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1. Full Text The regulation and pharmacology of endothelial nitric oxide synthase
by Dudzinski, David M and Igarashi, Junsuke and Greif, Daniel and Michel, Thomas
Annual Review of Pharmacology and Toxicology, ISSN 0362-1642, 2006, Volume 46, Issue 1, pp. 235 - 276
Nitric oxide (NO) is a small, diffusible, lipophilic free radical gas that mediates significant and diverse signaling functions in nearly every organ system in... 
Oxidative stress | eNOS | Caveolae | Cyclic GMP | VEGF | S-nitrosylation | Sphingosine 1-phosphate | Vascular signaling | FLUID SHEAR-STRESS | MESSENGER-RNA EXPRESSION | LIGHT-CHAIN PHOSPHATASE | cyclic GMP | LOW-DENSITY-LIPOPROTEIN | GROWTH-FACTOR RECEPTORS | SUPEROXIDE ANION PRODUCTION | VASCULAR SMOOTH-MUSCLE | vascular signaling | sphingosine l-phosphate | PROTEIN-PROTEIN INTERACTIONS | TOXICOLOGY | caveolae | oxidative stress | CORONARY-ARTERY-DISEASE | NEURONAL NO SYNTHASE | Animals | Isoenzymes - metabolism | Humans | Enzyme Inhibitors - pharmacology | Nitric Oxide - physiology | Nitric Oxide Synthase Type III - metabolism | Nitric Oxide - metabolism | Isoenzymes - antagonists & inhibitors | Gene Expression Regulation, Developmental - physiology | Nitric Oxide Synthase Type III - antagonists & inhibitors
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2. Full Text Beta(3)-Adrenoreceptor Stimulation Ameliorates Myocardial Ischemia-Reperfusion Injury Via Endothelial Nitric Oxide Synthase and Neuronal Nitric Oxide Synthase Activation
by Aragon, JP and Condit, ME and Bhushan, S and Predmore, BL and Patel, SS and Grinsfelder, DB and Gundewar, S and Jha, S and Calvert, JW and Barouch, LA and Lavu, M and Wright, HM and Lefer, DJ
JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY, ISSN 0735-1097, 12/2011, Volume 58, Issue 25, pp. 2683 - 2691
Objectives This paper examined whether nebivolol protects the heart via nitric oxide (NO) synthase and NO-dependent signaling in an in vivo model of acute... 
MOLECULAR CHARACTERIZATION | nitric oxide | CARDIAC & CARDIOVASCULAR SYSTEMS | RAT | CARDIOVASCULAR-SYSTEM | neuronal nitric oxide synthase | beta adrenergic receptor | NEBIVOLOL | MECHANISMS | DEFICIENT MICE | cardiac ischemia | HUMAN BETA-3-ADRENERGIC RECEPTOR | endothelial nitric oxide synthase | 3RD-GENERATION BETA-BLOCKER | HUMAN HEART | AGONIST
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3. Full Text Beta 3-adrenoreceptor stimulation ameliorates myocardial ischemia-reperfusion injury via endothelial nitric oxide synthase and neuronal nitric oxide synthase activation
by Aragón, Juan P and Condit, Marah E and Bhushan, Shashi and Predmore, Benjamin L and Patel, Sandeep S and Grinsfelder, D. Bennett and Gundewar, Susheel and Jha, Saurabh and Calvert, John W and Barouch, Lili A and Lavu, Madhav and Wright, Harold M and Lefer, David J
Journal of the American College of Cardiology, ISSN 0735-1097, 12/2011, Volume 58, Issue 25, pp. 2683 - 2691
Objectives: This paper examined whether nebivolol protects the heart via nitric oxide (NO) synthase and NO-dependent signaling in an in vivo model of acute... 
nitric oxide | adrenergic receptor | endothelial nitric oxide synthase | neuronal nitric oxide synthase | cardiac ischemia | beta
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4. Full Text Roles of Nitric Oxide Synthase Isoforms in Neurogenesis
by Chong, Cheong-Meng and Chong, Cheong-Meng and Ai, Nana and Ai, Nana and Ke, Minjing and Ke, Minjing and Tan, Yuan and Tan, Yuan and Huang, Zhijian and Huang, Zhijian and Li, Yong and Li, Yong and Lu, Jia-Hong and Lu, Jia-Hong and Ge, Wei and Ge, Wei and Su, Huanxing and Su, Huanxing
Molecular Neurobiology, ISSN 0893-7648, 3/2018, Volume 55, Issue 3, pp. 2645 - 2652
Nitric oxide (NO), a free radical gas, acts as a neurotransmitter or neuromodulator in the central nervous system (CNS). It has been widely explored as a... 
Neurology | Neurosciences | Biomedicine | Nitric oxide | Neurobiology | Nitric oxide synthase | Neural stem cells | Proliferation | Neurogenesis | Cell Biology | ADULT NEUROGENESIS | SUBVENTRICULAR ZONE | NEUROSCIENCES | NEURAL STEM-CELLS | GROWTH-FACTOR RECEPTOR | IN-VITRO | CENTRAL-NERVOUS-SYSTEM | DIFFERENTIATION | DENTATE GYRUS | BRAIN | Brain - cytology | Nitric Oxide Synthase - chemistry | Animals | Neurogenesis - physiology | Humans | Brain - enzymology | Nitric Oxide Synthase - physiology | Neural Stem Cells - enzymology | Isoenzymes - physiology | Enzymes | Medical colleges | Neurons | Stem cells | Brain | Neurodegeneration | Medical treatment | Central nervous system | Isoforms | Inflammation | Neuromodulation | Nitric-oxide synthase
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5. Full Text Neuronal nitric oxide synthase in vascular physiology and diseases
by Costa, Eduardo D and Rezende, Bruno A and Cortes, Steyner F and Lemos, Virginia S
Frontiers in Physiology, ISSN 1664-042X, 06/2016, Volume 7, p. 206
The family of nitric oxide synthases (NOS) has significant importance in various physiological mechanisms and is also involved in many pathological processes.... 
Hypertension | Neuronal nitric oxide synthase | Hydrogen peroxide | Vascular function | Nitric oxide | Atherosclerosis | atherosclerosis | INTERDOMAIN ELECTRON-TRANSFER | vascular function | nitric oxide | PHYSIOLOGY | ENDOTHELIAL DYSFUNCTION | SPONTANEOUSLY HYPERTENSIVE-RATS | NULL MUTANT MICE | hydrogen peroxide | neuronal nitric oxide synthase | SKELETAL-MUSCLE | SMOOTH-MUSCLE | L-ARGININE | HYDROGEN-PEROXIDE | CEREBRAL-BLOOD-FLOW | hypertension | NOS KNOCKOUT MICE | Physiological aspects | Neurons | Nitric Oxide | Hydrogen Peroxide
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6. Full Text Nitric oxide donors (nitrates), L-arginine, or nitric oxide synthase inhibitors for acute stroke
by Bath, Philip M.W and Krishnan, Kailash and Appleton, Jason P
Cochrane Database of Systematic Reviews, ISSN 1469-493X, 04/2017, Volume 2017, Issue 4, pp. CD000398 - CD000398
Background Nitric oxide (NO) has multiple effects that may be beneficial in acute stroke, including lowering blood pressure, and promoting reperfusion and... 
MIDDLE CEREBRAL-ARTERY | Stroke [drug therapy] | MICE DEFICIENT | Humans | TRANSDERMAL GLYCERYL TRINITRATE | Nitric Oxide Donors [therapeutic use] | Brain Ischemia [drug therapy] | RAPID INTERVENTION | BLOOD-PRESSURE | MEDICINE, GENERAL & INTERNAL | NEURONAL ISOFORM | Enzyme Inhibitors [therapeutic use] | ACUTE ISCHEMIC-STROKE | INFARCTION VOLUME | Arginine [therapeutic use] | RAT-BRAIN | Nitric Oxide Synthase [ antagonists & inhibitors] | ULTRA-ACUTE STROKE | Acute Disease | Nitroglycerin - therapeutic use | Nitric Oxide Synthase - antagonists & inhibitors | Stroke - drug therapy | Enzyme Inhibitors - therapeutic use | Randomized Controlled Trials as Topic | Brain Ischemia - drug therapy | Quality of Life | Nitroglycerin - adverse effects | Nitric Oxide Donors - therapeutic use | Stroke - mortality | Arginine - therapeutic use | Headache - chemically induced | Nitric Oxide Donors - adverse effects | Index Medicus
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7. The Adaptive Role of Neuronal Nitric Oxide Synthase in Maintaining Oxygen Homeostasis during Acute Anemia
by Tsui, Albert King-Yeung
06/2012
Mammals are well adapted to respond to changes in ambient oxygen concentration (O2) by activating homeostatic physiological and cellular responses which... 
Neuronal nitric oxide synthase | Knockout mice | Anemia | Cardiovascular | S-nitrosylation | Hemoglobin | Hypoxia-inducible factor | Hypoxia | 0719
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8. Full Text Nitric oxide synthase in hypoxic or ischemic brain injury
by Liu, Haiting and Li, Jiao and Zhao, Fengyan and Wang, Huiqing and Qu, Yi and Mu, Dezhi
Reviews in the Neurosciences, ISSN 0334-1763, 02/2015, Volume 26, Issue 1, pp. 105 - 117
Hypoxic or ischemic stress causes many serious brain injuries, including stroke and neonatal hypoxia ischemia encephalopathy. During brain hypoxia ischemia... 
hypoxia | brain injury | nitric oxide synthase | ischemia | Hypoxia | Nitric oxide synthase | Ischemia | Brain injury | INDUCIBLE FACTOR-1-ALPHA | DEVELOPING RAT-BRAIN | INOS EXPRESSION | REPERFUSION INJURY | NEUROSCIENCES | CELL-DEATH | S-NITROSYLATION | ATTENUATES NEURONAL APOPTOSIS | CEREBRAL-ARTERY OCCLUSION | SER847 PHOSPHORYLATION | NF-KAPPA-B | Brain Ischemia - enzymology | Animals | Hypoxia, Brain - enzymology | Humans | Brain Ischemia - pathology | Nitric Oxide Synthase - metabolism | Nitric Oxide Synthase Type III - metabolism | Hypoxia, Brain - pathology | Nitric Oxide Synthase Type II - metabolism | Physiological aspects | Cerebral ischemia | Health aspects | Nitric oxide
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9. Full Text Cyclic AMP-dependent phosphorylation of neuronal nitric oxide synthase mediates penile erection
by K. Joseph Hurt and Sena F. Sezen and Gwen F. Lagoda and Biljana Musicki and Gerald A. Rameau and Solomon H. Snyder and Arthur L. Burnett
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2012, Volume 109, Issue 41, pp. 16624 - 16629
Nitric oxide (NO) generated by neuronal NO synthase (nNOS) initiates penile erection, but has not been thought to participate in the sustained erection... 
Alternative splicing | Phosphorylation | Calcium | Penile erection | Electrical stimulation | Antibodies | Oxides | Smooth muscle | Physiology | Physiological regulation | Phosphoantibody | Smooth muscle relaxation | Gasotransmitter | Cyclic GMP | Endothelial NOS | NNOS | NERVOUS-SYSTEM | ACTIVATION | PROTEIN-KINASE | MULTIDISCIPLINARY SCIENCES | cyclic GMP | endothelial NOS | AKT | smooth muscle relaxation | phosphoantibody | IN-VITRO | CORPUS CAVERNOSUM | CA2 | gasotransmitter | GENE-EXPRESSION | MICE | Immunohistochemistry | Penis - physiology | Electric Stimulation | Male | Penis - innervation | Penile Erection - drug effects | Nitric Oxide Synthase Type I - antagonists & inhibitors | Cyclic AMP-Dependent Protein Kinases - antagonists & inhibitors | Phosphorylation - drug effects | Cyclic AMP - metabolism | Cyclic AMP-Dependent Protein Kinases - metabolism | NG-Nitroarginine Methyl Ester - pharmacology | Colforsin - pharmacology | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Rats | Rats, Sprague-Dawley | Serine - metabolism | Blotting, Western | Mice, Knockout | Animals | Penis - metabolism | Nitric Oxide Synthase Type I - metabolism | Mice | Nitric Oxide Synthase Type I - genetics | Penile Erection - physiology | Penis | Nitric oxide | Physiological aspects | Cyclic adenylic acid | Research | Health aspects | Biological Sciences
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10. Full Text Tapentadol and nitric oxide synthase systems
by Bujalska-Zadrożny, Magdalena and Wolińska, Renata and Gąsińska, Emilia and Nagraba, Łukasz
Behavioural Pharmacology, ISSN 0955-8810, 04/2015, Volume 26, Issue 3, pp. 282 - 288
Tapentadol, a new analgesic drug with a dual mechanism of action (μ-opioid receptor agonism and norepinephrine reuptake inhibition), is indicated for the... 
NOS | antinociception | l-NIO | rat | l-NIL | 7-NI | tapentadol | l-NOArg | RAT SPINAL-CORD | THERMAL HYPERALGESIA | L-NOArg | OPIOID RECEPTOR AGONIST | DORSAL-ROOT GANGLIA | INTRAVENOUS MORPHINE | NEUROSCIENCES | NEUROPATHIC PAIN | SPINOTHALAMIC TRACT NEURONS | L-NIL | INDUCED HYPERALGESIA | L-NIO | BEHAVIORAL SCIENCES | PHARMACOLOGY & PHARMACY | MORPHINE ANTINOCICEPTION | REUPTAKE INHIBITOR | Nitric Oxide Synthase - drug effects | Analgesics, Opioid - pharmacology | Rats | Male | Receptors, Opioid, mu - agonists | Dose-Response Relationship, Drug | Animals | Analgesics, Opioid - administration & dosage | Pain - drug therapy | Pain - physiopathology | Phenols - administration & dosage | Nitric Oxide Synthase - metabolism | Disease Models, Animal | Phenols - pharmacology
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11. Full Text Neuronal nitric oxide synthase contributes to chronic stress‐induced depression by suppressing hippocampal neurogenesis
by Zhou, Qi‐Gang and Hu, Yao and Hua, Yao and Hu, Mei and Luo, Chun‐Xia and Han, Xiao and Zhu, Xin‐Jian and Wang, Bin and Xu, Jin‐Shu and Zhu, Dong‐Ya
Journal of Neurochemistry, ISSN 0022-3042, 12/2007, Volume 103, Issue 5, pp. 1843 - 1854
Increasing evidence suggests that depression may be associated with a lack of hippocampal neurogenesis. It is well established that neuronal nitric oxide... 
neurogenesis | stress | neuronal nitric oxide synthase | nitric oxide | hippocampal | depression | Depression | Hippocampal | Neuronal nitric oxide synthase | Neurogenesis | Nitric oxide | Stress | PROGENITOR CELLS | RAT PARAVENTRICULAR NUCLEUS | ANTIDEPRESSANT-LIKE | BIOCHEMISTRY & MOLECULAR BIOLOGY | ADULT DENTATE GYRUS | CHRONIC MILD STRESS | ELEMENT-BINDING PROTEIN | CELL-PROLIFERATION | NEUROSCIENCES | PREFRONTAL CORTEX | ISCHEMIA STIMULATES NEUROGENESIS | ANXIOLYTIC-LIKE | Cell Proliferation | Depression - pathology | Enzyme Inhibitors - pharmacology | Zidovudine - pharmacology | Bromodeoxyuridine | Male | Hippocampus - pathology | Hindlimb Suspension - methods | Mice, Knockout | Nitric Oxide Synthase Type I - deficiency | Behavior, Animal | Depression - etiology | Indazoles - pharmacology | Animals | Drug Interactions | Analysis of Variance | Time Factors | Neurons - physiology | Mice | Nitric Oxide Synthase Type I - physiology | Stress, Psychological - complications | Neurons | Depression, Mental | Brain | Neurosciences | Chemistry | Brain research | Rodents | Mental depression
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12. Full Text Transplantation of enteric nervous system stem cells rescues nitric oxide synthase deficient mouse colon
by McCann, Conor J and Cooper, Julie E and Natarajan, Dipa and Jevans, Benjamin and Burnett, Laura E and Burns, Alan J and Thapar, Nikhil
Nature Communications, ISSN 2041-1723, 07/2017, Volume 8, Issue 1, p. 15937
Enteric nervous system neuropathy causes a wide range of severe gut motility disorders. Cell replacement of lost neurons using enteric neural stem cells (ENSC)... 
INHIBITORY NEUROTRANSMISSION | MULTIDISCIPLINARY SCIENCES | GASTROINTESTINAL-TRACT | NEURONS | SMALL-INTESTINE | CAJAL | HIRSCHSPRUNGS-DISEASE | REBOUND EXCITATION | KNOCKOUT MICE | SLOW-TRANSIT CONSTIPATION | INTERSTITIAL-CELLS | Colon - enzymology | Enteric Nervous System - cytology | Humans | Mice, Inbred C57BL | Male | Intestinal Pseudo-Obstruction - enzymology | Nitric Oxide Synthase - genetics | Intestinal Pseudo-Obstruction - genetics | Intestinal Pseudo-Obstruction - physiopathology | Gastrointestinal Motility | Enteric Nervous System - enzymology | Neurons - transplantation | Animals | Nitric Oxide Synthase - deficiency | Neural Stem Cells - transplantation | Colon - physiopathology | Female | Intestinal Pseudo-Obstruction - surgery | Mice | Therapy | Motility | Neurons | Disorders | Recovery of function | Stem cell transplantation | Interstitial cells | Nervous system | Transplantation | Neuropathy | Interstitial cells of Cajal | Transit | Nitric-oxide synthase | Enteric nervous system | Restoration | Nitric oxide | Stem cells | Neural stem cells | Colon | Digestive tract | Gastric motility
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13. Full Text Endothelial Nitric Oxide Synthase Regulates Brain-Derived Neurotrophic Factor Expression and Neurogenesis after Stroke in Mice
by Chen, Jieli and Zacharek, Alex and Zhang, Chunling and Jiang, Hao and Li, Yi and Roberts, Cynthia and Lu, Mei and Kapke, Alissa and Chopp, Michael
Journal of Neuroscience, ISSN 0270-6474, 03/2005, Volume 25, Issue 9, pp. 2366 - 2375
Here, we investigate the effects of endothelial nitric oxide synthase (eNOS) on angiogenesis, neurogenesis, neurotrophic factor expression, and neurological... 
Focal cerebral ischemia | Angiogenesis | Neural progenitor cells | eNOS | BDNF | Neurogenesis | PROGENITOR CELLS | angiogenesis | FOCAL CEREBRAL-ISCHEMIA | FIBROBLAST-GROWTH-FACTOR | SUBVENTRICULAR ZONE | neural progenitor cells | SYNAPTIC PLASTICITY | HIPPOCAMPAL-NEURONS | NEUROSCIENCES | NEURAL STEM-CELLS | TELOMERASE ACTIVITY | neurogenesis | focal cerebral ischemia | MARROW STROMAL CELLS | FACTOR-INDUCED ANGIOGENESIS | Enzyme-Linked Immunosorbent Assay - methods | Infarction, Middle Cerebral Artery - physiopathology | Nestin | Cell Proliferation | Gene Expression - drug effects | Neurites - physiology | Microtubule-Associated Proteins - metabolism | Ki-67 Antigen - metabolism | Male | Infarction, Middle Cerebral Artery - metabolism | Nitric Oxide Synthase Type III - deficiency | Psychomotor Performance - physiology | Cell Movement - genetics | Cell Movement - physiology | Corneal Neovascularization | Time Factors | Gene Expression - physiology | Nitric Oxide Synthase Type III - physiology | Telomerase - metabolism | Blood Pressure - physiology | Brain-Derived Neurotrophic Factor - metabolism | Bromodeoxyuridine - metabolism | Vascular Endothelial Growth Factor A - pharmacology | Endothelial Cells - physiology | Disease Models, Animal | Recovery of Function - genetics | Mice, Inbred C57BL | Neuropeptides - metabolism | Psychomotor Performance - drug effects | Mice, Knockout | Naphthalenes | Oxepins | Immunohistochemistry - methods | Nerve Tissue Proteins - metabolism | Animals | Cell Count - methods | Nitric Oxide Synthase Type III - pharmacology | Brain - pathology | Mice | Neurologic Examination - methods | Intermediate Filament Proteins - metabolism | Neurobiology of Disease
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14. Full Text S-Nitrosylation of STIM1 by Neuronal Nitric Oxide Synthase Inhibits Store-Operated Ca2+ Entry
by Gui, Le and Zhu, Jinhui and Lu, Xiangru and Lu, Wei-Yang and Sims, Stephen M and Stathopulos, Peter B and Feng, Qingping
Journal of Molecular Biology, ISSN 0022-2836, 06/2018, Volume 430, Issue 12, pp. 1773 - 1785
Store-operated Ca entry (SOCE) mediated by stromal interacting molecule-1 (STIM1) and Orai1 represents a major route of Ca entry in mammalian cells and is... 
stromal interaction molecule-1 | neuronal nitric oxide synthase | nitric oxide | store-operated calcium entry | S-nitrosylation | CARDIAC-FUNCTION | CHEMICAL-SHIFTS | BIOCHEMISTRY & MOLECULAR BIOLOGY | DEPLETION | RELEASE | CARDIOMYOCYTES | PLASMA-MEMBRANE | CALCIUM-ENTRY | INITIATION MECHANISM | OLIGOMERIZATION
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