN 0736-5748, 10/2015, Volume 45, pp. 75 - 85
Neonatal hypoxic-ischemic encephalopathy is a critical cerebral event occurring around birth with high mortality and neurological morbidity associated with...
Immature brain | Autophagy | Neuronal cell death | Hypoxia-ischemia | BECLIN 1 | BRAIN-INJURY | ACID-INDUCED EXCITOTOXICITY | N-TERMINAL KINASE | KAINIC ACID | DEVELOPMENTAL BIOLOGY | NEUROSCIENCES | NEONATAL-RATS | IN-VIVO | CENTRAL-NERVOUS-SYSTEM | METHYL-D-ASPARTATE | MOLECULAR-MECHANISMS | Brain - metabolism | Neurons - pathology | Animals | Humans | Brain - pathology | Neurons - metabolism | Hypoxia-Ischemia, Brain - pathology | Hypoxia-Ischemia, Brain - metabolism | Neurosciences | Ischemia | Neurons | Cell death | Analysis | Mortality | Biochemistry | Health aspects
Immature brain | Autophagy | Neuronal cell death | Hypoxia-ischemia | BECLIN 1 | BRAIN-INJURY | ACID-INDUCED EXCITOTOXICITY | N-TERMINAL KINASE | KAINIC ACID | DEVELOPMENTAL BIOLOGY | NEUROSCIENCES | NEONATAL-RATS | IN-VIVO | CENTRAL-NERVOUS-SYSTEM | METHYL-D-ASPARTATE | MOLECULAR-MECHANISMS | Brain - metabolism | Neurons - pathology | Animals | Humans | Brain - pathology | Neurons - metabolism | Hypoxia-Ischemia, Brain - pathology | Hypoxia-Ischemia, Brain - metabolism | Neurosciences | Ischemia | Neurons | Cell death | Analysis | Mortality | Biochemistry | Health aspects
Journal Article
Details
Digital rights
Loading Rights1999, ISBN 9780801859854, xx, 346
Book
Details
Oncogene, ISSN 0950-9232, 04/2004, Volume 23, Issue 16, pp. 2881 - 2890
For many years apoptosis research has focused on caspases and their putative role as sole executioners of programmed cell death. Accumulating information now...
Cathepsins | Spi2A | TNFα | OXIDATIVE STRESS | TNF alpha | HIPPOCAMPAL NEURONAL DEATH | cathepsins | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL BIOLOGY | TUMOR-NECROSIS-FACTOR | ONCOLOGY | MYOCLONUS EPILEPSY EPM1 | CATHEPSIN-B CONTRIBUTES | GENETICS & HEREDITY | PH-INDUCED INACTIVATION | TNF-ALPHA | CYTOCHROME-C RELEASE | CYSTEINE PROTEASES | MEDIATED HEPATOCYTE APOPTOSIS | Animals | Signal Transduction | Humans | Mice | Permeability | Neoplasms - pathology | Lysosomes - physiology | Apoptosis | Mice, Knockout
Cathepsins | Spi2A | TNFα | OXIDATIVE STRESS | TNF alpha | HIPPOCAMPAL NEURONAL DEATH | cathepsins | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL BIOLOGY | TUMOR-NECROSIS-FACTOR | ONCOLOGY | MYOCLONUS EPILEPSY EPM1 | CATHEPSIN-B CONTRIBUTES | GENETICS & HEREDITY | PH-INDUCED INACTIVATION | TNF-ALPHA | CYTOCHROME-C RELEASE | CYSTEINE PROTEASES | MEDIATED HEPATOCYTE APOPTOSIS | Animals | Signal Transduction | Humans | Mice | Permeability | Neoplasms - pathology | Lysosomes - physiology | Apoptosis | Mice, Knockout
Journal Article
Details
Digital rights
Loading RightsEpilepsy Research, ISSN 0920-1211, 2017, Volume 135, pp. 87 - 94
Highlights • Seizure-induced respiratory arrest (S-IRA) activity changes were imaged in DBA/1 mice. • S-IRA induced activity increases in raphe, amygdala, and...
Neurology | MEMRI | Raphe nuclei | DBA/1 mice | Periaqueductal gray | SUDEP | Audiogenic seizure | INDUCED RESPIRATORY ARREST | C-FOS | BRAIN-STEM | CLINICAL NEUROLOGY | AUDIOGENIC-SEIZURES | INFERIOR COLLICULUS | SUPERIOR COLLICULUS | NEURONAL NETWORK | MEDULLA-OBLONGATA | DBA/1 MOUSE MODEL | Respiratory Insufficiency - physiopathology | Brain - diagnostic imaging | Acoustic Stimulation | Brain - physiopathology | Mice, Inbred C57BL | Epilepsy - diagnostic imaging | Respiratory Insufficiency - etiology | Male | Manganese Compounds | Epilepsy - physiopathology | Respiratory Insufficiency - diagnostic imaging | Seizures - diagnostic imaging | Death, Sudden | Magnetic Resonance Imaging | Seizures - physiopathology | Animals | Contrast Media | Chlorides | Mice, Inbred DBA | Respiration | Disease Models, Animal | Prevention | Medicine, Experimental | Medical research | Seizures (Medicine) | Magnetic resonance imaging | Epilepsy
Neurology | MEMRI | Raphe nuclei | DBA/1 mice | Periaqueductal gray | SUDEP | Audiogenic seizure | INDUCED RESPIRATORY ARREST | C-FOS | BRAIN-STEM | CLINICAL NEUROLOGY | AUDIOGENIC-SEIZURES | INFERIOR COLLICULUS | SUPERIOR COLLICULUS | NEURONAL NETWORK | MEDULLA-OBLONGATA | DBA/1 MOUSE MODEL | Respiratory Insufficiency - physiopathology | Brain - diagnostic imaging | Acoustic Stimulation | Brain - physiopathology | Mice, Inbred C57BL | Epilepsy - diagnostic imaging | Respiratory Insufficiency - etiology | Male | Manganese Compounds | Epilepsy - physiopathology | Respiratory Insufficiency - diagnostic imaging | Seizures - diagnostic imaging | Death, Sudden | Magnetic Resonance Imaging | Seizures - physiopathology | Animals | Contrast Media | Chlorides | Mice, Inbred DBA | Respiration | Disease Models, Animal | Prevention | Medicine, Experimental | Medical research | Seizures (Medicine) | Magnetic resonance imaging | Epilepsy
Journal Article
Details
Digital rights
Loading RightsNature, ISSN 0028-0836, 02/2009, Volume 457, Issue 7232, pp. 981 - 989
Naturally occurring axonal pruning and neuronal cell death help to sculpt neuronal connections during development, but their mechanistic basis remains poorly...
PROGRAMMED CELL-DEATH | TNF RECEPTOR | NERVOUS-SYSTEM | ALZHEIMERS-DISEASE | COMMISSURAL AXONS | FAMILY-MEMBERS | MULTIDISCIPLINARY SCIENCES | BAX-DEFICIENT MICE | GENE-EXPRESSION | AMYLOID PRECURSOR PROTEIN | DEGENERATION | Receptors, Tumor Necrosis Factor - metabolism | Caspase 6 - metabolism | Peptide Fragments - metabolism | Signal Transduction | Amyloid beta-Protein Precursor - chemistry | Caspase 3 - metabolism | bcl-2-Associated X Protein - metabolism | Axons - metabolism | Neurons - cytology | Peptide Fragments - chemistry | Animals | Caspases - metabolism | Alzheimer Disease - metabolism | Amyloid beta-Protein Precursor - metabolism | Cell Death | Protein Binding | Ligands | Mice | Neurons - metabolism | bcl-2-Associated X Protein - genetics | Neural receptors | Neurons | Amyloid beta-protein | Physiological aspects | Nervous system | Degeneration | Research | Health aspects | Monoclonal antibodies | Alzheimers disease | Kinases | Binding sites | Apoptosis
PROGRAMMED CELL-DEATH | TNF RECEPTOR | NERVOUS-SYSTEM | ALZHEIMERS-DISEASE | COMMISSURAL AXONS | FAMILY-MEMBERS | MULTIDISCIPLINARY SCIENCES | BAX-DEFICIENT MICE | GENE-EXPRESSION | AMYLOID PRECURSOR PROTEIN | DEGENERATION | Receptors, Tumor Necrosis Factor - metabolism | Caspase 6 - metabolism | Peptide Fragments - metabolism | Signal Transduction | Amyloid beta-Protein Precursor - chemistry | Caspase 3 - metabolism | bcl-2-Associated X Protein - metabolism | Axons - metabolism | Neurons - cytology | Peptide Fragments - chemistry | Animals | Caspases - metabolism | Alzheimer Disease - metabolism | Amyloid beta-Protein Precursor - metabolism | Cell Death | Protein Binding | Ligands | Mice | Neurons - metabolism | bcl-2-Associated X Protein - genetics | Neural receptors | Neurons | Amyloid beta-protein | Physiological aspects | Nervous system | Degeneration | Research | Health aspects | Monoclonal antibodies | Alzheimers disease | Kinases | Binding sites | Apoptosis
Journal Article
Details
Digital rights
Loading RightsJournal of Cell Science, ISSN 0021-9533, 05/2013, Volume 126, Issue 9, pp. 1905 - 1912
Lysosomes serve as the cellular recycling centre and are filled with numerous hydrolases that can degrade most cellular macromolecules. Lysosomal membrane...
CANCER-CELLS | TUMOR-NECROSIS-FACTOR | NEURONAL DEATH | CATHEPSIN-B CONTRIBUTES | LYSOSOMOTROPIC DETERGENT | TNF-ALPHA | CYTOCHROME-C RELEASE | T-LYMPHOCYTES | MEDIATED HEPATOCYTE APOPTOSIS | MEMBRANE PERMEABILIZATION | CELL BIOLOGY | Animals | Cathepsins - metabolism | Humans | Lysosomes - enzymology | Apoptosis - physiology | Permeability | Intracellular Membranes - metabolism
CANCER-CELLS | TUMOR-NECROSIS-FACTOR | NEURONAL DEATH | CATHEPSIN-B CONTRIBUTES | LYSOSOMOTROPIC DETERGENT | TNF-ALPHA | CYTOCHROME-C RELEASE | T-LYMPHOCYTES | MEDIATED HEPATOCYTE APOPTOSIS | MEMBRANE PERMEABILIZATION | CELL BIOLOGY | Animals | Cathepsins - metabolism | Humans | Lysosomes - enzymology | Apoptosis - physiology | Permeability | Intracellular Membranes - metabolism
Journal Article
Details
Digital rights
Loading RightsMolecular Biology of the Cell, ISSN 1059-1524, 03/2014, Volume 25, Issue 6, pp. 936 - 947
Emerging findings suggest that two lineages of mitochondrial Ca2+ uptake participate during active and resting states: 1) the major eukaryotic membrane...
TRANSPORTER FAMILY SLC25 | CONFOCAL MICROSCOPY | ESSENTIAL COMPONENT | LIVER-MITOCHONDRIA | MG/P-I CARRIER | CALCIUM UNIPORTER | ENDOPLASMIC-RETICULUM | NEURONAL MITOCHONDRIA | VENTRICULAR MYOCYTES | PERMEABILITY TRANSITION | CELL BIOLOGY | Amino Acid Transport Systems, Acidic - antagonists & inhibitors | RNA, Small Interfering - genetics | Reactive Oxygen Species - metabolism | Calcium Channels - metabolism | Oxidative Stress | Calcium - metabolism | Humans | Amino Acid Transport Systems, Acidic - metabolism | Calcium-Binding Proteins - antagonists & inhibitors | Mitochondrial Proteins - genetics | Membrane Potential, Mitochondrial - drug effects | Mitochondrial Membrane Transport Proteins - genetics | Cation Transport Proteins - metabolism | Mitochondrial Proteins - metabolism | Adenosine Triphosphate - metabolism | Antiporters - genetics | Cell Death | Cation Transport Proteins - genetics | Ion Transport - drug effects | Clone Cells | Calcium Channels - genetics | Calcium Signaling | Calcium-Binding Proteins - metabolism | Protein Structure, Tertiary | Mitochondrial Membrane Transport Proteins - metabolism | Histamine - pharmacology | Mitochondrial Proteins - antagonists & inhibitors | Gene Expression Regulation | Antiporters - metabolism | Mitochondria - metabolism | Mitochondria - drug effects | Oxygen Consumption - drug effects | Amino Acid Transport Systems, Acidic - genetics | HeLa Cells | Antiporters - antagonists & inhibitors | Calcium-Binding Proteins - genetics | RNA, Small Interfering - metabolism
TRANSPORTER FAMILY SLC25 | CONFOCAL MICROSCOPY | ESSENTIAL COMPONENT | LIVER-MITOCHONDRIA | MG/P-I CARRIER | CALCIUM UNIPORTER | ENDOPLASMIC-RETICULUM | NEURONAL MITOCHONDRIA | VENTRICULAR MYOCYTES | PERMEABILITY TRANSITION | CELL BIOLOGY | Amino Acid Transport Systems, Acidic - antagonists & inhibitors | RNA, Small Interfering - genetics | Reactive Oxygen Species - metabolism | Calcium Channels - metabolism | Oxidative Stress | Calcium - metabolism | Humans | Amino Acid Transport Systems, Acidic - metabolism | Calcium-Binding Proteins - antagonists & inhibitors | Mitochondrial Proteins - genetics | Membrane Potential, Mitochondrial - drug effects | Mitochondrial Membrane Transport Proteins - genetics | Cation Transport Proteins - metabolism | Mitochondrial Proteins - metabolism | Adenosine Triphosphate - metabolism | Antiporters - genetics | Cell Death | Cation Transport Proteins - genetics | Ion Transport - drug effects | Clone Cells | Calcium Channels - genetics | Calcium Signaling | Calcium-Binding Proteins - metabolism | Protein Structure, Tertiary | Mitochondrial Membrane Transport Proteins - metabolism | Histamine - pharmacology | Mitochondrial Proteins - antagonists & inhibitors | Gene Expression Regulation | Antiporters - metabolism | Mitochondria - metabolism | Mitochondria - drug effects | Oxygen Consumption - drug effects | Amino Acid Transport Systems, Acidic - genetics | HeLa Cells | Antiporters - antagonists & inhibitors | Calcium-Binding Proteins - genetics | RNA, Small Interfering - metabolism
Journal Article
Details
Digital rights
Loading Rights