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JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY, ISSN 0091-6749, 04/2013, Volume 131, Issue 4, pp. 1048 - 1048
Background: Asthma is defined as a chronic inflammatory disease of the airways; however, the underlying physiologic and immunologic processes are not fully... 
ALLERGIC INFLAMMATION | ASPERGILLUS-FUMIGATUS | ASTHMATIC RESPONSE | INCREASED EXPRESSION | RECEPTOR EXPRESSION | CD4(+) T-CELLS | chronic airway hyperreactivity | IMMUNOLOGY | Asthma | TGF-beta | costimulation | EPITHELIAL-CELLS | ALLERGY | T(H)9 | IL-9 PRODUCTION | ENHANCING ACTIVITY MEA | Aspergillus fumigatus | programmed cell death ligand 2
Journal Article
Journal Article
The Journal of Allergy and Clinical Immunology, ISSN 0091-6749, 08/2018, Volume 142, Issue 2, pp. 530 - 541.e6
Inhaled protease allergens preferentially trigger T 2-mediated inflammation in allergic asthma. The role of dendritic cells (DCs) on induction of T 2 cell... 
mast cell | Protease allergen | PD-L2+ dendritic cells | fibrinogen cleavage product | Toll-like receptor 4 | PD-L2 | dendritic cells | IMMUNITY | INITIATION | MAST-CELLS | C-KIT | TYPE-2 RESPONSES | INNATE LYMPHOID-CELLS | IMMUNOLOGY | ALLERGY | IN-VIVO | ANTIGEN-PRESENTING CELLS | PD-L2(+) dendritic cells | LUNG INFLAMMATION | TH2 | Peptide Fragments - metabolism | Lymphocyte Activation | Dendritic Cells - immunology | Humans | Mice, Inbred C57BL | Hypersensitivity - immunology | Programmed Cell Death 1 Ligand 2 Protein - metabolism | Toll-Like Receptor 4 - genetics | Th2 Cells - immunology | Immunity, Innate | Interleukin-13 - metabolism | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Animals | Allergens - immunology | Peptide Fragments - immunology | Asthma - immunology | Fibrinogen - metabolism | Cell Differentiation | Mice | Fibrinogen - immunology | Disease Models, Animal | Peptide Hydrolases - metabolism | Fibrin | Biochemistry | Dendritic cells | Fibrinogen | Asthma | Apoptosis | Cell culture | Allergens | Proteinase | Lymphocytes T | Experiments | Cell surface | Lymph nodes | Proteins | Respiratory tract | Protease | Lymphocytes | Toll-like receptors | Bone marrow | Cleavage | Population | Respiratory tract diseases | Immune system | Antigens | Phenotypes | Lymphoid cells | Cytokines | Hypersensitivity | Inflammation | Intranasal administration | Interleukin 13 | TLR4 protein | Allergies | Studies | Histamine | Lungs | Cell death | Ligands | Mast cells | Laboratory animals | Lymph
Journal Article
American journal of physiology. Gastrointestinal and liver physiology, ISSN 0193-1857, 01/2019, Volume 316, Issue 1, pp. G106 - G114
The liver is an organ that, when dysfunctional in a septic patient, is strongly associated with morbidity and mortality. Understanding the pathophysiology of... 
inflammation | programmed cell death receptor ligand 1 | posttranslational modification | programmed cell death receptor-1 | sepsis | MORTALITY | APOPTOSIS | PHYSIOLOGY | MACROPHAGES | MEDIATOR | PD-L2 | GASTROENTEROLOGY & HEPATOLOGY
Journal Article
Cancer Science, ISSN 1347-9032, 11/2016, Volume 107, Issue 11, pp. 1696 - 1704
Immune escape and tolerance in the tumor microenvironment are closely involved in tumor progression, and are caused by T‐cell exhaustion and mediated by the... 
macrophage | CD163 | tumor‐associated macrophages | PD‐L2 | PD‐L1 | PD-L1 | PD-L2 | tumor-associated macrophages | B7-H1 EXPRESSION | OVARIAN-CANCER | LUNG-CANCER | THERAPY | ONCOLOGY | IMMUNE CHECKPOINT BLOCKADE | INTERLEUKIN-27 | IMMUNOTHERAPY | UP-REGULATION | PROGRESSION | Programmed Cell Death 1 Ligand 2 Protein - genetics | Humans | Programmed Cell Death 1 Ligand 2 Protein - metabolism | Culture Media, Conditioned - pharmacology | B7-H1 Antigen - genetics | Interleukins - metabolism | Leukemia-Lymphoma, Adult T-Cell - immunology | Up-Regulation - drug effects | B7-H1 Antigen - metabolism | Macrophages - metabolism | Minor Histocompatibility Antigens - metabolism | Leukemia-Lymphoma, Adult T-Cell - metabolism | B7-H1 Antigen - biosynthesis | Programmed Cell Death 1 Ligand 2 Protein - biosynthesis | Cell Line, Tumor | Macrophages - drug effects | STAT3 Transcription Factor - metabolism | Immunotherapy | Development and progression | Lymphomas | Biochemistry | T cells | Macrophages | Apoptosis | Cell culture | Phosphorylation | Transcription | Leukemia | Cytotoxicity | Paraffin | Cell activation | Immunoglobulins | Cytokines | Review boards | Research & development--R&D | Cloning | Stat3 protein | Tumor cell lines | Lymphoma | Immunological tolerance | Studies | Mucin | Immune checkpoint | Lymphocytes B | Cell death | Cell lines | Cancer | Tumors | Original
Journal Article
Journal Article
Journal Article
Blood, ISSN 0006-4971, 2013, Volume 122, Issue 17, pp. 3062 - 3073
Programmed death 1 (PD-1) and its ligands, PD-L1 and PD-L2, play an important role in the maintenance of peripheral tolerance. We explored the role of PD-1... 
EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | BONE-MARROW-TRANSPLANTATION | T-CELL-ACTIVATION | GASTROINTESTINAL-TRACT | ENDOTHELIAL-CELLS | IN-VIVO | TNF-ALPHA | HEMATOLOGY | PD-1 | GAMMA-DEPENDENT MECHANISM | LYMPHOCYTES | Glucose Transport Proteins, Facilitative - metabolism | Liver - pathology | Cell Proliferation | Spleen - immunology | Programmed Cell Death 1 Ligand 2 Protein - metabolism | Intestines - metabolism | Graft vs Host Disease - metabolism | CD4-Positive T-Lymphocytes - pathology | Graft vs Host Disease - immunology | Intestines - immunology | CD4-Positive T-Lymphocytes - immunology | Liver - immunology | Glucose Transport Proteins, Facilitative - immunology | Graft vs Host Disease - mortality | Bone Marrow Transplantation | Apoptosis Regulatory Proteins - genetics | Glucose Transport Proteins, Facilitative - genetics | Spleen - pathology | Transplantation Tolerance - immunology | Acute Disease | Apoptosis Regulatory Proteins - immunology | Intestines - pathology | Programmed Cell Death 1 Ligand 2 Protein - genetics | Liver - metabolism | CD4-Positive T-Lymphocytes - metabolism | Gene Expression Regulation | Oxidative Phosphorylation | Programmed Cell Death 1 Receptor - metabolism | B7-H1 Antigen - genetics | B7-H1 Antigen - immunology | Apoptosis Regulatory Proteins - metabolism | Graft vs Host Disease - pathology | Programmed Cell Death 1 Ligand 2 Protein - immunology | B7-H1 Antigen - metabolism | Animals | Spleen - metabolism | Peripheral Tolerance - immunology | Survival Analysis | Glycolysis | Mice | Programmed Cell Death 1 Receptor - immunology | Programmed Cell Death 1 Receptor - genetics | Transplantation
Journal Article
by Gao, Y and Li, S and Xu, DZ and Chen, SX and Cai, YC and Jiang, WQ and Zhang, XK and Sun, J and Wang, KF and Chang, BY and Wang, FH and Hong, MH
CHINESE JOURNAL OF CANCER, ISSN 1000-467X, 07/2017, Volume 36, Issue 1, pp. 61 - 14
Journal Article
ONCOLOGY REPORTS, ISSN 1021-335X, 08/2019, Volume 42, Issue 2, pp. 839 - 848
Platinum-based chemotherapy improves the clinical outcome of patients with non-small cell lung cancer (NSCLC), although tumors often become refractory after... 
non-small cell lung cancer | ACTIVATION | interferon gamma | PD-L1 EXPRESSION | NK group 2 member D ligand | CLASSIFICATION | NKG2D | IMPACT | NK CELLS | ONCOLOGY | programmed cell death-1 ligand 1 | cisplatin | CHAIN-RELATED MOLECULE | human leucocyte antigen class I | CYTOTOXICITY | B EXPRESSION | T-CELLS
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 10/2014, Volume 20, Issue 19, pp. 5064 - 5074
Journal Article