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Journal of the American College of Cardiology, ISSN 0735-1097, 10/2018, Volume 72, Issue 16, p. C109
Journal Article
Cardiovascular Research, ISSN 0008-6363, 09/2012, Volume 95, Issue 4, pp. 487 - 494
Innate mechanisms of inter-organ protection underlie the phenomenon of remote ischaemic preconditioning (RPc) in which episode(s) of ischaemia and reperfusion... 
Brain | Vagus nerve | Ischaemia/reperfusion injury | Preconditioning | Ischaemia | CARDIAC & CARDIOVASCULAR SYSTEMS | K-ATP CHANNELS | LIMB ISCHEMIA | MYOCARDIAL-ISCHEMIA | BRAIN-STEM | reperfusion injury | LENTIVIRAL VECTORS | CARDIOVASCULAR CONTROL | TRANSGENE EXPRESSION | RAT-HEART | AUTONOMIC NERVOUS-SYSTEM | Myocardial Infarction - genetics | Hindlimb | Receptors, G-Protein-Coupled - metabolism | Brain Stem - metabolism | Vagus Nerve - drug effects | Male | Receptors, Neuropeptide - metabolism | Drosophila Proteins - metabolism | Rhodopsin - metabolism | Recombinant Fusion Proteins - metabolism | Autonomic Fibers, Preganglionic - metabolism | Brain Stem - drug effects | Action Potentials | Myocardial Reperfusion Injury - pathology | Time Factors | Muscarinic Antagonists - pharmacology | Myocardial Infarction - pathology | Neuropeptides - pharmacology | Adenoviridae - genetics | Lentivirus - genetics | Myocardial Infarction - physiopathology | Neural Pathways - physiopathology | Myocardial Reperfusion Injury - genetics | Autonomic Fibers, Preganglionic - drug effects | Disease Models, Animal | Heart - innervation | Muscle, Skeletal - blood supply | Transduction, Genetic | Vagus Nerve - metabolism | Constriction | Rats | Receptors, Neuropeptide - genetics | Atropine - pharmacology | Myocardium - pathology | Myocardial Infarction - metabolism | Rats, Sprague-Dawley | Myocardial Reperfusion Injury - physiopathology | Vagus Nerve - physiopathology | Myocardial Reperfusion Injury - metabolism | Animals | Rhodopsin - genetics | Ischemic Preconditioning, Myocardial - methods | Brain Stem - physiopathology | Myocardial Infarction - prevention & control | Receptors, G-Protein-Coupled - genetics | Drosophila Proteins - genetics | Genetic Vectors | Myocardial Reperfusion Injury - prevention & control | Index Medicus | Original
Journal Article
Kidney International, ISSN 0085-2538, 12/2012, Volume 82, Issue 12, pp. 1271 - 1283
Autophagy is induced in renal tubular cells during acute kidney injury; however, whether this is protective or injurious remains controversial. We address this... 
acute kidney injury | autophagy | Atg7 | cisplatin | ischemia–reperfusion | Ischemia-reperfusion | APOPTOSIS | ACTIVATION | ACUTE-RENAL-FAILURE | PATHOLOGICAL ROLE | ischemia-reperfusion | CISPLATIN NEPHROTOXICITY | CELL-DEATH | PATHOPHYSIOLOGY | UROLOGY & NEPHROLOGY | MICE | BODY FORMATION | SELF-DIGESTION | Microtubule-Associated Proteins - genetics | Blood Urea Nitrogen | JNK Mitogen-Activated Protein Kinases - metabolism | Acute Kidney Injury - genetics | Autophagy - drug effects | Reperfusion Injury - blood | Chloroquine - pharmacology | Time Factors | Reperfusion Injury - chemically induced | Acute Kidney Injury - chemically induced | Microtubule-Associated Proteins - deficiency | Reperfusion Injury - genetics | Cytoprotection | Disease Models, Animal | Kidney Tubules, Proximal - pathology | Reperfusion Injury - pathology | Signal Transduction | Acute Kidney Injury - pathology | Acute Kidney Injury - blood | Mice, Inbred C57BL | Cells, Cultured | Tumor Suppressor Protein p53 - metabolism | Biomarkers - blood | Sirolimus - pharmacology | Acute Kidney Injury - prevention & control | Cisplatin | Mice, Knockout | Autophagy-Related Protein 7 | Animals | Reperfusion Injury - prevention & control | Kidney Tubules, Proximal - metabolism | Creatinine - blood | Mice | Enzyme Activation | Apoptosis | Kidney Tubules, Proximal - drug effects | Index Medicus
Journal Article
Kidney International, ISSN 0085-2538, 08/2012, Volume 82, Issue 4, pp. 412 - 427
Endothelial progenitor cells are known to reverse acute kidney injury by paracrine mechanisms. We previously found that microvesicles released from these... 
exosome | ischemia–reperfusion | acute kidney injury | ischemia-reperfusion | HORIZONTAL TRANSFER | STEM-CELLS | CONTRIBUTE | REGENERATION | FAILURE | EXPERIMENTAL GLOMERULONEPHRITIS | REPAIR | EPITHELIAL-CELLS | MESSENGER-RNA | UROLOGY & NEPHROLOGY | PLATELET-ACTIVATING-FACTOR | Chemotaxis, Leukocyte | Cell Proliferation | Epithelial Cells - metabolism | Kidney - blood supply | Capillaries - pathology | Kidney - pathology | Rats, Wistar | Endothelial Cells - transplantation | Cell-Derived Microparticles - transplantation | Male | MicroRNAs - metabolism | Acute Kidney Injury - genetics | Stem Cells - metabolism | Cell Hypoxia | Stem Cell Transplantation | Kidney - metabolism | Transfection | RNA Interference | Time Factors | Cell-Derived Microparticles - pathology | Cell-Derived Microparticles - metabolism | Kidney Tubules - pathology | Kidney Tubules - metabolism | Capillaries - metabolism | Reperfusion Injury - genetics | Reperfusion Injury - metabolism | Disease Models, Animal | Ribonuclease III - genetics | Ribonuclease III - metabolism | Reperfusion Injury - pathology | Acute Kidney Injury - pathology | Endothelial Cells - metabolism | Cells, Cultured | Gene Expression Regulation | Rats | Epithelial Cells - pathology | Acute Kidney Injury - prevention & control | Regeneration | Oligonucleotides - metabolism | Animals | Reperfusion Injury - prevention & control | Fibrosis | Stem Cells - pathology | Acute Kidney Injury - metabolism | Endothelial Cells - pathology | Apoptosis | Index Medicus | Cell proliferation | Intravenous administration | Paracrine signalling | mRNA | Leukocytes | Ribonuclease | Endothelial cells | Kidney | Angiogenesis | Ischemia | Stem cells | Hypoxia | miRNA | Capillaries | Injuries
Journal Article
Kidney International, ISSN 0085-2538, 04/2013, Volume 83, Issue 4, pp. 647 - 661
Ischemia–reperfusion activates innate immunity and sterile inflammation, resulting in acute kidney injury. Since pentraxin 3 (PTX3) regulates multiple aspects... 
sterile inflammation | hypoxia | macrophages | dendritic cells | risk factor | ischemia–reperfusion | ischemia-reperfusion | RESIDENT DENDRITIC CELLS | ISCHEMIA/REPERFUSION INJURY | COMPLEMENT ACTIVATION | ACUTE-RENAL-FAILURE | NEUTROPHIL RECRUITMENT | ISCHEMIA-REPERFUSION INJURY | EPITHELIAL-CELLS | APOPTOTIC CELLS | UROLOGY & NEPHROLOGY | SERUM-AMYLOID-P | CHEMOKINE RECEPTORS | C-Reactive Protein - administration & dosage | Kidney - blood supply | Kidney - pathology | P-Selectin - metabolism | Nerve Tissue Proteins - deficiency | Male | Neutrophil Infiltration | C-Reactive Protein - metabolism | Time Factors | Transendothelial and Transepithelial Migration | Kidney Tubular Necrosis, Acute - immunology | Renal Insufficiency, Chronic - genetics | C-Reactive Protein - deficiency | Acute Kidney Injury - immunology | Interleukin-6 - metabolism | Disease Models, Animal | Acute Kidney Injury - pathology | Acute Kidney Injury - blood | C-Reactive Protein - genetics | Mice, Knockout | Renal Insufficiency, Chronic - prevention & control | Fibrosis | Mice | Nerve Tissue Proteins - blood | Tumor Necrosis Factor-alpha - metabolism | Kidney - immunology | Acute Kidney Injury - genetics | Kidney Tubular Necrosis, Acute - prevention & control | Reperfusion Injury - blood | Atrophy | Leukocytes - immunology | Inflammation Mediators - metabolism | Female | Nerve Tissue Proteins - administration & dosage | Reperfusion Injury - genetics | Macrophages - immunology | Renal Insufficiency, Chronic - immunology | Reperfusion Injury - pathology | Mice, Inbred C57BL | Cells, Cultured | Nerve Tissue Proteins - genetics | Acute Kidney Injury - prevention & control | Recombinant Proteins - administration & dosage | Renal Insufficiency, Chronic - pathology | Nerve Tissue Proteins - metabolism | Renal Insufficiency, Chronic - blood | Injections | Animals | Reperfusion Injury - prevention & control | Reperfusion Injury - immunology | Kidney Tubular Necrosis, Acute - pathology | Index Medicus
Journal Article
Cardiovascular Research, ISSN 0008-6363, 05/2017, Volume 113, Issue 6, pp. 609 - 619
Aims The preconditioning-like infarct-sparing and anti-inflammatory effects of the peptide hormone relaxin following ischemic injury have been studied in the... 
LV function | eNOS | Ischemia-reperfusion injury | Serelaxin | Caspase-1 | SWINE MODEL | CARDIAC & CARDIOVASCULAR SYSTEMS | HYDROGEN-SULFIDE | PROTECTS | ACUTE HEART-FAILURE | FAMILY PEPTIDES | ROLES | DISEASE | NITRIC-OXIDE | ISCHEMIA-REPERFUSION | RECEPTORS | Inflammasomes - metabolism | Receptors, G-Protein-Coupled - metabolism | Caspase 1 - metabolism | Male | Nitric Oxide Synthase Type III - deficiency | Myocardial Reperfusion Injury - enzymology | Relaxin - pharmacology | Myocardial Reperfusion Injury - pathology | Time Factors | Myocardial Infarction - pathology | Nitrites - blood | Myocardial Infarction - physiopathology | Nitric Oxide Synthase Type III - metabolism | Disease Models, Animal | Myocardial Infarction - enzymology | NLR Family, Pyrin Domain-Containing 3 Protein - metabolism | Mice, Inbred C57BL | Ventricular Function, Left - drug effects | Cells, Cultured | Rats | Myocardium - pathology | Recombinant Proteins - pharmacology | Nitric Oxide Synthase Type III - genetics | Myocardial Reperfusion - methods | Myocardial Reperfusion Injury - physiopathology | Cardiovascular Agents - pharmacology | Mice, Knockout | Myocardial Reperfusion - adverse effects | Myocardium - enzymology | Animals | Signal Transduction - drug effects | Myocardial Infarction - prevention & control | Myocardial Reperfusion Injury - prevention & control | Index Medicus
Journal Article
Journal Article
Cardiovascular Research, ISSN 0008-6363, 07/2013, Volume 99, Issue 1, pp. 164 - 174
Nucleotide-binding oligomerization domain-Like Receptor with a Pyrin domain 3 (NLRP3) is considered necessary for initiating a profound sterile inflammatory... 
Heart | Myocardial infarction | Inflammasome | Ischaemia-reperfusion | IL-1β | Potassium - metabolism | Up-Regulation | Inflammasomes - metabolism | Cytoskeletal Proteins - genetics | NLR Family, Pyrin Domain-Containing 3 Protein | Caspase 1 - metabolism | Male | NF-kappa B - metabolism | Interleukin-1beta - genetics | RNA, Messenger - metabolism | Myocardial Reperfusion Injury - pathology | Time Factors | Interleukin-1beta - metabolism | Adenosine Triphosphate - metabolism | Myocardial Infarction - physiopathology | Toll-Like Receptors - metabolism | Myocardial Reperfusion Injury - genetics | Disease Models, Animal | Fibroblasts - metabolism | Myocardial Reperfusion Injury - immunology | Rats | Receptors, Cytoplasmic and Nuclear - genetics | Myocardial Infarction - metabolism | Myocardial Reperfusion Injury - physiopathology | Mice, Knockout | Fibroblasts - drug effects | Lipopolysaccharides - pharmacology | Fibroblasts - immunology | Heart Ventricles - metabolism | Mice | Interleukin-18 - genetics | Interleukin-18 - metabolism | Myocardial Reperfusion Injury - prevention & control | Heart Ventricles - drug effects | Receptors, Cytoplasmic and Nuclear - metabolism | Myocardial Infarction - genetics | Rats, Wistar | Ventricular Function, Left | Heart Ventricles - immunology | Myocardial Infarction - immunology | Cytoskeletal Proteins - deficiency | Myocardial Infarction - pathology | Heart Ventricles - pathology | Myocardial Contraction | Mice, Inbred C57BL | Cells, Cultured | Inflammasomes - genetics | Carrier Proteins - genetics | Myocardial Reperfusion Injury - metabolism | Animals | Apoptosis Regulatory Proteins | Carrier Proteins - metabolism | Heart Ventricles - physiopathology | CARD Signaling Adaptor Proteins | Apoptosis | Index Medicus
Journal Article
Journal of Pineal Research, ISSN 0742-3098, 10/2018, Volume 65, Issue 3, pp. e12503 - n/a
Journal Article
Journal Article
Journal Article