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Journal of Clinical Investigation, ISSN 0021-9738, 12/2007, Volume 117, Issue 12, pp. 3846 - 3856
Persistently activated or tyrosine-phosphorylated STAT3 (pSTAT3) is found in 50% of lung adenocarcinomas. pSTAT3 is found in primary adenocarcinomas and cell... 
RECEPTOR GENE-MUTATIONS | BREAST-CANCER | MEDICINE, RESEARCH & EXPERIMENTAL | SIGNALING PATHWAYS | CONSTITUTIVE ACTIVATION | EPIDERMAL-GROWTH-FACTOR | PROSTATE-CANCER CELLS | SQUAMOUS-CELL CARCINOMA | TYROSINE KINASES | NF-KAPPA-B | MAMMARY EPITHELIAL-CELLS | Janus Kinases | Neoplasm Transplantation | Receptor, Epidermal Growth Factor - genetics | Transcription, Genetic - drug effects | Adenocarcinoma - pathology | Interleukin-6 - antagonists & inhibitors | Cytokine Receptor gp130 - genetics | Humans | Lung Neoplasms - metabolism | Lung Neoplasms - pathology | Receptor, Epidermal Growth Factor - metabolism | Adenocarcinoma - metabolism | Female | Adenocarcinoma - genetics | Gene Expression Regulation, Neoplastic - drug effects | Phosphorylation - drug effects | Gene Expression Regulation, Neoplastic - genetics | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Lung Neoplasms - genetics | Cytokine Receptor gp130 - metabolism | Interleukin-6 - genetics | RNA, Small Interfering - pharmacology | Enzyme Inhibitors - pharmacology | Lung Neoplasms - therapy | Signal Transduction - genetics | Animals | Adenocarcinoma - therapy | Signal Transduction - drug effects | Mice, Nude | Cell Line, Tumor | Interleukin-6 - biosynthesis | Mice | Mutation | Transcription, Genetic - genetics | Research | Gene mutations | Lung cancer | Risk factors
Journal Article
Journal Article
Journal Article
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2013, Volume 131, Issue 4, pp. 1146 - 1156.e5
Background The generation of high-affinity antibodies requires the presence of a population of CD4+ T cells (follicular TH [TFH ] cells) in the lymph node... 
Allergy and Immunology | Human | naive B cells | signal transducer and activator of transcription 3 deficiency | T cells | cell activation | IMMUNOGLOBULIN PRODUCTION | IL-21 RECEPTOR | IMMUNOLOGY | CUTTING EDGE | HYPER-IGE SYNDROME | CXC CHEMOKINE RECEPTOR-5 | MEMORY B-CELLS | HUMAN NAIVE | FOLLICULAR-HELPER-CELLS | ALLERGY | COMMON GAMMA-CHAIN | ANTIBODY-RESPONSES | Job Syndrome - immunology | T-Lymphocyte Subsets - immunology | Job Syndrome - genetics | Immunoglobulin A - biosynthesis | Humans | Middle Aged | Male | Leukocyte Common Antigens - immunology | Immunoglobulin G - biosynthesis | Immunoglobulin G - immunology | Interleukins - immunology | STAT3 Transcription Factor - deficiency | Adult | Female | Cell Differentiation | B-Lymphocytes - pathology | Interleukins - biosynthesis | Child | Job Syndrome - metabolism | B-Lymphocytes - metabolism | STAT3 Transcription Factor - genetics | Gene Expression | Receptors, CXCR5 - genetics | Signal Transduction | Lymphocyte Activation | Receptors, CXCR5 - immunology | B-Lymphocytes - immunology | T-Lymphocyte Subsets - pathology | T-Lymphocyte Subsets - metabolism | Immunoglobulin A - immunology | Adolescent | Lymphocyte Count | Heterozygote | Leukocyte Common Antigens - genetics | Job Syndrome - pathology | Mutation | STAT3 Transcription Factor - immunology | Antigens | Immunoglobulins | Cytokines | Lymphocytes | Ligands | Blood | Immune system
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, p. e92478
Raf kinase inhibitor protein (RKIP) is a member of the phosphatidylethanolamine-binding-protein (PEBP) family that modulates the action of many kinases... 
PLASMA-MEMBRANES | MULTIPLE-MYELOMA | MOLECULAR TARGETS | EPITHELIAL-CELLS | TUMOR PROGRESSION | INDUCED APOPTOSIS | CONSTITUTIVE ACTIVATION | COLORECTAL-CANCER | MULTIDISCIPLINARY SCIENCES | MYELOMA CELLS | ANTITUMOR-ACTIVITY | Neoplasm Transplantation | Interleukin-6 - antagonists & inhibitors | Phosphatidylethanolamine Binding Protein - biosynthesis | Tubulin Modulators - pharmacology | Humans | Phosphatidylethanolamine Binding Protein - metabolism | Male | Prostatic Neoplasms - physiopathology | Janus Kinase 1 - antagonists & inhibitors | Breast Neoplasms - physiopathology | Animals | Transfection | Interleukin-6 - pharmacology | Estrenes - pharmacology | Cell Line, Tumor | Female | Cell Proliferation - drug effects | Mice | STAT3 Transcription Factor - antagonists & inhibitors | STAT3 Transcription Factor - metabolism | Tyrosine | Analysis | Breast cancer | Genetic transcription | Metastasis | Prostate cancer | Stomach cancer | Phosphotransferases | Protein binding | Apoptosis | Adenocarcinoma | Biotechnology | Transcription factors | Phosphorylation | Mesenchyme | Serine | Raf protein | Activation | Kinases | Medical schools | Cell adhesion & migration | Metastases | Interleukin 6 | Proteins | Rodents | Transcription activation | Xenografts | Cell cycle | Janus kinase | Inhibition | Growth factors | Transducers | Phosphatidylethanolamine | Cytokines | Stat3 protein | Tumor cell lines | Src protein | Medicine | Hospitals | Inhibitors | Medical prognosis | Breast | Laboratory animals | Prostate | Cancer
Journal Article
Journal of Pharmacological Sciences, ISSN 1347-8613, 2011, Volume 115, Issue 1, pp. 84 - 88
Journal Article