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The Plant Cell, ISSN 1040-4651, 10/2011, Volume 23, Issue 10, pp. 3824 - 3841
Necrotrophic pathogens are important plant pathogens that cause many devastating plant diseases. Despite their impact, our understanding of the plant defense... 
Proteins | Pathogens | Yeasts | RNA | DNA | Genes | Infections | Plants | Transgenic plants | Plant cells | MICROBIAL PATHOGENS | BOTRYTIS-CINEREA | BIOCHEMISTRY & MOLECULAR BIOLOGY | NECROTROPHIC FUNGAL PATHOGENS | RESPONSE PATHWAYS | PLANT SCIENCES | CELL BIOLOGY | SALICYLIC-ACID | TOMATO SEEDLINGS | ALTERNARIA-SOLANI | GENE-EXPRESSION | RESISTANCE | BASAL DEFENSE | Sigma Factor - metabolism | Arabidopsis - physiology | Plant Diseases - immunology | Arabidopsis - immunology | Cytochrome P-450 Enzyme System - metabolism | Molecular Sequence Data | Plant Diseases - microbiology | Recombinant Fusion Proteins | Arabidopsis Proteins - metabolism | Botrytis - pathogenicity | Cell Nucleus - metabolism | Plants, Genetically Modified | Gene Expression Regulation, Plant | Botrytis - immunology | Sigma Factor - genetics | DNA, Plant - genetics | Protein Structure, Tertiary | Amino Acid Sequence | Arabidopsis Proteins - genetics | Gene Silencing | Transcription Factors - genetics | Protein Interaction Mapping | Arabidopsis - genetics | Transcription Factors - metabolism | Carrier Proteins - genetics | Sequence Alignment | Carrier Proteins - metabolism | Cytochrome P-450 Enzyme System - genetics | Mutagenesis, Insertional | Arabidopsis thaliana | Transcription factors | Plant defenses | Plant genetics | Physiological aspects | Genetic aspects | Research | Binding proteins
Journal Article
Nature Immunology, ISSN 1529-2908, 05/2010, Volume 11, Issue 5, pp. 411 - 418
Sensors of pathogens, such as Toll-like receptors (TLRs), detect microbes to activate transcriptional programs that orchestrate adaptive responses to specific... 
UNFOLDED PROTEIN RESPONSE | PATHWAY | ER STRESS | HOST-DEFENSE | ENDOPLASMIC-RETICULUM STRESS | FRANCISELLA-TULARENSIS INFECTION | IMMUNOLOGY | LIVE VACCINE STRAIN | INFLAMMATORY RESPONSE | NEGATIVE REGULATOR | SYSTEMS BIOLOGY | RNA, Small Interfering - genetics | Endoribonucleases - genetics | Tularemia - metabolism | Toll-Like Receptor 2 - genetics | Membrane Glycoproteins - metabolism | Lipopeptides - pharmacology | Transcriptional Activation - drug effects | NADPH Oxidases - metabolism | Tularemia - genetics | Protein Splicing - genetics | Transcriptional Activation - immunology | X-Box Binding Protein 1 | Protein Splicing - drug effects | NADPH Oxidases - genetics | Stress, Physiological - drug effects | TNF Receptor-Associated Factor 6 - genetics | Transcription Factor CHOP - biosynthesis | Transcription Factors - immunology | Cytokines - genetics | Protein-Serine-Threonine Kinases - metabolism | DNA-Binding Proteins - immunology | Endoribonucleases - metabolism | Macrophages - pathology | Stress, Physiological - genetics | Myeloid Differentiation Factor 88 - genetics | Toll-Like Receptor 4 - genetics | Signal Transduction - genetics | Toll-Like Receptor 4 - immunology | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Macrophages - metabolism | Signal Transduction - drug effects | Tunicamycin - pharmacology | Lipopolysaccharides - pharmacology | Toll-Like Receptor 2 - immunology | Mice | Stress, Physiological - immunology | Endoribonucleases - immunology | Transcription Factor CHOP - genetics | NADPH Oxidases - immunology | DNA-Binding Proteins - metabolism | Signal Transduction - immunology | Macrophages - virology | Mice, Mutant Strains | Tularemia - immunology | Membrane Glycoproteins - immunology | Macrophages - immunology | Cytokines - immunology | Cell Line | Francisella tularensis - immunology | Protein Splicing - immunology | Protein-Serine-Threonine Kinases - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Immunity, Innate | NADPH Oxidase 2 | Regulatory Factor X Transcription Factors | Mice, Inbred C3H | Membrane Glycoproteins - genetics | Transcription Factors - metabolism | Animals | TNF Receptor-Associated Factor 6 - metabolism | Macrophages - drug effects | Protein-Serine-Threonine Kinases - immunology | Francisella tularensis - pathogenicity | Myeloid Differentiation Factor 88 - metabolism | Cytokines - biosynthesis | Cell receptors | Transcription factors | Immune response | Physiological aspects | Genetic aspects | Research | Health aspects | Francisella tularensis
Journal Article
APMIS, ISSN 0903-4641, 10/2015, Volume 123, Issue 10, pp. 823 - 831
Journal Article
The Plant Cell, ISSN 1040-4651, 3/2011, Volume 23, Issue 3, pp. 1153 - 1170
Journal Article
New Phytologist, ISSN 0028-646X, 07/2017, Volume 215, Issue 1, pp. 338 - 350
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 04/2014, Volume 124, Issue 4, pp. 1537 - 1551
Development of host protective immunity against Mycobacterium tuberculosis infection is critically dependent on the inflammatory cytokine TNF. TNF signals... 
MEDICINE, RESEARCH & EXPERIMENTAL | TUMOR-NECROSIS-FACTOR | BACILLUS-CALMETTE-GUERIN | TRANSMEMBRANE TNF | DEFICIENT MICE | BACTERIAL-INFECTION | HUMAN ALVEOLAR MACROPHAGES | DENDRITIC CELL MATURATION | FACTOR RECEPTOR | INDUCED GRANULOMA-FORMATION | FACTOR-ALPHA | Tumor Necrosis Factor-alpha - metabolism | Tuberculosis, Pulmonary - microbiology | Tumor Necrosis Factor Decoy Receptors - genetics | Receptors, Tumor Necrosis Factor, Type I - immunology | Tumor Necrosis Factor Decoy Receptors - immunology | Dendritic Cells - immunology | Mycobacterium tuberculosis - immunology | Male | Receptors, Tumor Necrosis Factor, Type II - immunology | Host-Pathogen Interactions - immunology | CD4-Positive T-Lymphocytes - immunology | Mycobacterium tuberculosis - pathogenicity | Receptors, Tumor Necrosis Factor, Type I - deficiency | Signal Transduction - immunology | Female | Interleukin-12 Subunit p40 - metabolism | Tumor Necrosis Factor Decoy Receptors - deficiency | Lymphocyte Activation | Mice, Inbred C57BL | Solubility | Tuberculosis, Pulmonary - pathology | Receptors, Tumor Necrosis Factor, Type I - genetics | Receptors, Tumor Necrosis Factor, Type II - genetics | Mice, Knockout | Receptors, Tumor Necrosis Factor, Type II - deficiency | Granuloma - pathology | Tuberculosis, Pulmonary - immunology | Animals | Mice | Prevention | Care and treatment | Immune response | Mycobacterium tuberculosis | Tumor necrosis factor | Physiological aspects | Drug targeting | Regulation | Research | Studies | Tuberculosis | Cytokines | Rodents | Mortality | Ligands | Infections
Journal Article
Nature, ISSN 0028-0836, 05/2008, Volume 453, Issue 7195, pp. 620 - 625
Humans are colonized by multitudes of commensal organisms representing members of five of the six kingdoms of life; however, our gastrointestinal tract... 
HOST IMMUNE-SYSTEM | SUPPRESS | BACTERIA | CROHNS-DISEASE | GUT MICROBIOME | MULTIDISCIPLINARY SCIENCES | ULCERATIVE-COLITIS | REGULATORY T-CELLS | HELICOBACTER-HEPATICUS | INTERLEUKIN-10-DEFICIENT MICE | BOWEL-DISEASE | Inflammation - chemically induced | Intestines - drug effects | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Inflammatory Bowel Diseases - immunology | Symbiosis - physiology | Intestines - immunology | CD4-Positive T-Lymphocytes - immunology | Polysaccharides, Bacterial - genetics | Colitis - chemically induced | Helicobacter hepaticus - pathogenicity | Inflammatory Bowel Diseases - pathology | Interleukin-10 - metabolism | Colitis - immunology | Disease Models, Animal | Polysaccharides, Bacterial - pharmacology | Bacteroides fragilis - immunology | Inflammation - microbiology | Inflammatory Bowel Diseases - prevention & control | Intestines - pathology | Bacteroides fragilis - physiology | CD4-Positive T-Lymphocytes - metabolism | Polysaccharides, Bacterial - metabolism | Polysaccharides, Bacterial - immunology | Interleukin-17 - metabolism | Animals | Colitis - microbiology | Intestines - microbiology | Interleukin-10 - genetics | Germ-Free Life | Bacteroides fragilis - genetics | Inflammatory Bowel Diseases - microbiology | Interleukin-10 - biosynthesis | Inflammation - prevention & control | Mice | Colitis - prevention & control | Tumor Necrosis Factor-alpha - biosynthesis | CD4-Positive T-Lymphocytes - drug effects | Prevention | Physiological aspects | Development and progression | Inflammatory bowel diseases | Pathogenic microorganisms | Research | Inflammatory bowel disease | Molecules | Cytokines | Clinical trials | Bacteria | Small intestine | Cells | Crohns disease | Index Medicus
Journal Article
AIDS research and human retroviruses, 12/2012, Volume 28, Issue 12, pp. 1543 - 1551
Human immunodeficiency virus type 1 (HIV-1) requires the cellular transcription factor core binding factor subunit β (CBFβ) to stabilize its viral infectivity... 
Index Medicus | AIDS/HIV
Journal Article
Developmental and Comparative Immunology, ISSN 0145-305X, 2012, Volume 36, Issue 1, pp. 39 - 50
► TNF superfamily gene, TNFR superfamily gene and lipopolysaccharide-induced TNF-α factor gene were cloned. ► In S2 cells, LvTNFRSF localized to the membrane... 
LvLITAF | TNF–TNFR system | WSSV | LvTNFRSF | Litopenaeus vannamei | Vibrio alginolyticus | LvTNFSF | TNF-TNFR system | TOLL RECEPTOR | CRASSOSTREA-GIGAS | HOMOLOG | IMMUNOLOGY | WSSV INFECTION | DROSOPHILA | SHRIMP | ZOOLOGY | ANTIMICROBIAL PEPTIDE GENES | HOST-DEFENSE | TRANSCRIPTION FACTOR | EIGER | Tumor Necrosis Factor-alpha - metabolism | Species Specificity | Humans | Tumor Necrosis Factor-alpha - genetics | Molecular Sequence Data | Gene Expression Profiling | Transcriptional Activation - immunology | White spot syndrome virus 1 - pathogenicity | Vibrio alginolyticus - pathogenicity | Cloning, Molecular | Tumor Necrosis Factor-alpha - immunology | Transcription Factors - immunology | White spot syndrome virus 1 - immunology | Receptors, Tumor Necrosis Factor - genetics | Candida albicans - pathogenicity | Multigene Family - genetics | Receptors, Tumor Necrosis Factor - metabolism | Amino Acid Sequence | Cell Line | Vibrio alginolyticus - immunology | Infection - immunology | Transcription Factors - genetics | Candida albicans - immunology | Pectinidae | Staphylococcus aureus - pathogenicity | Transcription Factors - metabolism | Infection - genetics | Infection - metabolism | Sequence Alignment | Animals | Staphylococcus aureus - immunology | Receptors, Tumor Necrosis Factor - immunology | Drosophila melanogaster | Immunity - genetics | Evolution, Molecular | Penaeidae - immunology
Journal Article