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Clinical Biochemistry, ISSN 0009-9120, 10/2019, Volume 72, pp. 71 - 80
Altered amyloid metabolism and mitochondrial dysfunction play key roles in the development of Alzheimer's disease (AD). We asked whether an association exists... 
Plasma | Mitochondria | Platelet | Amyloid beta | Alzheimer's disease | CASCADE HYPOTHESIS | DIAGNOSIS | OXIDATIVE STRESS | PROTEIN | BIOMARKERS | VASCULAR DEMENTIA | CONTROL MECHANISMS | A-BETA | DYSFUNCTION | MEDICAL LABORATORY TECHNOLOGY | BLOOD | Care and treatment | Amyloid beta-protein
Journal Article
Journal Article
Experimental Gerontology, ISSN 0531-5565, 09/2019, p. 110737
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2011, Volume 6, Issue 12, p. e28263
Journal Article
Journal of Alzheimer's Disease, ISSN 1387-2877, 2011, Volume 26, Issue 2, pp. 365 - 375
Plasma amyloid-beta (A beta) levels have been proposed as biomarkers of Alzheimer's disease (AD), but studies have produced inconsistent results. We present a... 
amyloid-β | meta-analysis | plasma | Alzheimer's disease | PROTEIN | DEMENTIA | OLDER-ADULTS | LATE-LIFE | FOLLOW-UP | RISK | MILD COGNITIVE IMPAIRMENT | NEUROSCIENCES | amyloid-beta | CEREBROSPINAL-FLUID BIOMARKERS | MOUSE MODEL | CSF | Humans | Middle Aged | Aged, 80 and over | Female | Male | Aged | Biomarkers - blood | Amyloid beta-Peptides - blood | Disease Progression | Alzheimer Disease - blood
Journal Article
Journal of Alzheimer's Disease, ISSN 1387-2877, 2009, Volume 17, Issue 3, pp. 681 - 697
Journal Article
Journal of Alzheimer's Disease, ISSN 1387-2877, 2017, Volume 60, Issue 3, pp. 977 - 987
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2011, Volume 6, Issue 12, p. e29480
Journal Article
by An, SSA and Lee, BS and Yu, JS and Lim, K and Kim, GJ and Lee, R and Kim, S and Kang, S and Park, YH and Wang, MJ and Yang, YS and Youn, YC
ALZHEIMERS RESEARCH & THERAPY, ISSN 1758-9193, 10/2017, Volume 9
Background: A reliable blood-based assay is required to properly diagnose and monitor Alzheimer's disease (AD). Many attempts have been made to develop such a... 
Synthetic amyloid-beta | EARLY-DIAGNOSIS | ALZHEIMERS-DISEASE | MILD COGNITIVE IMPAIRMENT | SOLUBLE-PROTEIN OLIGOMERS | NEUROSCIENCES | CLINICAL NEUROLOGY | Oligomers | BIOMARKERS | PEPTIDES | Blood biomarker | URINE | Multimer detection system | Alzheimer's disease | BRAIN | Amyloid-beta | ELISA | Plasma test | NEURONAL THREAD PROTEIN
Journal Article
Current Pharmaceutical Design, ISSN 1381-6128, 2014, Volume 20, Issue 15, pp. 2547 - 2554
Journal Article
Journal of Alzheimer's Disease, ISSN 1387-2877, 08/2015, Volume 47, Issue 4, pp. 1069 - 1078
Journal Article
Alzheimer's Research and Therapy, ISSN 1758-9193, 06/2018, Volume 10, Issue 1, pp. 63 - 8
Journal Article
Acta Neuropathologica, ISSN 0001-6322, 8/2017, Volume 134, Issue 2, pp. 207 - 220
Clearance of amyloid-beta (Aβ) from the brain is an important therapeutic strategy for Alzheimer’s disease (AD). Current studies mainly focus on the central... 
Pathology | Neurosciences | Peripheral clearance | Medicine & Public Health | Alzheimer’s disease | Neurodegeneration | Peritoneal dialysis | Amyloid-beta | CLEARANCE | NEPRILYSIN | PROTEIN | THERAPEUTIC STRATEGY | PATHOLOGY | BLOOD-BRAIN-BARRIER | NEUROSCIENCES | CLINICAL NEUROLOGY | IN-VIVO | DISEASE | A-BETA | HEMODIALYSIS | Alzheimer's disease | COGNITIVE DEFICITS | Amyloid Precursor Protein Secretases - blood | Humans | Amyloid beta-Peptides - blood | Case-Control Studies | Alzheimer Disease - pathology | Brain - metabolism | DNA-Binding Proteins - metabolism | Renal Insufficiency, Chronic - therapy | Excitatory Postsynaptic Potentials | Cognition Disorders - etiology | Aspartic Acid Endopeptidases - blood | Disease Models, Animal | Alzheimer Disease - therapy | Presenilin-1 - genetics | Mice, Transgenic | Nerve Tissue Proteins - metabolism | Renal Insufficiency, Chronic - blood | Phenotype | Amyloid beta-Protein Precursor - genetics | Animals | Mice | Peritoneal Dialysis - methods | Apoptosis - physiology | Cognition Disorders - therapy | Alzheimer Disease - genetics | Alzheimer Disease - blood | Advertising executives | Medical research | Chronic kidney failure | Neurons | Medicine, Experimental | Genetic aspects | Research | Health aspects | Potentiation | Phosphorylation | Neurodegenerative diseases | Long-term potentiation | Cognitive ability | Inflammation | Neuronal-glial interactions | Amyloid precursor protein | Microglia | Microdialysis | Tau protein | Presenilin 1 | Rodents | β-Amyloid | Plasma levels | Alzheimers disease | Phagocytosis | Age | Peritoneum | Kidney transplantation
Journal Article