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1. Full Text The repair Schwann cell and its function in regenerating nerves
by Jessen, K. R and Mirsky, R
The Journal of physiology, ISSN 0022-3751, 07/2016, Volume 594, Issue 13, pp. 3521 - 3531
Schwann Cells - physiology | Myelin Sheath - physiology | Nervous System Diseases - physiopathology | Animals | Humans | Nerve Regeneration - physiology | Physiological aspects | Analysis | Neurophysiology | Spinal cord injuries | Index Medicus | Axon regeneration and remyelination in the peripheral and central nervous systems | Symposium Review | Symposium section reviews | Neurological Conditions, Disorders and Treatments | Cellular and Molecular Neuroscience
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2. Full Text Mechanisms of demyelination and remyelination in the young and aged brain following white matter stroke
by Marin, Miguel Alejandro and Carmichael, S. Thomas
Neurobiology of disease, ISSN 0969-9961, 06/2019, Volume 126, pp. 5 - 12
Stroke | Oligodendrocyte | Myelin | White matter stroke | Oligodendrocyte precursor cell, remyelination | Activity dependent remyelination | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Stroke - pathology | Demyelinating Diseases - physiopathology | Animals | Aging - physiology | Humans | Remyelination - physiology | Brain - pathology | Demyelinating Diseases - pathology | Aging - pathology | White Matter - pathology | Aged | Dementia | Index Medicus
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3. Full Text Myelin repair invivo is increased by targeting oligodendrocyte precursor cells with nanoparticles encapsulating leukaemia inhibitory factor (LIF)
by Rittchen, Sonja and Boyd, Amanda and Burns, Alasdair and Park, Jason and Fahmy, Tarek M and Metcalfe, Su and Williams, Anna
Biomaterials, ISSN 0142-9612, 07/2015, Volume 56, pp. 78 - 85
Multiple sclerosis | Leukaemia inhibitory factor | Myelin | Nanotherapy | Remyelination
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4. Full Text Butyrate suppresses demyelination and enhances remyelination
by Chen, Tong and Noto, Daisuke and Hoshino, Yasunobu and Mizuno, Miho and Miyake, Sachiko
Journal of neuroinflammation, ISSN 1742-2094, 08/2019, Volume 16, Issue 1, pp. 165 - 165
Neurosciences | Immunology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Cuprizone - toxicity | Oligodendroglia - metabolism | Butyrates - pharmacology | Mice, Inbred C57BL | Myelin Sheath - drug effects | Male | Gastrointestinal Microbiome - physiology | Demyelinating Diseases - metabolism | Demyelinating Diseases - prevention & control | Myelin Sheath - metabolism | Animals | Gastrointestinal Microbiome - drug effects | Oligodendroglia - drug effects | Cell Differentiation - drug effects | Remyelination - physiology | Butyrates - therapeutic use | Demyelinating Diseases - chemically induced | Mice | Anti-Bacterial Agents - toxicity | Remyelination - drug effects | Cell Differentiation - physiology | Organ Culture Techniques | Development and progression | Multiple sclerosis | Care and treatment | Myelination | Microbiota (Symbiotic organisms) | Health aspects | Index Medicus | Short-chain fatty acids | Oligodendrocyte | Demyelination | Remyelination
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5. Full Text Lineage tracing reveals dynamic changes in oligodendrocyte precursor cells following cuprizone‐induced demyelination
by Baxi, Emily G and DeBruin, Joseph and Jin, Jing and Strasburger, Hayley J and Smith, Matthew D and Orthmann‐Murphy, Jennifer L and Schott, Jason T and Fairchild, Amanda N and Bergles, Dwight E and Calabresi, Peter A
Glia, ISSN 0894-1491, 12/2017, Volume 65, Issue 12, pp. 2087 - 2098
oligodendrocyte progenitor cells | demyelination | multiple sclerosis | gray matter | remyelination | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Age Factors | RNA, Untranslated - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Hippocampus - drug effects | Cell Lineage - drug effects | RNA, Untranslated - genetics | Cell Differentiation - genetics | Oligodendrocyte Precursor Cells - drug effects | Demyelinating Diseases - chemically induced | Demyelinating Diseases - pathology | Cell Lineage - genetics | Disease Models, Animal | Cuprizone - toxicity | Receptor, Platelet-Derived Growth Factor alpha - metabolism | Mice, Inbred C57BL | Bacterial Proteins - genetics | Corpus Callosum - drug effects | Mice, Transgenic | Hippocampus - pathology | Monoamine Oxidase Inhibitors - toxicity | Animals | Receptor, Platelet-Derived Growth Factor alpha - genetics | Corpus Callosum - pathology | Remyelination - physiology | Bacterial Proteins - metabolism | Luminescent Proteins - genetics | Mice | Remyelination - drug effects | Luminescent Proteins - metabolism | Medicine, Experimental | Medical research | Multiple sclerosis | Platelet-derived growth factor | Cortex (cingulate) | Myelin | Central nervous system | Axonal plasticity | Substantia alba | Substantia grisea | Glial stem cells | Corpus callosum | Atrophy | Regeneration | Myelination | Cuprizone | Sheaths | Demyelination | Oligodendrocytes | Cells (biology) | In vivo methods and tests | Differentiation | Cortex (temporal) | Index Medicus
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6. Full Text Hormonal Regulation of Oligodendrogenesis II: Implications for Myelin Repair
by Breton, Jocelyn M and Long, Kimberly L. P and Barraza, Matthew K and Perloff, Olga S and Kaufer, Daniela
Biomolecules (Basel, Switzerland), ISSN 2218-273X, 02/2021, Volume 11, Issue 2, p. 290
peptides | steroids | hormones | oligodendrogenesis | remyelination
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7. Full Text Vitamin C promotes oligodendrocytes generation and remyelination
by Guo, Yu‐e and Suo, Na and Cui, Xue and Yuan, Qianting and Xie, Xin
Glia, ISSN 0894-1491, 07/2018, Volume 66, Issue 7, pp. 1302 - 1316
myelin | differentiation | vitamin C | oligodendrocyte progenitor cell | oligodendrocyte | remyelination | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Coculture Techniques | Neurons - cytology | Ganglia, Spinal - cytology | Brain - physiology | Dose-Response Relationship, Drug | Neuroprotective Agents - pharmacology | Oligodendroglia - drug effects | Oligodendroglia - physiology | Cuprizone | Time Factors | Neurons - physiology | Female | Oligodendroglia - cytology | Demyelinating Diseases - pathology | Neurons - drug effects | Cell Differentiation - physiology | Demyelinating Diseases - drug therapy | Disease Models, Animal | Demyelinating Diseases - physiopathology | Brain - cytology | Ascorbic Acid - analogs & derivatives | Mice, Inbred C57BL | Ganglia, Spinal - physiology | Antioxidants - pharmacology | Brain - drug effects | Animals | Cell Differentiation - drug effects | Remyelination - physiology | Ascorbic Acid - pharmacology | Brain - pathology | Mice | Remyelination - drug effects | Ganglia, Spinal - drug effects | Multiple sclerosis | Analysis | Alternative medicine | Dietary supplements | Vitamin C | Neurophysiology | Cell culture | Ascorbic acid | Demyelinating diseases | Myelin | Central nervous system | Glial stem cells | Molecular chains | Antioxidants | Time dependence | Myelination | Diet | Restoration | Sheaths | Demyelination | Oligodendrocytes | Differentiation | Index Medicus
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8. Full Text The Xenopus tadpole: An in vivo model to screen drugs favoring remyelination
by Mannioui, Abdelkrim and Vauzanges, Quentin and Fini, Jean Baptiste and Henriet, Esther and Sekizar, Somya and Azoyan, Loris and Thomas, Jean Léon and Pasquier, David Du and Giovannangeli, Carine and Demeneix, Barbara and Lubetzki, Catherine and Zalc, Bernard
Multiple sclerosis, ISSN 1352-4585, 10/2018, Volume 24, Issue 11, pp. 1421 - 1432
Multiple sclerosis | demyelination | Xenopus laevis | oligodendrocyte | Crispr/Cas9 | transgenic | gene editing | remyelination | Neurosciences | Clinical Neurology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Xenopus | Animals | Benzyl Compounds - pharmacology | Larva | Animals, Genetically Modified | Remyelination - physiology | Receptors, Lysosphingolipid - agonists | Female | Male | Azetidines - pharmacology | Remyelination - drug effects | Disease Models, Animal | CRISPR | Myelination | Optic nerve | Myelin | Demyelination | Oligodendrocytes | Nitroreductase | Green fluorescent protein | Metronidazole | Drug development | Sphingosine 1-phosphate | Index Medicus | Life Sciences
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9. Full Text After nerve injury, lineage tracing shows that myelin and Remak Schwann cells elongate extensively and branch to form repair Schwann cells, which shorten radically on remyelination
by Gomez-Sanchez, Jose A and Pilch, Kjara S and Van Der Lans, Milou and Fazal, Shaline V and Benito, Cristina and Wagstaff, Laura J and Mirsky, Rhona and Jessen, Kristjan R
The Journal of neuroscience, ISSN 0270-6474, 2017, Volume 37, Issue 37, pp. 9086 - 9099
Nerve | Regeneration | PNS | Schwann cell | Injury | Remyelination | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Animals | Neuronal Outgrowth | Peripheral Nerve Injuries - physiopathology | Nerve Regeneration - physiology | Peripheral Nerve Injuries - pathology | Female | Male | Mice, Transgenic | Schwann Cells - pathology | Mice | Myelin Sheath - metabolism | Scanning electron microscopy | Myelin | Schwann cells | Cell lineage | Electron microscopy | Myelination | Banded structure | Nerves | Repair | Elongation | Injuries | Columns (structural) | Index Medicus | injury | regeneration | nerve | remyelination
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