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1. Full Text Podocyte injury and its consequences
by Nagata, Michio
Kidney international, ISSN 0085-2538, 06/2016, Volume 89, Issue 6, pp. 1221 - 1230
focal segmental glomerulosclerosis | cytoskeleton | renal pathology | podocyte | Life Sciences & Biomedicine | Urology & Nephrology | Science & Technology | Glomerular Basement Membrane - metabolism | Endothelial Cells | Podocytes - metabolism | Oxidative Stress | Humans | Cell Communication | Stress, Mechanical | Bowman Capsule - pathology | Podocytes - pathology | Proteinuria - pathology | Glomerular Basement Membrane - cytology | Sclerosis | Podocytes - cytology | Glomerular Basement Membrane - pathology | Bowman Capsule - cytology | Cell Differentiation | Glomerulosclerosis, Focal Segmental - pathology | Apoptosis | Index Medicus
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2. Full Text Activation of peroxisome proliferator-activated receptor-γ coactivator 1α ameliorates mitochondrial dysfunction and protects podocytes from aldosterone-induced injury
by Yuan, Yanggang and Huang, Songming and Wang, Wenyan and Wang, Yingying and Zhang, Ping and Zhu, Chunhua and Ding, Guixia and Liu, Bicheng and Yang, Tianxin and Zhang, Aihua
Kidney international, ISSN 0085-2538, 10/2012, Volume 82, Issue 7, pp. 771 - 789
mitochondrial dysfunction | podocytes | SIRT1 | aldosterone | PGC-1a | Life Sciences & Biomedicine | Urology & Nephrology | Science & Technology | Nephrology. Urinary tract diseases | Errors of metabolism | Biological and medical sciences | Metabolic diseases | Medical sciences | Miscellaneous hereditary metabolic disorders | Sirtuin 1 - metabolism | Apoptosis - drug effects | Receptors, Mineralocorticoid - metabolism | Podocytes - virology | Respiratory Syncytial Virus Infections - metabolism | Stilbenes - pharmacology | Aldosterone | Sirtuin 1 - genetics | Kidney Diseases - genetics | Receptors, Mineralocorticoid - drug effects | Transfection | RNA Interference | Time Factors | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Kidney Diseases - chemically induced | Mineralocorticoid Receptor Antagonists - pharmacology | Trans-Activators - genetics | Respiratory Syncytial Virus Infections - virology | Genes, Reporter | Cytoprotection | Disease Models, Animal | Kidney Diseases - metabolism | Cell Line | Promoter Regions, Genetic | Podocytes - metabolism | Kidney Diseases - pathology | Kidney Diseases - prevention & control | Mice, Inbred C57BL | Gene Expression Regulation | Respiratory Syncytial Virus Infections - genetics | Mitochondria - metabolism | Mitochondria - drug effects | Mitochondria - pathology | Podocytes - pathology | Animals | Podocytes - drug effects | Mitochondria - virology | Trans-Activators - metabolism | Mice | Transcription Factors | Enzyme Activation | Respiratory Syncytial Viruses - pathogenicity | Index Medicus
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3. Full Text mTORC1 activation in podocytes is a critical step in the development of diabetic nephropathy in mice
by Inoki, Ken and Mori, Hiroyuki and Wang, Junying and Suzuki, Tsukasa and Hong, SungKi and Yoshida, Sei and Blattner, Simone M and Ikenoue, Tsuneo and Rüegg, Markus A and Hall, Michael N and Kwiatkowski, David J and Rastaldi, Maria P and Huber, Tobias B and Kretzler, Matthias and Holzman, Lawrence B and Wiggins, Roger C and Guan, Kun-Liang
The Journal of clinical investigation, ISSN 0021-9738, 06/2011, Volume 121, Issue 6, pp. 2181 - 2196
Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Phosphorylation | Ribosomal Protein S6 Kinases - metabolism | Diabetic Nephropathies - etiology | Diabetes Mellitus, Type 2 - genetics | Multiprotein Complexes | Podocytes - enzymology | Diabetic Nephropathies - drug therapy | Endoplasmic Reticulum - metabolism | Male | Mechanistic Target of Rapamycin Complex 1 | Mice, Mutant Strains | Tumor Suppressor Proteins - deficiency | Tumor Suppressor Proteins - genetics | Glomerular Basement Membrane - pathology | Regulatory-Associated Protein of mTOR | Cell Differentiation | Membrane Proteins - metabolism | Diabetic Nephropathies - prevention & control | Diabetes Mellitus, Type 2 - complications | Disease Models, Animal | Carrier Proteins - physiology | Proteins - physiology | Sirolimus - therapeutic use | Mice, Inbred C57BL | Podocytes - pathology | Mice, Knockout | Carrier Proteins - genetics | Tumor Suppressor Proteins - physiology | Adaptor Proteins, Signal Transducing | Glomerular Mesangium - pathology | Animals | Podocytes - drug effects | Podocytes - physiology | Mice | Protein Processing, Post-Translational | TOR Serine-Threonine Kinases | Enzyme Activation | Proteins - antagonists & inhibitors | Diabetic nephropathies | Index Medicus | Abridged Index Medicus
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4. Full Text The spectrum of podocytopathies: A unifying view of glomerular diseases
by Wiggins, R.-C
Kidney international, ISSN 0085-2538, 06/2007, Volume 71, Issue 12, pp. 1205 - 1214
glomerulonephropathy | glomerulosclerosis | glomerulonephritis | glomerulus | nephrotic syndrome | podocyte | Glomerulonephritis | Glomerulosclerosis | Podocyte | Glomerulus | Nephrotic syndrome | Glomerulonephropathy | Life Sciences & Biomedicine | Urology & Nephrology | Science & Technology | Nephrology. Urinary tract diseases | Biological and medical sciences | Medical sciences | Nephropathies. Renovascular diseases. Renal failure | Glomerulonephritis - therapy | Podocytes - drug effects | Podocytes - physiology | Kidney Glomerulus - growth & development | Glomerulonephritis - pathology | Humans | Glomerulonephritis - etiology | Kidney Glomerulus - pathology | Podocytes - pathology | Disease Progression | Index Medicus
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5. Full Text Histone deacetylase 4 selectively contributes to podocyte injury in diabetic nephropathy
by Wang, Xiaojie and Liu, Jiang and Zhen, Junhui and Zhang, Chun and Wan, Qiang and Liu, Guangyi and Wei, Xinbing and Zhang, Yan and Wang, Ziying and Han, Huirong and Xu, Huiyan and Bao, Chanchan and Song, Zhenyu and Zhang, Xiumei and Li, Ningjun and Yi, Fan
Kidney international, ISSN 0085-2538, 10/2014, Volume 86, Issue 4, pp. 712 - 725
diabetic complications | gene therapy | histone deacetylases | autophagy | podocyte | Life Sciences & Biomedicine | Urology & Nephrology | Science & Technology | Histone Deacetylases - analysis | Up-Regulation | Diabetic Nephropathies - etiology | Humans | Male | RNA, Messenger - metabolism | Autophagy | Repressor Proteins - analysis | Repressor Proteins - antagonists & inhibitors | Inflammation - metabolism | STAT1 Transcription Factor - metabolism | Diabetes Mellitus, Experimental - complications | Podocytes - chemistry | Repressor Proteins - metabolism | Diabetic Nephropathies - pathology | Podocytes - metabolism | Histone Deacetylases - genetics | Signal Transduction | Diabetic Nephropathies - metabolism | Cells, Cultured | Enzyme Inhibitors - pharmacology | Gene Silencing | Rats | Histone Deacetylases - metabolism | Glucose - pharmacology | Podocytes - pathology | Rats, Sprague-Dawley | Transforming Growth Factor beta - pharmacology | Animals | Glycation End Products, Advanced - pharmacology | Inflammation - genetics | Mice | Histone Deacetylase 2 - metabolism | Index Medicus
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6. Full Text Berberine enhances the AMPK activation and autophagy and mitigates high glucose-induced apoptosis of mouse podocytes
by Jin, Yingli and Liu, Shuping and Ma, Qingshan and Xiao, Dong and Chen, Li
European journal of pharmacology, ISSN 0014-2999, 01/2017, Volume 794, pp. 106 - 114
Podocyte | Berberine | AMPK | Autophagy | High glucose | Life Sciences & Biomedicine | Pharmacology & Pharmacy | Science & Technology | Cell Survival - drug effects | AMP-Activated Protein Kinases - metabolism | Podocytes - metabolism | TOR Serine-Threonine Kinases - metabolism | Apoptosis - drug effects | Glucose - pharmacology | Enzyme Activation - drug effects | Autophagy - drug effects | Dose-Response Relationship, Drug | Podocytes - cytology | Berberine - pharmacology | Animals | Podocytes - drug effects | Signal Transduction - drug effects | Cytoprotection - drug effects | Mice | Phosphorylation - drug effects | Glucose | Protein kinases | Dextrose | Diabetic nephropathies | Apoptosis | Index Medicus
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7. Full Text Actin dynamics at focal adhesions: a common endpoint and putative therapeutic target for proteinuric kidney diseases
by Sever, Sanja and Schiffer, Mario
Kidney international, ISSN 0085-2538, 06/2018, Volume 93, Issue 6, pp. 1298 - 1307
Bis-T-23 | actin | podocytes | dynamin | Life Sciences & Biomedicine | Urology & Nephrology | Science & Technology | Humans | Actins - metabolism | Proteinuria - pathology | Focal Adhesions - pathology | Molecular Targeted Therapy | Renal Insufficiency, Chronic - drug therapy | Renal Insufficiency, Chronic - metabolism | Actin Cytoskeleton - drug effects | Glomerular Basement Membrane - pathology | Renal Insufficiency, Chronic - genetics | Glomerular Basement Membrane - physiopathology | Glomerular Basement Membrane - metabolism | Actin Cytoskeleton - metabolism | Glomerular Filtration Rate | Podocytes - metabolism | Signal Transduction | Proteinuria - metabolism | Podocytes - pathology | Proteinuria - genetics | Renal Agents - therapeutic use | Renal Insufficiency, Chronic - pathology | Focal Adhesions - drug effects | Glomerular Basement Membrane - drug effects | Animals | Proteinuria - drug therapy | Podocytes - drug effects | Focal Adhesions - metabolism | Actin Cytoskeleton - pathology | Patents | Cell culture | Antigens | Animal models | Phenotypes | Kidneys | Epithelial cells | Therapeutic applications | Diabetes mellitus | Polymerization | Feet | Nephropathy | Microscopy | Actin | Glomerulus | Cytoskeleton | Kidney diseases | Diaphragm | Regulatory proteins | Proteinuria | Integrins | Index Medicus
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