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CANCER EPIDEMIOLOGY, ISSN 1877-7821, 04/2013, Volume 37, Issue 2, pp. 172 - 178
Journal Article
Cancer Epidemiology, ISSN 1877-7821, 2012, Volume 37, Issue 2, pp. 172 - 178
Abstract Background : It is well known that both heat shock protein (HSP) and Toll-like receptor (TLR)3 agonist polyinosinic:polycytidylic acid (poly(I:C)) are... 
Hematology, Oncology and Palliative Medicine | Internal Medicine | Oxygen-regulated protein 150 | Polyinosinic:polycytidylic acid | Epitope | E749–57 peptide | Cytotoxic T cells | Adjuvant | HPV16 | peptide | Proteins | Human papillomavirus | Immunology | Peptides | Immune system | Tumors
Journal Article
Journal Article
Journal of Cellular Biochemistry, ISSN 0730-2312, 08/2018, Volume 119, Issue 8, pp. 6795 - 6805
Chitinase 3‐like protein 1 (CHI3L1) is a novel biomarker of systemic inflammation. However, the effects of CHI3L1 on the progression of atherosclerosis remain... 
apoptosis | chitinase 3‐like protein 1 | peroxisome proliferator‐activated receptor delta | inflammation | HUVECs | oxygen‐regulated protein 150 | chitinase 3-like protein 1 | oxygen-regulated protein 150 | peroxisome proliferator-activated receptor delta | FOLLISTATIN-LIKE 1 | MYOCARDIAL-INFARCTION | HUMAN CARTILAGE GP-39 | BIOCHEMISTRY & MOLECULAR BIOLOGY | LDLR-/-MICE | ENDOPLASMIC-RETICULUM STRESS | ADHESION MOLECULES | ACUTE CORONARY SYNDROME | CELL BIOLOGY | REDUCES ATHEROSCLEROSIS | ENDOTHELIAL-CELLS | SMOOTH-MUSCLE CELLS | Human Umbilical Vein Endothelial Cells | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Atherosclerosis - pathology | Lipopolysaccharides - toxicity | Atherosclerosis - chemically induced | Atherosclerosis - genetics | Humans | Tumor Necrosis Factor-alpha - genetics | THP-1 Cells | Chemokine CCL2 - genetics | NF-kappa B - metabolism | Chitinase-3-Like Protein 1 - genetics | Atherosclerosis - metabolism | Chitinase-3-Like Protein 1 - metabolism | Inflammation - metabolism | NF-kappa B - genetics | Endoplasmic Reticulum Stress | Inflammation - genetics | Chemokine CCL2 - metabolism | PPAR delta - metabolism | PPAR delta - genetics | Stresses | Chitinase | Phosphorylation | Cytokines | Secretion | Inflammation | siRNA | Tumor necrosis factor-α | Ribonucleic acid--RNA | Adhesion | Molecular chains | Stress | Endothelium | Cell adhesion molecules | Lipopolysaccharides | Proteins | Arteriosclerosis | Atherosclerosis | Biomarkers | Endoplasmic reticulum | Nuclear reactions | Apoptosis
Journal Article
Acta Ophthalmologica, ISSN 1755-375X, 06/2018, Volume 96, Issue 4, pp. e460 - e467
Purpose 150‐kDa oxygen‐regulated protein (ORP150), a member of heat‐shock protein family located in endoplasmic reticulum (ER), has a critical role in... 
150‐kDa oxygen‐regulated protein | proliferative diabetic retinopathy | angiogenesis | vascular endothelial growth factor | 150-kDa oxygen-regulated protein | FACTOR VEGF | CELLS | OXIDATIVE STRESS | ORP150 | HYPOXIA | TOTAL ANTIOXIDANT CAPACITY | PATHOGENESIS | FLUID | OPHTHALMOLOGY | EXPRESSION | Immunohistochemistry | Vascular Endothelial Growth Factor A - biosynthesis | Retina - metabolism | Humans | Male | Diabetic Retinopathy - pathology | Vascular Endothelial Growth Factor A - genetics | RNA - genetics | Vitreous Body - metabolism | Diabetes Mellitus, Experimental | HSP70 Heat-Shock Proteins - biosynthesis | Enzyme-Linked Immunosorbent Assay | Cells, Cultured | Gene Expression Regulation | Rats | HSP70 Heat-Shock Proteins - genetics | Rats, Sprague-Dawley | Blotting, Western | Diabetic Retinopathy - metabolism | Diabetic Retinopathy - genetics | Animals | Endothelium, Vascular - metabolism | Vitreous Body - pathology | Endothelium, Vascular - pathology | Retina - pathology | Antioxidants | Diabetic retinopathy | Enzymes | Diabetics | Interleukins | Analysis | Neovascularization | Ophthalmology | Vascular endothelial growth factor | Enzyme-linked immunosorbent assay | Cell proliferation | Membranes | Retinopathy | Interleukin | Retina | Vascularization | Proteins | Angiogenesis | Rodents | Tumor necrosis factor-TNF | Oxygen | Cytokines | Secretion | Diabetes mellitus | Blood vessels | Inflammation | Patients | Endothelial cells | Tumor necrosis factor | Stromal cells | Sampling methods | Microvasculature | Diabetes | Endoplasmic reticulum | Tumors
Journal Article
International Journal of Clinical and Experimental Medicine, ISSN 1940-5901, 10/2016, Volume 9, Issue 10, pp. 19512 - 19520
To clarify whether sexual arousal could be enhanced by hypoxia, and to explain the pathogenesis of autoerotic death (AED)/autoerotic asphyxiation (AEA), we... 
Hypoxia | Oxytocin | Autoerotic asphyxia (AEA) | Sexual stimulation | Oxygen-regulated protein 150 (ORP150) | MEDICINE, RESEARCH & EXPERIMENTAL | hypoxia | oxygen-regulated protein 150 (ORP150) | BEHAVIOR | autoerotic asphyxia (AEA) | sexual stimulation | HORMONE | ASPHYXIA | AUTOEROTIC DEATHS | VASOPRESSIN | COPEPTIN | EXPRESSION | AXIS
Journal Article
Molecular and Cellular Biochemistry, ISSN 0300-8177, 7/2009, Volume 327, Issue 1, pp. 211 - 218
Journal Article
Journal Article
Biochemical Journal, ISSN 0264-6021, 03/2007, Volume 402, Issue 3, pp. 581 - 589
A beta (amyloid-beta peptides) generated by proteolysis of APP (beta-amyloid precursor protein), play an important role in the pathogenesis of AD (Alzheimer's... 
Amyloid-β peptides (Aβ) | Endoplasmic reticulum (ER) chaperone | β-amyloid precursor protein (APP) | Glucose-regulated protein 78 (GRP78) | Protein maturation | GRP78 PROMOTER | protein maturation | ACTIVATION | ALZHEIMERS-DISEASE | ER STRESS | BIOCHEMISTRY & MOLECULAR BIOLOGY | beta-amyloid precursor protein (APP) | CELL-DEATH | GLUCOSE-REGULATED PROTEINS | amyloid-beta peptides (A beta) | UNFOLDED PROTEIN RESPONSE | endoplasmic reticulum (ER) chaperone | NEURONAL DEATH | PRECURSOR PROTEIN | GAMMA-SECRETASE | glucose-regulated protein 78 (GRP78) | Cell Line | RNA, Small Interfering - genetics | Up-Regulation | Molecular Chaperones - metabolism | Amyloid beta-Peptides - biosynthesis | Humans | RNA, Messenger - genetics | Molecular Chaperones - genetics | Activating Transcription Factor 4 - genetics | Endoplasmic Reticulum - metabolism | Mice, Transgenic | Activating Transcription Factor 6 - genetics | Hippocampus - metabolism | Animals | Protein Binding | Mice | imAPP, immature APP | ATF6, activating transcription factor 6 | CTFα, C-terminal fragment α | PS1, presenilin 1 | DAPT, N-[N-(3,5-difluorophenacetyl-L-alanyl)]-S-phenylglycine t-butyl ester | DMEM, Dulbecco's modified Eagle's medium | APPwt, wild-type APP | ERAD, ER-associated degradation | RT, reverse transcriptase | mAPP, mature APP | CRT, calreticulin | Co-IP, co-immunoprecipitation | HEK-293, human embryonic kidney 293 | ERdj4ΔJ, J domain-deleted ERdj4 | FAD, familial AD | amyloid-β peptides (Aβ) | IRE1, protein-kinase and site-specific endoribonuclease | GRP, glucose-regulated protein | EDEM, endoplasmic reticulum degradation-enhancing α-mannosidase I-like protein | AICD, β-amyloid precursor protein intracellular domain | siRNA, small interfering RNA | Aβ, amyloid-β peptides | APPsw, Swedish mutant of APP | APP, β-amyloid precursor protein | AD, Alzheimer's disease | CNX, calnexin | sELISA, sandwich ELISA | HSP, heat shock protein | PERK, protein kinase R-like ER kinase | ORP150, 150 kDa oxygen-regulated protein | eIF2α, eukaryotic initiation factor-2α | ER, endoplasmic reticulum
Journal Article