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The Journal of clinical investigation, ISSN 0021-9738, 05/2012, Volume 122, Issue 5, pp. 1791 - 1802
Journal Article
Cell (Cambridge), ISSN 0092-8674, 09/2007, Volume 130, Issue 5, pp. 811 - 823
Estrogen prevents osteoporotic bone loss by attenuating bone resorption; however, the molecular basis for this is unknown. Here, we report a critical role for... 
HUMDISEASE | DEVBIO | SIGNALING | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Fas Ligand Protein - metabolism | Male | fas Receptor - metabolism | Bone Remodeling - drug effects | Selective Estrogen Receptor Modulators - therapeutic use | Estrogen Receptor alpha - metabolism | Female | Integrases - metabolism | Mice, Inbred DBA | Cell Differentiation | Estradiol - metabolism | Osteoclasts - pathology | Bone Density | Cathepsin K | Ovariectomy | Signal Transduction | Bone Diseases, Metabolic - prevention & control | Mice, Inbred C57BL | Cells, Cultured | Mice, Transgenic | Osteoclasts - metabolism | Estrogen Receptor alpha - deficiency | Mice, Knockout | Bone Diseases, Metabolic - metabolism | Estrogen Receptor alpha - drug effects | Phenotype | Animals | Estrogen Receptor alpha - genetics | Cathepsins - genetics | Cathepsins - metabolism | Tamoxifen - therapeutic use | Tamoxifen - pharmacology | Mice | Bone Diseases, Metabolic - physiopathology | Fas Ligand Protein - genetics | Integrases - genetics | Selective Estrogen Receptor Modulators - pharmacology | Osteoclasts - drug effects | Apoptosis | Index Medicus | Cathepsins | Antigens, CD95 | Estradiol | Life Sciences | Estrogen Receptor alpha | Biochemistry, Molecular Biology | Tamoxifen | Bone Diseases, Metabolic | Integrases | Fas Ligand Protein | Selective Estrogen Receptor Modulators | Osteoclasts | Molecular biology | Bone Remodeling
Journal Article
Nature medicine, ISSN 1546-170X, 10/2014, Volume 20, Issue 11, pp. 1270 - 1278
Osteogenesis during bone modeling and remodeling is coupled with angiogenesis. A recent study showed that a specific vessel subtype, strongly positive for CD31... 
Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Cell Biology | Research & Experimental Medicine | Neovascularization, Physiologic - drug effects | Cell Count | Tartrate-Resistant Acid Phosphatase | Osteoclasts - secretion | Culture Media, Conditioned - pharmacology | Femur - diagnostic imaging | X-Ray Microtomography | Protease Inhibitors - pharmacology | Acid Phosphatase - metabolism | Mesenchymal Stromal Cells - cytology | Isoenzymes - metabolism | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Female | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Cathepsin K - antagonists & inhibitors | Mesenchymal Stromal Cells - drug effects | Ovariectomy | Endothelial Cells - metabolism | Femur - metabolism | Focal Adhesion Protein-Tyrosine Kinases - metabolism | Mice, Inbred C57BL | Osteogenesis - drug effects | Mesenchymal Stromal Cells - metabolism | Femur - drug effects | Cell Movement - drug effects | Animals | Cathepsin K - metabolism | Endothelial Cells - cytology | Proto-Oncogene Proteins c-sis - secretion | Osteoclasts - enzymology | Osteoclasts - drug effects | Endothelial Cells - drug effects | Platelet-derived growth factor | Growth | Analysis | Physiological aspects | Bones | Research | Osteoclasts (Biology) | Homeostasis | Angiogenesis | Cell growth | Blood platelets | Index Medicus
Journal Article
Nature medicine, ISSN 1546-170X, 10/2014, Volume 20, Issue 11, pp. 1279 - 1288
The WNT16 locus is a major determinant of cortical bone thickness and nonvertebral fracture risk in humans. The disability, mortality and costs caused by... 
Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Cell Biology | Research & Experimental Medicine | Osteocytes - drug effects | Humans | Wnt Proteins - deficiency | RNA, Messenger - metabolism | Wnt Proteins - metabolism | Cell Lineage - drug effects | Fractures, Bone - genetics | Cell Differentiation - genetics | RANK Ligand - pharmacology | Wnt Proteins - genetics | Skull - pathology | Gene Deletion | Organ Size - genetics | Osteogenesis - genetics | Cell Line | Osteoclasts - pathology | Disease Susceptibility | Osteocytes - metabolism | Mice, Inbred C57BL | Osteoblasts - drug effects | Osteogenesis - drug effects | RNA, Messenger - genetics | Cells, Cultured | Signal Transduction - genetics | Osteoclasts - metabolism | Fractures, Bone - metabolism | Aging - pathology | Gene Expression Regulation - drug effects | Osteoblasts - pathology | Organ Size - drug effects | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Osteocytes - pathology | Fractures, Bone - pathology | Osteoprotegerin - metabolism | Osteoblasts - metabolism | Fractures, Bone - prevention & control | Osteoclasts - drug effects | Physiological aspects | Fractures | Research | Wnt proteins | Analysis | Osteoclasts (Biology) | Osteoporosis | Signal transduction | Bones | Glycoproteins | Porosity | Index Medicus | Clinical Medicine | Endokrinologi och diabetes | Klinisk medicin | Endocrinology and Diabetes
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 0027-8424, 4/2013, Volume 110, Issue 18, pp. 7294 - 7299
Despite recent insights gained from the effects of targeted deletion of the Finkel-Biskis-Jinkins osteosarcoma oncogene (c-fos). Spleen focus-forming virus... 
Transcription factors | Osteopetrosis | Genes | Cell lines | Bones | Osteoclasts | Giant cells | Gene expression regulation | Macrophages | Binding sites | Cell lineage commitment | Osteoclast differentiation | Bone homeostasis | Osteopetrosis phenotype | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Osteopetrosis - diagnostic imaging | CCAAT-Enhancer-Binding Protein-alpha - metabolism | Femur - pathology | Osteopetrosis - physiopathology | Monocytes - cytology | Molecular Sequence Data | CCAAT-Enhancer-Binding Protein-alpha - deficiency | Femur - diagnostic imaging | Cell Lineage - drug effects | Promoter Regions, Genetic - genetics | Tibia - pathology | Cell Differentiation - genetics | RANK Ligand - pharmacology | Base Sequence | Cell Lineage - genetics | Osteogenesis - genetics | Tibia - physiopathology | Animals, Newborn | Biomarkers - metabolism | Cell Line | Osteoclasts - pathology | Osteopetrosis - pathology | Osteogenesis - drug effects | Proto-Oncogene Proteins c-fos - metabolism | Tibia - diagnostic imaging | Femur - drug effects | DNA - metabolism | Macrophages - cytology | Femur - physiopathology | Tibia - drug effects | Osteoclasts - metabolism | Regulatory Sequences, Nucleic Acid - genetics | Mice, Knockout | Gene Expression Regulation - drug effects | Radiography | Macrophages - metabolism | Organ Size - drug effects | Phenotype | Animals | Cathepsin K - metabolism | Cell Differentiation - drug effects | Macrophages - drug effects | Mice | Osteoclasts - enzymology | Osteoclasts - drug effects | Index Medicus | gene expression regulation | Biological Sciences | bone homeostasis | cell lineage commitment | osteoclast differentiation | osteopetrosis phenotype
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 0027-8424, 8/2008, Volume 105, Issue 33, pp. 11691 - 11696
Journal Article
Journal of bone and mineral research, ISSN 0884-0431, 12/2017, Volume 32, Issue 12, pp. 2415 - 2430
Journal Article