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Nature, ISSN 0028-0836, 06/2009, Volume 459, Issue 7248, pp. 852 - 856
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 0027-8424, 8/2014, Volume 111, Issue 34, pp. 12544 - 12549
Nervous system injury or disease leads to activation of glia, which govern postinjury responses in the nervous system. Axonal injury in Drosophila results in... 
Axons | Axotomy | Receptors | Neurons | Neuroglia | Drosophila | Internalization | Antennal lobe | Glomerulus | P branes | Draper pathway | Reactive glia | Engulfment signaling | Wallerian degeneration | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | ras Proteins - genetics | Drosophila melanogaster - physiology | Cytoskeletal Proteins - genetics | Phagosomes - physiology | Axons - physiology | Drosophila melanogaster - genetics | Drosophila Proteins - physiology | Son of Sevenless Protein, Drosophila - genetics | Proto-Oncogene Proteins c-crk - physiology | rac GTP-Binding Proteins - genetics | Cytoskeletal Proteins - physiology | Eye Proteins - genetics | rac GTP-Binding Proteins - physiology | ras Proteins - physiology | Genes, Insect | Animals, Genetically Modified | Drosophila melanogaster - cytology | Neuroglia - physiology | Proto-Oncogene Proteins c-crk - genetics | Son of Sevenless Protein, Drosophila - physiology | Adaptor Proteins, Signal Transducing - physiology | Animals | Adaptor Proteins, Signal Transducing - genetics | Eye Proteins - physiology | Drosophila Proteins - genetics | Mutation | Nerve Degeneration | Nervous system diseases | Neurogenetics | Physiological aspects | Nervous system | Genetic aspects | Degeneration | Research | Neurological research | Guanosine triphosphatase | Biological Sciences | engulfment signaling | reactive glia
Journal Article
2004, Molecular biology intelligence unit, ISBN 9780306479922, 235
Book
The EMBO journal, ISSN 1460-2075, 2002, Volume 21, Issue 13, pp. 3358 - 3369
In this study, we investigated the subcellular and molecular mechanisms underlying promyelocytic leu kemia (PML)‐induced premature senescence. We demonstrate... 
senescence | PML nuclear bodies | telomerase | PML | p53 | Senescence | Telomerase | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Phosphorylation | Reactive Oxygen Species | Humans | Neoplasm Proteins - physiology | Organelles - physiology | Transcription Factors - deficiency | Recombinant Fusion Proteins - physiology | SUMO-1 Protein - physiology | Proto-Oncogene Proteins p21(ras) - physiology | Structure-Activity Relationship | Antigens, Nuclear | Telomerase - physiology | Protein Isoforms - deficiency | Acetylation | Neoplasm Proteins - genetics | DNA-Binding Proteins | Carrier Proteins - physiology | Cellular Senescence - genetics | Transcription Factors - physiology | Tumor Suppressor Protein p53 - metabolism | Cellular Senescence - physiology | Intracellular Signaling Peptides and Proteins | Transcription Factors - genetics | Cell Nucleus - ultrastructure | Protein Isoforms - physiology | Macromolecular Substances | Mice, Knockout | Autoantigens - physiology | Adaptor Proteins, Signal Transducing | Animals | Fibroblasts - cytology | Mice | Neoplasm Proteins - deficiency | Nuclear Proteins - physiology | Protein Processing, Post-Translational | Tumor Suppressor Protein p53 - chemistry | Promyelocytic Leukemia Protein | Tumor Suppressor Proteins | Protein Isoforms - genetics
Journal Article
Developmental biology, ISSN 0012-1606, 10/2016, Volume 418, Issue 2, pp. 258 - 267
Journal Article
Leukemia, ISSN 1476-5551, 2011, Volume 25, Issue 7, pp. 1080 - 1094
The Ras/Raf/mitogen-activated protein kinase (MEK)/extracellular signal-regulated kinase (ERK... 
targeted therapy | Ras | therapeutic sensitivity | Raf | resistance | ERK | Oncology | Life Sciences & Biomedicine | Hematology | Science & Technology | ras Proteins - genetics | MAP Kinase Signaling System - physiology | Mitogen-Activated Protein Kinase Kinases - genetics | Apoptosis - drug effects | raf Kinases - antagonists & inhibitors | Humans | Neoplasm Proteins - physiology | Mitogen-Activated Protein Kinase Kinases - physiology | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | Neoplasm Proteins - antagonists & inhibitors | Antineoplastic Agents - therapeutic use | Molecular Targeted Therapy | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Extracellular Signal-Regulated MAP Kinases - genetics | Gene Expression Regulation, Leukemic - genetics | raf Kinases - physiology | MAP Kinase Signaling System - genetics | Drug Design | Antineoplastic Agents - pharmacology | Extracellular Signal-Regulated MAP Kinases - physiology | Neoplasm Proteins - genetics | raf Kinases - genetics | ras Proteins - physiology | Cell Division - genetics | Mitogen-Activated Protein Kinase Kinases - antagonists & inhibitors | ras Proteins - antagonists & inhibitors | Gene Expression Regulation, Leukemic - drug effects | Leukemia - drug therapy | Cell Division - drug effects | Phosphatidylinositol 3-Kinases - genetics | Drug Resistance, Neoplasm - genetics | MAP Kinase Signaling System - drug effects | Models, Biological | Phosphatidylinositol 3-Kinases - physiology | Apoptosis - physiology | Drug Resistance, Neoplasm - physiology | Drug Resistance, Neoplasm - drug effects | Chemotherapy | Extracellular signal-regulated kinases | Leukemia | Physiological aspects | Genetic aspects | Research | Drug therapy | Health aspects | Mitogen-activated protein kinases | Cancer
Journal Article