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Applied and Environmental Microbiology, ISSN 0099-2240, 07/2017, Volume 83, Issue 13
NAD and its reduced form NADH function as essential redox cofactors and have major roles in determining cellular metabolic features. NAD can be synthesized... 
NADPH | Nicotinic acid | Biosynthesis | Colorimetry | Cofactors | Proteins | Engineering | NADH | E coli | Vitamin B | Catalysis | Chemical synthesis | Stationary phase | Adenine | Protein biosynthesis | Metabolism | Malic enzyme | Mutants | NAD | Acids | Nicotinamide adenine dinucleotide | Metabolic engineering | Nicotinamide | Organic chemicals | Viability
Journal Article
Journal Article
Journal Article
FEBS Letters, ISSN 0014-5793, 03/2014, Volume 588, Issue 6, pp. 1016 - 1023
Journal Article
Biochimie Open, ISSN 2214-0085, 2015, Volume 1, Issue C, pp. 61 - 69
is an intestinal protozoan parasite that causes giardiasis, a disease of high prevalence in Latin America, Asia and Africa. Giardiasis leads to poor absorption... 
NAD metabolism | Giardia lamblia | Enzyme activity | NMNAT
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 02/2002, Volume 277, Issue 5, pp. 3698 - 3707
The nadD gene, encoding the enzyme nicotinic acid mononucleotide (NaMN) adenylyltransferase (AT), is essential for the synthesis of NAD and subsequent... 
nicotinic acid mononucleotide adenylyltransferase | nadD gene
Journal Article
Toxicological Sciences, ISSN 1096-6080, 03/2015, Volume 144, Issue 1, pp. 163 - 172
Nicotinamide phosphoribosyltransferase (NAMPT) is a pleiotropic protein with intra-and extra-cellular functions as an enzyme, cytokine, growth factor, and... 
Eye | Pathology | Tumor metabolism | Messenger rna | Nicotinamide adenine dinucleotide | Retinal degeneration | Nicotinic acid mononucleotide | Retinal pigmented epithelial cell culture | retinal degeneration | pathology | ACID | EFFICACY | NAMPT INHIBITORS | DEGENERATION | eye | nicotinamide adenine dinucleotide | LEBER CONGENITAL AMAUROSIS | THERAPY | messenger RNA | nicotinic acid mononucleotide | PATHWAY | BIOSYNTHESIS | TOXICOLOGY | MUTATIONS | retinal pigmented epithelial cell culture | PHASE-I | tumor metabolism | Species Specificity | Nicotinamide-Nucleotide Adenylyltransferase - metabolism | Humans | Male | Structure-Activity Relationship | Nicotinamide Phosphoribosyltransferase - antagonists & inhibitors | Pentosyltransferases - metabolism | RNA, Messenger - metabolism | Niacin - pharmacology | Retinal Pigment Epithelium - pathology | Heterocyclic Compounds, 2-Ring - toxicity | Nicotinamide Phosphoribosyltransferase - metabolism | Enzyme Inhibitors - chemistry | Enzyme Inhibitors - toxicity | Female | Molecular Structure | Cytokines - genetics | Retinal Pigment Epithelium - drug effects | Cell Line | Cell Survival - drug effects | Cytokines - metabolism | Risk Assessment | Sulfones - toxicity | Cyanides - toxicity | Retinal Pigment Epithelium - enzymology | Rats, Sprague-Dawley | Gene Expression Regulation, Enzymologic | Nicotinamide Phosphoribosyltransferase - genetics | Animals | Mice, Nude | Guanidines - toxicity | Cytokines - antagonists & inhibitors | Index Medicus
Journal Article
Journal Article
Cardiovascular Toxicology, ISSN 1530-7905, 07/2017, Volume 17, Issue 3, pp. 307 - 318
Nicotinamide phosphoribosyltransferase (NAMPT) is a pleiotropic protein that functions as an enzyme, cytokine, growth factor and hormone. As a target for... 
Heart | Pathology | Myocardial degeneration | Tumor metabolism | Nicotinamide adenine dinucleotide | Cardiomyocytes | Nicotinic acid mononucleotide | Cardiotoxicity | Impedance | Viability | CARDIAC & CARDIOVASCULAR SYSTEMS | ACID | EFFICACY | VIVO | NAMPT INHIBITORS | FK866 | BIOSYNTHESIS | CHS-828 | TOXICOLOGY | PHASE-I | EXPRESSION | Induced Pluripotent Stem Cells - enzymology | Cytokines - metabolism | Induced Pluripotent Stem Cells - drug effects | Species Specificity | Sulfones - toxicity | Humans | Rats | Embryonic Stem Cells - enzymology | Male | Nicotinamide Phosphoribosyltransferase - antagonists & inhibitors | Rats, Sprague-Dawley | Dose-Response Relationship, Drug | Myocytes, Cardiac - enzymology | Heterocyclic Compounds, 2-Ring - toxicity | Animals | Myocytes, Cardiac - drug effects | Embryonic Stem Cells - drug effects | Nicotinamide Phosphoribosyltransferase - metabolism | Enzyme Inhibitors - toxicity | Female | Cytokines - antagonists & inhibitors | Heart failure | Niacinamide | Purines | Rodents | Physiological aspects | Rankings | Niacin | Cells | NADPH | Energy metabolism | Toxicity | Nicotinic acid | Congestive heart failure | Assaying | Salvage | Biocompatibility | Degeneration | Safety | Inhibition | Effusion | Pharmacology | Thorax | Metabolism | Mitigation | Inhibitors | Nicotinamide phosphoribosyltransferase | Death | In vivo methods and tests | Nicotinamide | In vitro methods and tests | ATP | Adenosine triphosphatase | Index Medicus
Journal Article