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Cardiovascular Research, ISSN 0008-6363, 7/2011, Volume 91, Issue 2, pp. 289 - 299
Aims Human congenital heart disease linked to mutations in the homeobox transcription factor, NKX2-5, is characterized by cardiac anomalies, including atrial... 
Conduction | Heart defects | Genetics | Congenital | Contractility | LIGHT-CHAIN KINASE | CARDIAC & CARDIOVASCULAR SYSTEMS | CONDUCTION SYSTEM | CONNEXIN40-DEFICIENT MICE | HOMEODOMAIN PROTEIN | CONTRACTION DEFECTS | ATRIOVENTRICULAR-CONDUCTION | CONGENITAL HEART-DISEASE | VERTEBRATE HEART | RAT-HEART | HOMEOBOX GENE | Heart Defects, Congenital - embryology | Heart - embryology | Transcription Factors - deficiency | Male | Acetylcholinesterase - genetics | Heart Conduction System - embryology | Heart Defects, Congenital - genetics | Myocardial Contraction - genetics | Heart Defects, Congenital - diagnostic imaging | Ultrasonography | Heart Septal Defects, Atrial - metabolism | Female | Heart Septal Defects, Atrial - genetics | Atrial Septum - metabolism | Heart - physiopathology | Heart Conduction System - metabolism | Morphogenesis - genetics | Heart Rate - genetics | Gene Expression Regulation, Developmental - drug effects | Heart Septal Defects, Atrial - embryology | Transcription Factors - genetics | Gestational Age | Homeodomain Proteins - genetics | Mice, Knockout | Heart Ventricles - abnormalities | Homeobox Protein Nkx-2.5 | Animals | Analysis of Variance | Tamoxifen - pharmacology | Atrial Septum - embryology | Heart Defects, Congenital - metabolism | Heart Defects, Congenital - physiopathology | Heart Ventricles - metabolism | Mice | Calcium Signaling - genetics | Acetylcholinesterase - metabolism | Original | Spotlight on Cardiac Development
Journal Article
Journal Article
Journal Article
Circulation, ISSN 0009-7322, 05/2009, Volume 119, Issue 19, pp. 2587 - 2596
Background-We report that elevated connexin-43 (Cx-43) in stem cells preconditioned with insulin-like growth factor-1 (IGF-1) is cytoprotective and reprograms... 
Insulin-like growth factor-1 | Angiogenesis | Connexin 43 | Stem cells | Apoptosis | stem cells | SIGNALING PATHWAYS | OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | MYOCARDIAL-INFARCTION | angiogenesis | GAP JUNCTIONAL COMMUNICATION | BONE-MARROW | apoptosis | insulin-like growth factor-1 | ISCHEMIC-HEART | CARDIAC REPAIR | PERIPHERAL VASCULAR DISEASE | connexin 43 | HEMATOLOGY | SKELETAL MYOBLAST TRANSPLANTATION | EXPRESSION | GROWTH-FACTOR-I | Insulin-Like Growth Factor I - pharmacology | Apoptosis - drug effects | Multipotent Stem Cells - metabolism | Rats, Inbred F344 | Cells, Cultured - transplantation | Male | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Membrane Proteins - analysis | Cell Hypoxia | Connexin 43 - physiology | Multipotent Stem Cells - drug effects | RNA Interference | Multipotent Stem Cells - transplantation | Myocardial Infarction - pathology | Female | Myocardial Infarction - physiopathology | Connexin 43 - genetics | Myocardial Infarction - surgery | Mesenchymal Stromal Cells - drug effects | Cells, Cultured - drug effects | Mice, Inbred C57BL | RNA, Small Interfering - pharmacology | Mesenchymal Stromal Cells - metabolism | Rats | Glucose - pharmacology | Proto-Oncogene Proteins c-akt - physiology | Recombinant Proteins - pharmacology | Antigens, Ly - analysis | Animals | Signal Transduction - drug effects | Ischemic Preconditioning, Myocardial | Phosphatidylinositol 3-Kinases - physiology | Cells, Cultured - metabolism | Mice | Apoptosis - physiology | Mesenchymal Stem Cell Transplantation | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Vasomotor conditioning | Patient outcomes | Physiological aspects | Research | Cell differentiation | Insulin-like growth factor 1 | Heart attack | Membrane proteins | differentiation | preconditioning | connexin-43
Journal Article
Journal Article