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Current Medicinal Chemistry, ISSN 0929-8673, 08/2017, Volume 24, Issue 27, pp. 2988 - 3001
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2013, Volume 8, Issue 4, p. e61988
The neurodegenerative processes that underlie Alzheimer's disease are mediated, in part, by soluble oligomeric amyloid beta, a neurotoxic protein that inhibits... 
SPHINGOSINE 1-PHOSPHATE RECEPTORS | MULTIPLE-SCLEROSIS | MESSENGER-RNA | ADULT-RAT | ALZHEIMERS-DISEASE | MULTIDISCIPLINARY SCIENCES | CENTRAL-NERVOUS-SYSTEM | SPINAL-CORD-INJURY | AXONAL-TRANSPORT | FULL-LENGTH | SYNAPTIC PLASTICITY | Neurons - pathology | Amyloid beta-Peptides - toxicity | Humans | Neurotoxins - toxicity | Extracellular Signal-Regulated MAP Kinases - metabolism | Organophosphates - pharmacology | Sphingosine - pharmacology | Sphingosine - analogs & derivatives | Animals | MAP Kinase Signaling System - drug effects | Neuroprotective Agents - pharmacology | Neurons - enzymology | Cytoprotection - drug effects | Brain-Derived Neurotrophic Factor - biosynthesis | Brain-Derived Neurotrophic Factor - metabolism | Mice | Neurons - metabolism | Neurons - drug effects | Receptors, Lysosphingolipid - metabolism | Receptor, trkB - metabolism | Protein Multimerization - drug effects | Potentiation | Phosphates | Brain | Neuroprotection | Reactive oxygen species | Multiple sclerosis | Memory | Nervous system | Kinases | Sphingosine 1-phosphate | Proteins | Neurotoxicity | Lymphocytes | Rodents | Localization | Alzheimer's disease | Medical research | Neurodegenerative diseases | Neurons | Long-term potentiation | Cortex | Environmental health | Extracellular signal-regulated kinase | Pharmacology | Plasticity (hippocampal) | Gene expression | Chemical compounds | Neurological diseases | Medicine | Signaling | Brain research | Brain-derived neurotrophic factor | Plasticity (synaptic) | Inhibitors | FTY720 | TrkB receptors | Alzheimers disease | Hippocampus
Journal Article
Journal Article
Metabolic Brain Disease, ISSN 0885-7490, 12/2017, Volume 32, Issue 6, pp. 2045 - 2061
Amyloid beta (Aβ) peptide deposition is the primary cause of neurodegeneration in Alzheimer’s disease (AD) pathogenesis. Several reports point towards the role... 
Biochemistry, general | Chlorpyrifos | Neurosciences | Alzheimer’s disease | Amyloid beta protein | APE1 | Metabolic Diseases | Oncology | Monocrotophos | Neurology | Biomedicine | Nrf2 | Curcumin | APURINIC/APYRIMIDINIC ENDONUCLEASE-1 | OXIDATIVE STRESS | ALZHEIMERS-DISEASE | TRANSGENIC MOUSE | MITOCHONDRIA-MEDIATED APOPTOSIS | NEUROBLASTOMA-CELLS | NEUROSCIENCES | NEURODEGENERATIVE DISEASES | IN-VITRO | BASE EXCISION-REPAIR | ENDOCRINOLOGY & METABOLISM | NITRIC-OXIDE | Alzheimer's disease | Cell Survival - drug effects | Reactive Oxygen Species - metabolism | Amyloid beta-Peptides - pharmacology | Humans | Curcumin - pharmacology | Peptide Fragments - pharmacology | Monocrotophos - pharmacology | Organophosphates - pharmacology | Up-Regulation - drug effects | Neuroprotective Agents - pharmacology | DNA-(Apurinic or Apyrimidinic Site) Lyase - metabolism | Signal Transduction - drug effects | NF-E2-Related Factor 2 - metabolism | Cell Line, Tumor | Chlorpyrifos - pharmacology | Neurons - metabolism | Oxidative Stress - drug effects | Neurons - drug effects | Enzymes | Enzyme inhibitors | Nitric oxide | Pesticides | Oxidative stress | DNA polymerase | Pathogenesis | DNA damage | Confocal | DNA repair | Base excision repair | Antioxidants | Proteins | Neurotoxicity | Agrochemicals | Neurodegeneration | Organophosphorus pesticides | Pretreatment | Repair | Deoxyribonucleic acid--DNA | Cell survival | Neurodegenerative diseases | Gene expression | Survival | Nitric-oxide synthase | Polymerase | Poly(ADP-ribose) Polymerase 1 | Cell lines | β-Amyloid | DNA-directed DNA polymerase | Index Medicus
Journal Article