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Diabetes, ISSN 0012-1797, 02/2014, Volume 63, Issue 2, pp. 514 - 525
The number and activity of brown adipocytes are linked to the ability of mammals to resist body fat accumulation. In some conditions, certain white adipose... 
OBESITY | GENE | ENDOCRINOLOGY & METABOLISM | MUSCLE | RECEPTOR | PROLIFERATION | DIFFERENTIATION | EXPRESSION | FAT-CELL | EXERCISE | ADIPOSE-TISSUE | MAP Kinase Signaling System - physiology | Nitriles - pharmacology | Adipose Tissue, White - metabolism | Caenorhabditis elegans Proteins - metabolism | Adipocytes - cytology | Male | Adipocytes - drug effects | Extracellular Signal-Regulated MAP Kinases - metabolism | Recombinant Proteins | Extracellular Signal-Regulated MAP Kinases - genetics | Pichia - metabolism | Membrane Transport Proteins - genetics | Dietary Fats | Membrane Transport Proteins - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Butadienes - pharmacology | Fibronectins - pharmacology | Fibronectins - administration & dosage | Mice, Inbred C57BL | Rats | p38 Mitogen-Activated Protein Kinases - genetics | Imidazoles - pharmacology | 3T3-L1 Cells | Rats, Sprague-Dawley | Animals | Adipocytes - metabolism | Pichia - genetics | Protein Binding | Adipose Tissue, Brown - drug effects | Adipose Tissue, Brown - metabolism | Fibronectins - genetics | Mice | Pyridines - pharmacology | Mutation | Mitochondrial Uncoupling Proteins | Caenorhabditis elegans Proteins - genetics | Adipose Tissue, White - drug effects | Physiological research | Cellular signal transduction | Research | Identification and classification | Membrane proteins | Proteins | Homeostasis | Diabetes | Kinases | Genes | Cells | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Circulation Research, ISSN 0009-7330, 02/2014, Volume 114, Issue 4, pp. 616 - 625
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 09/2011, Volume 286, Issue 38, pp. 33335 - 33344
Journal Article
MOLECULAR AND CELLULAR BIOLOGY, ISSN 0270-7306, 08/2009, Volume 29, Issue 16, pp. 4341 - 4351
Activation of p38 mitogen-activated protein kinase (MAPK) plays an important role in the G(2)/M cell cycle arrest induced by DNA damage, but little is known... 
UV-IRRADIATION | CELLS | PHOSPHORYLATION | DIFFERENTIAL ACTIVATION | STABILITY | BIOCHEMISTRY & MOLECULAR BIOLOGY | DNA-DAMAGE | P21(WAF1/CIP1) EXPRESSION | S-PHASE | ATAXIA-TELANGIECTASIA | P53 | CELL BIOLOGY | ELAV-Like Protein 1 | Gamma Rays | RNA, Small Interfering - genetics | RNA-Binding Proteins - genetics | Phosphorylation | Humans | RNA, Messenger - metabolism | ELAV Proteins | Tumor Suppressor Protein p53 - genetics | DNA-Binding Proteins - metabolism | Cell Line - physiology | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Tumor Suppressor Proteins - genetics | Antigens, Surface - metabolism | Cell Cycle Proteins - genetics | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Protein-Serine-Threonine Kinases - metabolism | Tumor Suppressor Proteins - metabolism | RNA, Messenger - genetics | Antigens, Surface - genetics | Cell Cycle Proteins - metabolism | Protein-Serine-Threonine Kinases - genetics | Tumor Suppressor Protein p53 - metabolism | p38 Mitogen-Activated Protein Kinases - genetics | Ataxia Telangiectasia Mutated Proteins | DNA-Binding Proteins - genetics | G1 Phase - physiology | RNA Stability | Cell Line - radiation effects | Animals | S Phase - physiology | Mice | DNA Damage | Enzyme Activation | RNA-Binding Proteins - metabolism | RNA, Small Interfering - metabolism | Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 07/2010, Volume 120, Issue 7, pp. 2457 - 2473
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 12/2012, Volume 32, Issue 49, pp. 17714 - 17724
Journal Article
Journal Article
Journal Article
Journal Article
Cancer Research, ISSN 0008-5472, 01/2010, Volume 70, Issue 2, pp. 832 - 841
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 11/2009, Volume 29, Issue 47, pp. 14741 - 14751
Alzheimer's disease (AD) is characterized by memory impairment, neurochemically by accumulation of beta-amyloid peptide (namely Abeta(1-42)) and... 
Memory Disorders - physiopathology | Presynaptic Terminals - pathology | Rats, Wistar | Peptide Fragments - toxicity | Nerve Degeneration - physiopathology | Male | Adenosine A2 Receptor Antagonists | Neuroprotective Agents - pharmacology | Nerve Degeneration - prevention & control | Caffeine - pharmacology | Receptor, Adenosine A2A - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | Phosphorylation - drug effects | Cell Survival - physiology | Cell Survival - drug effects | Alzheimer Disease - physiopathology | Maze Learning - physiology | Presynaptic Terminals - drug effects | Amyloid beta-Peptides - toxicity | Mice, Inbred C57BL | Cells, Cultured | Alzheimer Disease - drug therapy | Phosphodiesterase Inhibitors - pharmacology | Rats | Pyrimidines - pharmacology | Receptor, Adenosine A2A - metabolism | Maze Learning - drug effects | Down-Regulation - genetics | Mice, Knockout | Triazoles - pharmacology | Amyloid beta-Peptides - antagonists & inhibitors | Synaptic Membranes - drug effects | p38 Mitogen-Activated Protein Kinases - drug effects | Animals | Memory Disorders - drug therapy | Peptide Fragments - antagonists & inhibitors | Alzheimer Disease - metabolism | Presynaptic Terminals - metabolism | Mice | Synaptic Membranes - metabolism | Nerve Degeneration - drug therapy | Memory Disorders - chemically induced | Index Medicus
Journal Article