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Inflammation, ISSN 0360-3997, 4/2015, Volume 38, Issue 2, pp. 510 - 519
Sepsis is often initiated by invasive infection, characterized by overwhelming induction of pro-inflammatory cytokines. The incidence and mortality of sepsis... 
Pathology | nuclear factor kappa B signaling pathway | alpha-lipoic acid | Medicine & Public Health | inflammation | Rheumatology | Internal Medicine | acute kidney injury | Pharmacology/Toxicology | sepsis | PROTECTS MICE | ACTIVATION | HUMAN-DISEASE | COX-2 | IMMUNOLOGY | CELL BIOLOGY | SEPTIC SHOCK | INFLAMMATORY RESPONSE | UP-REGULATION | EXPRESSION | Dinoprostone - secretion | Inflammation - chemically induced | Tumor Necrosis Factor-alpha - metabolism | Nitric Oxide Synthase Type II - biosynthesis | Phosphorylation | I-kappa B Proteins - metabolism | Lipopolysaccharides | Mesangial Cells - drug effects | Cyclooxygenase 2 - biosynthesis | Sepsis - pathology | I-kappa B Kinase - metabolism | Inflammation - drug therapy | Interleukin-1beta - metabolism | Interleukin-6 - metabolism | Cell Line | Cell Survival - drug effects | NF-KappaB Inhibitor alpha | NF-kappa B - antagonists & inhibitors | Sepsis - immunology | Signal Transduction | Acute Kidney Injury - drug therapy | Acute Kidney Injury - pathology | Anti-Inflammatory Agents - pharmacology | Mesangial Cells - immunology | Rats | Thioctic Acid - pharmacology | Inflammation - immunology | Antioxidants - pharmacology | Interleukin-6 - secretion | Acute Kidney Injury - prevention & control | Dinoprostone - metabolism | Animals | Transcription Factor RelA - metabolism | Acetylcysteine - pharmacology | Nitric Oxide - metabolism | Medicine, Experimental | Medical research | Research | Anti-inflammatory drugs
Journal Article
International Journal of Molecular Sciences, ISSN 1661-6596, 08/2015, Volume 16, Issue 9, pp. 20195 - 20211
Journal Article
Journal of Cancer Research and Clinical Oncology, ISSN 0171-5216, 5/2012, Volume 138, Issue 5, pp. 785 - 797
Pancreatic cancer is an aggressive malignancy, which generally develops resistance to chemotherapy. Agents that are safe and can sensitize cancer to... 
Medicine & Public Health | Hematology | Gemcitabine | Pancreatic cancer | Escin | Internal Medicine | Cancer Research | Oncology | Nuclear factor-κB | Nuclear factor-kappa B | CARCINOMA CELL-LINES | APOPTOSIS | CURCUMIN | PROLIFERATION | BETA-ESCIN | ANTITUMOR-ACTIVITY | HEPATOCELLULAR-CARCINOMA | ONCOLOGY | SIGNALING PATHWAY | CHEMORESISTANCE | EXPRESSION | Humans | Deoxycytidine - pharmacology | Male | NF-kappa B - metabolism | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Pancreatic Neoplasms - drug therapy | Escin - pharmacology | Gene Expression Regulation, Neoplastic - drug effects | Tumor Cells, Cultured | Carcinoma - pathology | Escin - administration & dosage | Carcinoma - drug therapy | Deoxycytidine - administration & dosage | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - genetics | Down-Regulation - drug effects | Down-Regulation - genetics | Drug Synergism | Xenograft Model Antitumor Assays | Animals | NF-kappa B - genetics | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Mice, Nude | Models, Biological | NF-kappa B - physiology | Carcinoma - genetics | Mice | Mice, Inbred BALB C | Deoxycytidine - analogs & derivatives | triterpene saponins | survivin | Bcl-2 protein | Chemoresistance | c-Myc protein | Data processing | Malignancy | Caspase-3 | Chemotherapy | NF- Kappa B protein | Bcl-x protein | Xenografts | Antitumor activity | cyclin D1 | gemcitabine | Cyclooxygenase-2 | Apoptosis | Tumors
Journal Article