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Journal Article
Journal Article
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 01/2013, Volume 430, Issue 1, pp. 208 - 212
► EGCG down-regulated UHRF1 and DNMT1 expressions in jurkat cells. ► Down-regulation of UHRF1 enhanced expression of p16 and p73. ► UHRF1 over-expression... 
P16INK4A | DNA methyltransferase 1 | Ubiquitin-like containing PHD and Ring finger 1 | Epigallocatechin-3-gallate | Tumor suppressor gene | CANCER-CELLS | HEMI-METHYLATED DNA | CCAAT-BINDING-PROTEIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRAIL-MEDIATED APOPTOSIS | P16(INK4A) | II-ALPHA EXPRESSION | BIOPHYSICS | (-)-EPIGALLOCATECHIN-3-GALLATE | SRA DOMAIN | JURKAT CELLS | SKIN PHOTOPROTECTION | ICBP90 | CCAAT-Enhancer-Binding Proteins - metabolism | Promoter Regions, Genetic | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | DNA (Cytosine-5-)-Methyltransferase 1 | Jurkat Cells | Down-Regulation | Humans | Cyclin-Dependent Kinase Inhibitor p16 - genetics | Vascular Endothelial Growth Factor A - metabolism | DNA (Cytosine-5-)-Methyltransferases - antagonists & inhibitors | DNA-Binding Proteins - genetics | Genes, Tumor Suppressor - drug effects | DNA (Cytosine-5-)-Methyltransferases - metabolism | Tumor Protein p73 | CCAAT-Enhancer-Binding Proteins - antagonists & inhibitors | Tumor Suppressor Proteins - genetics | Catechin - analogs & derivatives | Gene Expression Regulation, Neoplastic - drug effects | Catechin - pharmacology | Cell Cycle - drug effects | Nuclear Proteins - genetics | Catechin | Methyltransferases | Gene expression | DNA | Tumors | Drugs | Demethylation | epigallocatechin-3-gallate | Reactive oxygen species | Overexpression | DNMT1 protein | INK4a protein | DNA methyltransferase | Tumor suppressor genes | Promoters | p16 protein | Apoptosis | Life Sciences | Cellular Biology | Biochemistry, Molecular Biology
Journal Article
Cell Cycle, ISSN 1538-4101, 03/2018, Volume 17, Issue 5, pp. 557 - 567
P53 tumor suppressor gene mutations occur in the majority of human cancers and contribute to tumor development, progression and therapy resistance. Direct... 
ubiquitin proteasome system | apoptosis | autophagy | mutant p53 degradation | p53 pathway restoration | NOXA | CANCER-CELLS | DRUG | ACTIVATION | RESTORATION | OPPORTUNITIES | P73 | CELL BIOLOGY | MUTANT P53 | GAIN | ATG5
Journal Article
Journal of Clinical Oncology, ISSN 0732-183X, 06/2005, Volume 23, Issue 17, pp. 3886 - 3896
Journal Article
JOURNAL OF MOLECULAR CELL BIOLOGY, ISSN 1674-2788, 03/2019, Volume 11, Issue 3, pp. 212 - 223
Activation of the tumour suppressor p53 upon cellular stress can induce a number of different cellular processes. The diverse actions of these processes are... 
TARGET | APOPTOSIS | CELLS | tumour suppression | cell death | cell cycle arrest | P73 | P21 | p53 | CELL BIOLOGY | senescence | ARREST | MUTATION | ABSENCE | PUMA | GAIN
Journal Article
Human Molecular Genetics, ISSN 0964-6906, 06/2013, Volume 22, Issue 11, pp. 2263 - 2272
Androgen receptor (AR) expression by immunohistochemistry correlates with better prognosis and survival among breast cancer patients. We and others have shown... 
PROGNOSTIC FACTORS | PROSTATE | KILLIN | GENE | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENETICS & HEREDITY | ESTROGEN | ALPHA | CELL-PROLIFERATION | 5-ALPHA-DIHYDROTESTOSTERONE | IDENTIFICATION | EXPRESSION |