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Cell Biology International, ISSN 1065-6995, 07/2017, Volume 41, Issue 7, pp. 798 - 808
Under normal conditions, the activity of platelets is stringently and precisely balanced between activation and quiescent state. This guarantees rapid... 
platelet | endothelium‐derived inhibitory mechanisms | xenotransplantation | porcine aortic endothelial cells | endothelium-derived inhibitory mechanisms | OXIDATIVE STRESS | ACTIVATION | BABOONS | NITRIC-OXIDE SYNTHASE | PLATELET-FUNCTION | RECEPTOR | GENE-KNOCKOUT PIGS | MICE LACKING | CELL BIOLOGY | HUMAN-BLOOD | INFLAMMATORY RESPONSE | Intramolecular Oxidoreductases - biosynthesis | Prostaglandin-Endoperoxide Synthases - biosynthesis | Epoprostenol - metabolism | Endothelial Cells - metabolism | Humans | Nitric Oxide Synthase Type III - biosynthesis | Cytochrome P-450 Enzyme System - metabolism | Transplantation, Heterologous | Aorta - metabolism | Aorta - pathology | Animals | Blood Platelets - metabolism | Platelet Activation | Swine | Thromboxane-A Synthase - biosynthesis | Adenosine - metabolism | Cytochrome P-450 Enzyme System - biosynthesis | Serum | Nitric Oxide Synthase Type III - metabolism | Endothelial Cells - pathology | Intramolecular Oxidoreductases - metabolism | Nitric Oxide - metabolism | Cell culture | Animal models | Xenotransplantation | Activation | Prostacyclin | Arteries | Control | Blood platelets | Hemostasis | Xenografts | Aorta | CD73 antigen | Hemostatics | Thrombotic microangiopathy | Adenosine | Exposure | Gene expression | Thrombosis | Endothelial cells | Endothelium | Prostacyclin PGI2 | Nitric oxide | Regulatory mechanisms (biology) | Prostacyclin synthase | Platelets | Index Medicus
Journal Article
Journal Article
Carcinogenesis, ISSN 0143-3334, 03/2010, Volume 31, Issue 3, pp. 427 - 434
Hypoxia-inducible factors (HIFs), in particular HIF-1 alpha, have been implicated in tumor biology. However, HIF target genes in the esophageal tumor... 
CANCER-CELLS | ESOPHAGEAL EPITHELIAL-CELLS | INDUCIBLE FACTOR-1-ALPHA | TRANSCRIPTIONAL ACTIVITY | ONCOLOGY | PROSTAGLANDIN-E SYNTHASE-1 | GASTRIC-CANCER | COLORECTAL-CANCER | SQUAMOUS-CELL CARCINOMA | 15-HYDROXYPROSTAGLANDIN DEHYDROGENASE | UP-REGULATION | Intramolecular Oxidoreductases - biosynthesis | Cell Hypoxia - physiology | Insulin-Like Growth Factor Binding Proteins - physiology | Epithelial Cells - metabolism | Dinoprostone - biosynthesis | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Humans | Intramolecular Oxidoreductases - physiology | Neoplasm Proteins - physiology | Recombinant Fusion Proteins - physiology | Intramolecular Oxidoreductases - antagonists & inhibitors | Gene Expression Profiling | Interleukin-1beta - genetics | Oxygen - metabolism | Esophageal Neoplasms - pathology | RNA, Messenger - biosynthesis | Cobalt - pharmacology | RNA Interference | Esophageal Neoplasms - metabolism | Interleukin-1beta - biosynthesis | Hypoxia-Inducible Factor 1, alpha Subunit - physiology | Cell Line | RNA, Messenger - genetics | RNA, Small Interfering - pharmacology | Cyclooxygenase 2 - physiology | Hydroxyprostaglandin Dehydrogenases - biosynthesis | Tumor Cells, Cultured - metabolism | Hydroxyprostaglandin Dehydrogenases - genetics | Oxygen - administration & dosage | RNA, Neoplasm - biosynthesis | Esophageal Neoplasms - genetics | Intramolecular Oxidoreductases - genetics | Prostaglandin-E Synthases | Insulin-Like Growth Factor Binding Protein 3 | RNA, Neoplasm - genetics | Enzyme Activation | Index Medicus | Cancer Biology
Journal Article
Hypertension, ISSN 0194-911X, 09/2009, Volume 54, Issue 3, pp. 652 - 658
Drugs and antibodies that interrupt vascular endothelial growth factor (VEGF) signaling pathways improve outcomes in patients with a variety of cancers by... 
Hypertension | Angiogenesis | NO | Vascular endothelial growth factor | Cancer | TYROSINE KINASE-1 | BEVACIZUMAB | angiogenesis | vascular endothelial growth factor | VEGF | MOUSE | PERIPHERAL VASCULAR DISEASE | cancer | MONOCLONAL-ANTIBODY | MICE | hypertension | MOLECULAR-MECHANISMS | Gene Expression - drug effects | Kidney - enzymology | Antibodies, Monoclonal - adverse effects | Sodium Chloride, Dietary - administration & dosage | Male | Vascular Endothelial Growth Factor Receptor-2 - immunology | RNA, Messenger - metabolism | Aldosterone - urine | Hypertension - etiology | Vascular Endothelial Growth Factor Receptor-2 - antagonists & inhibitors | Renin - genetics | Prostaglandins - urine | Nitric Oxide Synthase Type I - antagonists & inhibitors | Blood Pressure - drug effects | Blood Pressure - physiology | Vascular Endothelial Growth Factor Receptor-2 - physiology | Nitric Oxide Synthase Type III - metabolism | Manometry | NG-Nitroarginine Methyl Ester - pharmacology | Nitric Oxide - biosynthesis | Kidney - drug effects | Antibodies, Monoclonal - pharmacology | RNA, Messenger - genetics | Sodium Chloride, Dietary - adverse effects | Enzyme Inhibitors - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Nitric Oxide Synthase Type III - genetics | Hypertension - physiopathology | Nitric Oxide Synthase Type III - antagonists & inhibitors | Animals | Signal Transduction - drug effects | Nitric Oxide Synthase Type I - metabolism | Hybridomas | Mice | Nitric Oxide Synthase Type I - genetics | Vascular Endothelial Growth Factor A - physiology | Index Medicus | nitric oxide
Journal Article
Oxidative Medicine and Cellular Longevity, ISSN 1942-0900, 2014, Volume 2014, pp. 212576 - 12
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2016, Volume 113, Issue 4, pp. 972 - 977
Journal Article
Circulation, ISSN 0009-7322, 02/2005, Volume 111, Issue 6, pp. 796 - 803
Background - Endothelium-dependent dilatation is mediated by 3 principal vasodilators: nitric oxide (NO), prostacyclin (PGI(2)), and endothelium-derived... 
Hypertension | Nitric oxide | Endothelium | endothelium | nitric oxide | CARDIAC & CARDIOVASCULAR SYSTEMS | MESENTERIC-ARTERIES | MOUSE AORTA | CAROTID-ARTERY | NATRIURETIC PEPTIDE | CORONARY-ARTERIES | ESTROGEN-RECEPTOR-BETA | GAP-JUNCTIONS | RESISTANCE VESSELS | GENE DISRUPTION | PERIPHERAL VASCULAR DISEASE | CEREBRAL-ARTERIES | HEMATOLOGY | hypertension | Blood Pressure - genetics | Cyclooxygenase 1 | Nitric Oxide Synthase - physiology | Endothelium, Vascular - drug effects | Male | Aorta - metabolism | Endothelium, Vascular - enzymology | Nitric Oxide Synthase - genetics | Nitric Oxide Synthase Type II | Vascular Resistance - physiology | Dose-Response Relationship, Drug | Vascular Resistance - genetics | Vasodilation - genetics | Capillary Resistance - physiology | Vascular Resistance - drug effects | Female | Blood Pressure - drug effects | Blood Pressure - physiology | Aorta - enzymology | Bradykinin - pharmacology | Prostaglandin-Endoperoxide Synthases - genetics | Acetylcholine - pharmacology | Aorta - drug effects | Mice, Inbred C57BL | Nitric Oxide Synthase Type III | Genotype | Capillary Resistance - drug effects | Prostaglandin-Endoperoxide Synthases - deficiency | Mesenteric Arteries - metabolism | Mice, Knockout | Membrane Proteins | Animals | Electrophysiology - methods | Endothelium-Dependent Relaxing Factors - metabolism | Nitric Oxide Synthase - deficiency | Endothelium, Vascular - metabolism | Mice | Vasodilation - drug effects | Mesenteric Arteries - enzymology | 6-Ketoprostaglandin F1 alpha - blood | Capillary Resistance - genetics | Prostaglandin-Endoperoxide Synthases - physiology | Index Medicus | Abridged Index Medicus
Journal Article
Biochemical Pharmacology, ISSN 0006-2952, 2008, Volume 75, Issue 2, pp. 484 - 493
Aldo-keto reductase (AKR) 1C3 (type 2 3α-HSD, type 5 17β-HSD, and prostaglandin F synthase) regulates ligand access to steroid hormone and prostaglandin... 
Prostaglandin metabolism | Inhibitor design | Steroid hormone metabolism | Cancer treatment | NSAIDs | Chemoprevention | cancer treatment | ANDROGEN RECEPTOR | SIGNALING PATHWAYS | INCREASED EXPRESSION | CRYSTAL-STRUCTURE | prostaglandin metabolism | 15-DEOXY-DELTA(12,14)-PROSTAGLANDIN J | PROLIFERATOR-ACTIVATED RECEPTOR | 3-ALPHA-HYDROXYSTEROID DEHYDROGENASE | chemoprevention | NONSTEROIDAL ANTIINFLAMMATORY DRUGS | ACID-BINDING PROTEIN | CYCLOOXYGENASE-2 INHIBITORS | inhibitor design | PHARMACOLOGY & PHARMACY | steroid hormone metabolism | Indomethacin - analogs & derivatives | Oxidation-Reduction | Humans | Enzyme Inhibitors - pharmacology | Hydroxyprostaglandin Dehydrogenases - metabolism | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | 3-Hydroxysteroid Dehydrogenases - antagonists & inhibitors | Hydroxyprostaglandin Dehydrogenases - antagonists & inhibitors | Melatonin - analogs & derivatives | Aldo-Keto Reductase Family 1 Member C3 | 3-Hydroxysteroid Dehydrogenases - metabolism | Indoles - pharmacology | Antineoplastic Agents - pharmacology | Catalysis | Neoplasms, Hormone-Dependent - drug therapy | Melatonin - pharmacology | Phenanthrenes - metabolism | Dihydrotestosterone - metabolism | Enzymes | Care and treatment | Synthetic prostaglandins F | Crystals | Hormones | Organic acids | Heterocyclic compounds | Physiological aspects | Glycols | Hormones, Sex | Indomethacin | Structure | Cancer | Index Medicus
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 03/2016, Volume 310, Issue 6, pp. H756 - H764
Journal Article