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Kidney International, ISSN 0085-2538, 10/2012, Volume 82, Issue 7, pp. 771 - 789
Glomerular podocytes are highly specialized epithelial cells whose injury in glomerular diseases causes proteinuria. Since mitochondrial dysfunction is an... 
mitochondrial dysfunction | podocytes | SIRT1 | aldosterone | PGC-1a | OXIDATIVE STRESS | PGC-1 alpha | INDUCED APOPTOSIS | PGC-1-ALPHA | TRANSCRIPTIONAL COACTIVATOR | CELL APOPTOSIS | METABOLIC REGULATOR | UROLOGY & NEPHROLOGY | RAT PODOCYTES | FOCAL SEGMENTAL GLOMERULOSCLEROSIS | CALORIE RESTRICTION | Sirtuin 1 - metabolism | Apoptosis - drug effects | Receptors, Mineralocorticoid - metabolism | Podocytes - virology | Respiratory Syncytial Virus Infections - metabolism | Stilbenes - pharmacology | Aldosterone | Sirtuin 1 - genetics | Kidney Diseases - genetics | Receptors, Mineralocorticoid - drug effects | Transfection | RNA Interference | Time Factors | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Kidney Diseases - chemically induced | Mineralocorticoid Receptor Antagonists - pharmacology | Trans-Activators - genetics | Respiratory Syncytial Virus Infections - virology | Genes, Reporter | Cytoprotection | Disease Models, Animal | Kidney Diseases - metabolism | Cell Line | Promoter Regions, Genetic | Podocytes - metabolism | Kidney Diseases - pathology | Kidney Diseases - prevention & control | Mice, Inbred C57BL | Gene Expression Regulation | Respiratory Syncytial Virus Infections - genetics | Mitochondria - metabolism | Mitochondria - drug effects | Mitochondria - pathology | Podocytes - pathology | Animals | Podocytes - drug effects | Mitochondria - virology | Trans-Activators - metabolism | Mice | Transcription Factors | Enzyme Activation | Respiratory Syncytial Viruses - pathogenicity | Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 07/2010, Volume 285, Issue 30, pp. 23457 - 23465
Vascular endothelial growth factor (VEGF) is a dimeric glycoprotein that plays a crucial role in microvascular complications of diabetes, including diabetic... 
KIDNEY GLOMERULI | INHIBITION | REPRESSION | VEGF | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENE-EXPRESSION | HIGH GLUCOSE | DIABETIC-NEPHROPATHY | MICE | MOUSE PODOCYTES | CELL-LINES | Diabetes Mellitus - pathology | Diabetes Mellitus - genetics | Humans | 3' Untranslated Regions - genetics | Molecular Sequence Data | MicroRNAs - metabolism | Minichromosome Maintenance Complex Component 7 | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Promoter Regions, Genetic - genetics | Hyperglycemia - genetics | Dose-Response Relationship, Drug | Base Sequence | Hyperglycemia - pathology | Cell Cycle Proteins - genetics | Transcription, Genetic | Hyperglycemia - physiopathology | Nuclear Proteins - genetics | Microvessels - cytology | Reproducibility of Results | Podocytes - metabolism | Endothelial Cells - metabolism | Diabetes Mellitus - metabolism | Morpholines - pharmacology | Glucose - pharmacology | Podocytes - pathology | DNA-Binding Proteins - genetics | Down-Regulation - drug effects | Morpholines - chemistry | Hyperglycemia - metabolism | Podocytes - cytology | Animals | Podocytes - drug effects | Diabetes Mellitus - physiopathology | Polymers - chemistry | Mice | MicroRNAs - genetics | HeLa Cells | Endothelial Cells - drug effects | Index Medicus | Molecular Bases of Disease | MicroRNA | Signal Transduction | Gene regulation | Diabetes | Growth factors | Kidney
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 06/2011, Volume 121, Issue 6, pp. 2181 - 2196
Diabetic nephropathy (DN) is among the most lethal complications that occur in type 1 and type 2 diabetics. Podocyte dysfunction is postulated to be a critical... 
EPITHELIAL-MESENCHYMAL TRANSITION | KIDNEY-DISEASE | MAMMALIAN TARGET | MEDICINE, RESEARCH & EXPERIMENTAL | TUBEROUS SCLEROSIS COMPLEX | SLIT DIAPHRAGM | GLOMERULAR PROTEIN | ANGIOTENSIN-II | NEPHRIN EXPRESSION | ENDOPLASMIC-RETICULUM STRESS | NEPHROTIC SYNDROME | Phosphorylation | Ribosomal Protein S6 Kinases - metabolism | Diabetic Nephropathies - etiology | Diabetes Mellitus, Type 2 - genetics | Multiprotein Complexes | Podocytes - enzymology | Diabetic Nephropathies - drug therapy | Endoplasmic Reticulum - metabolism | Male | Mechanistic Target of Rapamycin Complex 1 | Mice, Mutant Strains | Tumor Suppressor Proteins - deficiency | Tumor Suppressor Proteins - genetics | Glomerular Basement Membrane - pathology | Regulatory-Associated Protein of mTOR | Cell Differentiation | Membrane Proteins - metabolism | Diabetic Nephropathies - prevention & control | Diabetes Mellitus, Type 2 - complications | Disease Models, Animal | Carrier Proteins - physiology | Proteins - physiology | Sirolimus - therapeutic use | Mice, Inbred C57BL | Podocytes - pathology | Mice, Knockout | Carrier Proteins - genetics | Tumor Suppressor Proteins - physiology | Adaptor Proteins, Signal Transducing | Glomerular Mesangium - pathology | Animals | Podocytes - drug effects | Podocytes - physiology | Mice | Protein Processing, Post-Translational | TOR Serine-Threonine Kinases | Enzyme Activation | Proteins - antagonists & inhibitors | Diabetic nephropathies | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Nature Medicine, ISSN 1078-8956, 06/2015, Volume 21, Issue 6, pp. 601 - 609
Journal Article
Journal Article
Kidney International, ISSN 0085-2538, 01/2012, Volume 81, Issue 1, pp. 40 - 55
Journal Article
Kidney International, ISSN 0085-2538, 10/2014, Volume 86, Issue 4, pp. 712 - 725
Journal Article
Oncotarget, ISSN 1949-2553, 2017, Volume 8, Issue 24, pp. 39592 - 39604
Journal Article
Kidney International, ISSN 0085-2538, 03/2011, Volume 79, Issue 6, pp. 624 - 634
All- retinoic acid protects against the development of HIV-associated nephropathy (HIVAN) in HIV-1 transgenic mice (Tg26). , all- retinoic acid inhibits... 
kidney disease | HIV | podocytes | proteinuria | retinoic acid receptor | ANTIRETROVIRAL THERAPY | IN-VITRO | PATHOLOGICAL CLASSIFICATION | PODOCYTE PROLIFERATION | UROLOGY & NEPHROLOGY | MICE | HIV-1-ASSOCIATED NEPHROPATHY | DIFFERENTIATION | FOCAL SEGMENTAL GLOMERULOSCLEROSIS | EXPRESSION | GLOMERULAR-DISEASE | Retinoids - metabolism | Cell Proliferation | Glomerulonephritis - prevention & control | Humans | Podocytes - virology | Male | Glomerulonephritis - metabolism | Receptors, Retinoic Acid - genetics | AIDS-Associated Nephropathy - genetics | Proteinuria - prevention & control | Time Factors | Receptors, Retinoic Acid - agonists | AIDS-Associated Nephropathy - virology | Female | Hydroxysteroid Dehydrogenases - metabolism | Cell Differentiation | AIDS-Associated Nephropathy - prevention & control | Disease Models, Animal | Severity of Illness Index | Podocytes - metabolism | Receptors, Retinoic Acid - deficiency | Proteinuria - virology | Proteinuria - metabolism | Alcohol Oxidoreductases - metabolism | Glomerulonephritis - virology | Mice, Transgenic | Receptors, Retinoic Acid - metabolism | Podocytes - pathology | HIV-1 - genetics | AIDS-Associated Nephropathy - pathology | Mice, Knockout | Animals | Podocytes - drug effects | Signal Transduction - drug effects | Tetrahydronaphthalenes - pharmacology | Benzoates - pharmacology | AIDS-Associated Nephropathy - metabolism | Mice | Retinoic Acid Receptor alpha | Index Medicus
Journal Article