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1. Full Text Inhibition of MYC attenuates tumor cell self‐renewal and promotes senescence in SMARCB1‐deficient Group 2 atypical teratoid rhabdoid tumors to suppress tumor growth in vivo
by Alimova, Irina and Pierce, Angela and Danis, Etienne and Donson, Andrew and Birks, Diane K and Griesinger, Andrea and Foreman, Nicholas K and Santi, Mariarita and Soucek, Laura and Venkataraman, Sujatha and Vibhakar, Rajeev
International journal of cancer, ISSN 0020-7136, 04/2019, Volume 144, Issue 8, pp. 1983 - 1995
rhabdoid tumor | SMARCB1 | ATRT | MYC | SWI/SNF complex | Life Sciences & Biomedicine | Oncology | Science & Technology | Chromatin - metabolism | Humans | Cellular Senescence - drug effects | SMARCB1 Protein - genetics | Rhabdoid Tumor - pathology | Cell Self Renewal - drug effects | Cell Self Renewal - genetics | Bone Morphogenetic Proteins - metabolism | Cell Transformation, Neoplastic - genetics | Female | Gene Expression Regulation, Neoplastic - drug effects | Bone Morphogenetic Proteins - genetics | Gene Expression Regulation, Neoplastic - genetics | Cellular Senescence - genetics | Proto-Oncogene Proteins c-myc - metabolism | Azepines - pharmacology | Triazoles - pharmacology | Xenograft Model Antitumor Assays | Teratoma - pathology | Animals | Rhabdoid Tumor - genetics | Mice, Nude | Cell Line, Tumor | Teratoma - genetics | Mice | Proto-Oncogene Proteins c-myc - genetics | Chromatin - genetics | Cell culture | Brain | Senescence | Brain tumors | Xenotransplantation | Transgenic | Myc protein | Subgroups | Chromatin remodeling | Organic chemistry | Molecular modelling | Depletion | Clonal deletion | Xenografts | Tumorigenesis | Inhibition | Children | Pluripotency | Tumors | Index Medicus
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2. Full Text No small surprise – small cell carcinoma of the ovary, hypercalcaemic type, is a malignant rhabdoid tumour
by Foulkes, William D and Clarke, Blaise A and Hasselblatt, Martin and Majewski, Jacek and Albrecht, Steffen and McCluggage, W Glenn
The Journal of pathology, ISSN 0022-3417, 07/2014, Volume 233, Issue 3, pp. 209 - 214
SMARCB1 | rhabdoid tumour | small cell carcinoma of the ovary | hypercalcaemic type | Pathology | Oncology | Life Sciences & Biomedicine | Science & Technology | Immunohistochemistry | Carcinoma, Small Cell - genetics | Humans | Ovarian Neoplasms - pathology | Rhabdoid Tumor - pathology | Exome | Ovarian Neoplasms - genetics | SMARCB1 Protein | Carcinoma, Small Cell - chemistry | Female | Nuclear Proteins - genetics | DNA Helicases - genetics | Hypercalcemia - genetics | Genetic Predisposition to Disease | Ovarian Neoplasms - chemistry | Hypercalcemia - pathology | Genetic Association Studies | Biomarkers, Tumor - analysis | Rhabdoid Tumor - chemistry | Hypercalcemia - metabolism | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Sequence Analysis, DNA | Chromosomal Proteins, Non-Histone - genetics | Phenotype | Rhabdoid Tumor - genetics | Biomarkers, Tumor - genetics | Mutation | Carcinoma, Small Cell - pathology | Carcinoma | Ovarian cancer | Cancer | Index Medicus
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3. Full Text p53 Is a Master Regulator of Proteostasis in SMARCB1-Deficient Malignant Rhabdoid Tumors
by Carugo, Alessandro and Minelli, Rosalba and Sapio, Luigi and Soeung, Melinda and Carbone, Federica and Robinson, Frederick S and Tepper, James and Chen, Ziheng and Lovisa, Sara and Svelto, Maria and Amin, Samirkumar and Srinivasan, Sanjana and Del Poggetto, Edoardo and Loponte, Sara and Puca, Francesca and Dey, Prasenjit and Malouf, Gabriel G and Su, Xiaoping and Li, Liren and Lopez-Terrada, Dolores and Rakheja, Dinesh and Lazar, Alexander J and Netto, George J and Rao, Priya and Sgambato, Alessandro and Maitra, Anirban and Tripathi, Durga N and Walker, Cheryl L and Karam, Jose A and Heffernan, Timothy P and Viale, Andrea and Roberts, Charles W.M and Msaouel, Pavlos and Tannir, Nizar M and Draetta, Giulio F and Genovese, Giannicola
Cancer cell, ISSN 1535-6108, 02/2019, Volume 35, Issue 2, pp. 204 - 220.e9
SMARCB1 | autophagy | renal medullary carcinoma | MYC | embryonic mosaic GEM models | ER stress | BIRC5 | proteasome inhibitors | rhabdoid tumors | p53 | Oncology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Humans | Gene Expression Regulation, Neoplastic | SMARCB1 Protein - genetics | Male | Mice, 129 Strain | Rhabdoid Tumor - pathology | Autophagy - drug effects | Tumor Suppressor Protein p53 - genetics | Female | Antineoplastic Agents - pharmacology | Rhabdoid Tumor - metabolism | SMARCB1 Protein - deficiency | Tumor Cells, Cultured | Proteostasis - drug effects | Rhabdoid Tumor - drug therapy | Endoplasmic Reticulum Stress - drug effects | Proteasome Inhibitors - pharmacology | Signal Transduction | Mice, Inbred C57BL | Tumor Suppressor Protein p53 - metabolism | Cyclin-Dependent Kinase Inhibitor p16 - genetics | Tumor Suppressor Protein p53 - deficiency | Unfolded Protein Response | Proto-Oncogene Proteins c-myc - metabolism | Mice, Knockout | Animals | Rhabdoid Tumor - genetics | Cell Line, Tumor | Cyclin-Dependent Kinase Inhibitor p16 - metabolism | Proto-Oncogene Proteins c-myc - genetics | Tumor proteins | Analysis | Tumors | Index Medicus
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4. Full Text INI1-deficient tumors: Diagnostic features and molecular genetics
by Hollmann, Travis J and Hornick, Jason L
The American journal of surgical pathology, ISSN 0147-5185, 10/2011, Volume 35, Issue 10, pp. e47 - e63
malignant rhabdoid tumor | SMARCB1 | epithelioid sarcoma | schwannomatosis | SNF5 | soft tissue tumors | renal medullarycarcinoma | INI1 | Pathology | Life Sciences & Biomedicine | Surgery | Science & Technology | Myoepithelioma - metabolism | Kidney Neoplasms - genetics | Soft Tissue Neoplasms - diagnosis | Humans | Transcription Factors - deficiency | Carcinoma, Medullary - diagnosis | Carcinoma, Medullary - metabolism | Kidney Neoplasms - metabolism | DNA-Binding Proteins - deficiency | Kidney Neoplasms - diagnosis | DNA-Binding Proteins - metabolism | SMARCB1 Protein | Biomarkers, Tumor - metabolism | Sarcoma - diagnosis | Cytogenetic Analysis | Rhabdoid Tumor - metabolism | Sarcoma - genetics | Soft Tissue Neoplasms - genetics | Epithelioid Cells - pathology | Myoepithelioma - diagnosis | Chondrosarcoma - diagnosis | Chromosomal Proteins, Non-Histone - metabolism | Carcinoma, Medullary - genetics | Nerve Sheath Neoplasms - diagnosis | Soft Tissue Neoplasms - metabolism | Nerve Sheath Neoplasms - metabolism | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Chromosomal Proteins, Non-Histone - deficiency | Chromosomal Proteins, Non-Histone - genetics | Epithelioid Cells - metabolism | Immunohistochemistry - methods | Sarcoma - metabolism | Transcription Factors - metabolism | Rhabdoid Tumor - genetics | Rhabdoid Tumor - diagnosis | Index Medicus
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5. Full Text Treatment of Anaplastic Histology Wilms' Tumor: Results From the Fifth National Wilms' Tumor Study
by Jeffrey S. Dome and Cecilia A. Cotton and Elizabeth J. Perlman and Norman E. Breslow and John A. Kalapurakal and Michael L. Ritchey and Paul E. Grundy and Marcio Malogolowkin and J. Bruce Beckwith and Robert C. Shamberger and Gerald M. Haase and Max J. Coppes and Peter Coccia and Morris Kletzel and Robert M. Weetman and Milton Donaldson and Roger M. Macklis and Daniel M. Green
Journal of clinical oncology, ISSN 0732-183X, 05/2006, Volume 24, Issue 15, pp. 2352 - 2358
Tumors of the urinary system | Nephrology. Urinary tract diseases | Biological and medical sciences | Kidneys | Medical sciences | Tumors | Prognosis | Prospective Studies | Kidney - pathology | Humans | Child, Preschool | Infant | Male | Rhabdoid Tumor - pathology | Nephrectomy | Vincristine - administration & dosage | Female | Child | Infant, Newborn | Kidney Neoplasms - therapy | Doxorubicin - administration & dosage | Anaplasia | Etoposide - administration & dosage | Dactinomycin - administration & dosage | Radiotherapy | Wilms Tumor - pathology | Rhabdoid Tumor - therapy | Wilms Tumor - therapy | Cyclophosphamide | Sarcoma, Clear Cell - pathology | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Adolescent | Survival Analysis | Kidney Neoplasms - pathology | Neoplasm Staging | Sarcoma, Clear Cell - therapy | Index Medicus
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6. Full Text Current standards of care and future directions for “high-risk” pediatric renal tumors: Anaplastic Wilms tumor and Rhabdoid tumor
by Geller, James I., M.D
Urologic oncology, ISSN 1078-1439, 2016, Volume 34, Issue 1, pp. 50 - 56
Urology | Rhabdoid tumor | Pediatric renal cancer | Anaplastic Wilms tumor | p53 | P53 | Oncology | Life Sciences & Biomedicine | Urology & Nephrology | Science & Technology | Kidney Neoplasms - epidemiology | Prognosis | Standard of Care | Humans | Rhabdoid Tumor - pathology | Rhabdoid Tumor - epidemiology | Wilms Tumor - pathology | Rhabdoid Tumor - therapy | Wilms Tumor - therapy | Wilms Tumor - epidemiology | Carcinoma - epidemiology | Carcinoma - therapy | Kidney Neoplasms - pathology | Carcinoma - pathology | Child | Kidney Neoplasms - therapy | Sarcoma | Nephroblastoma | Pediatrics | Index Medicus
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7. Full Text Rhabdoid Tumor Predisposition Syndrome
by Sredni, Simone T and Tomita, Tadanori
Pediatric and developmental pathology, ISSN 1093-5266, 1/2015, Volume 18, Issue 1, pp. 49 - 58
Malignant rhabdoid tumors | Schwannomatosis | SMARCB1 | AT/RT | Rhab-doid tumor predisposition syndrome | SMARCA4 | Pathology | Pediatrics | Life Sciences & Biomedicine | Science & Technology | Immunohistochemistry | Age Factors | Humans | Child, Preschool | Family Health | Infant | Male | Neurofibromatoses - genetics | SMARCB1 Protein | Neurilemmoma - genetics | Germ-Line Mutation | Female | Nuclear Proteins - genetics | Child | DNA Helicases - genetics | Infant, Newborn | Diagnosis, Differential | Genetic Predisposition to Disease | Rhabdoid Tumor - classification | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Chromosomal Proteins, Non-Histone - genetics | Rhabdoid Tumor - genetics | Skin Neoplasms - genetics | Rhabdoid Tumor - diagnosis | Index Medicus
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