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Blood Cells, Molecules and Diseases, ISSN 1079-9796, 04/2014, Volume 52, Issue 4, pp. 147 - 151
Journal Article
Journal Article
Cancer Letters, ISSN 0304-3835, 2014, Volume 356, Issue 2, pp. 454 - 461
Abstract Radiotherapy is an important treatment option for many human cancers. Current research is investigating the use of molecular targeted drugs in order... 
Hematology, Oncology and Palliative Medicine | Radiation-induced bystander effect | BRCA | Fanconi anaemia | DNA damage response | Ionising radiation | GLIOMA-CELLS | ALPHA-PARTICLES | COMET ASSAY | FANCONI-ANEMIA/BRCA PATHWAY | HISTONE H2AX | BREAST-CANCER | ONCOLOGY | IRRADIATED-CELLS | HUMAN FIBROBLASTS | SISTER-CHROMATID EXCHANGES | UNIRRADIATED CELLS | Protein Kinases - metabolism | Humans | Brain Neoplasms - pathology | DNA Repair - radiation effects | DNA Repair - genetics | Glioma - radiotherapy | Brain Neoplasms - metabolism | Immunoenzyme Techniques | Glioma - metabolism | Glioma - genetics | Flow Cytometry | BRCA1 Protein - metabolism | DNA Replication - radiation effects | Glioma - pathology | DNA Damage - genetics | Bystander Effect - genetics | Signal Transduction - radiation effects | Tumor Cells, Cultured | Brain Neoplasms - radiotherapy | Bystander Effect - radiation effects | Brain Neoplasms - genetics | Mutation - genetics | Blotting, Western | Phosphorylation - radiation effects | Fanconi Anemia Complementation Group D2 Protein - metabolism | X-Rays | DNA Replication - genetics | Checkpoint Kinase 1 | Ataxia Telangiectasia Mutated Proteins - genetics | DNA Damage - radiation effects | Cell Proliferation - radiation effects | Genetic research | Radiotherapy | DNA repair | DNA damage | DNA | Radiation | Medical research | Cell cycle | Breast cancer | Mutation | Kinases | ionising radiation
Journal Article
Journal Article
Cellular Signalling, ISSN 0898-6568, 05/2007, Volume 19, Issue 5, pp. 892 - 901
The cellular response to genotoxic stress that damages DNA includes cell cycle arrest, activation of DNA repair, and in the event of irreparable damage,... 
PKCδ | Phosphorylation | Caspase-3 | DNA damage | Apoptosis | PROTEIN-KINASE-C | ABL TYROSINE KINASE | PKC delta | apoptosis | RADIATION-INDUCED APOPTOSIS | CELL BIOLOGY | HIGH-AFFINITY RECEPTOR | TUMOR-NECROSIS-FACTOR | GLAND ACINAR-CELLS | PROSTATE-CANCER CELLS | ETOPOSIDE-INDUCED APOPTOSIS | SRC FAMILY KINASES | phosphorylation | NF-KAPPA-B | caspase-3
Journal Article
Mutation Research - Fundamental and Molecular Mechanisms of Mutagenesis, ISSN 0027-5107, 04/2005, Volume 571, Issue 1-2, pp. 121 - 132
Journal Article