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Clinical, cosmetic and investigational dermatology, ISSN 1178-7015, 2019, Volume 12, pp. 497 - 508
Human JAKs are responsible for generating docking sites for human SSTAT phosphorylation. The role of JAKs in psoriasis pathogenesis has not been clearly... 
Immunoglobulins | Psoriasis | Disease | Cytokines | Dermatology | Pathogenesis | Kinases | Patients | Medicine | Studies | Proteins | Signal transduction | Histopathology | Skin | immunohistochemistry | psoriasis | STAT3 | JAK1
Journal Article
Journal of Allergy and Clinical Immunology, ISSN 0091-6749, 03/2007, Volume 119, Issue 3, pp. 542 - 543
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2016, Volume 139, Issue 5, pp. 1667 - 1676
Background Mesenchymal stem cells (MSCs) display a therapeutic plasticity because of their ability to modulate immunity, foster tissue repair, and... 
Allergy and Immunology | T cells | immunomodulation | IFN-γ | mammalian target of rapamycin | signal transducer and activator of transcription 1 | mesenchymal stem cells | signal transducer and activator of transcription 3 | TGF-BETA | STAT3 | SELF-RENEWAL | IMMUNOLOGY | T cells | MTOR | IFN-gamma | INTERFERON-GAMMA | ALLERGY | IMMUNOSUPPRESSION | DISEASE | STROMAL CELLS | MOLECULAR-BASIS | EXPRESSION | Mesenchymal Stromal Cells - drug effects | Humans | Cells, Cultured | Hypersensitivity, Delayed - immunology | STAT1 Transcription Factor - immunology | Mesenchymal Stromal Cells - immunology | TOR Serine-Threonine Kinases - immunology | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Cell Proliferation - drug effects | Mice | STAT3 Transcription Factor - immunology | Interferon-gamma - pharmacology | Brain | Cell culture | Polarization | Regulations | Graft-versus-host reaction | Transcription factors | Disease | Anergy | Embryo cells | AKT protein | Inactivation | B7 antigen | Proteins | Connective tissues | Signal transduction | Cell growth | Filters | Mathematical analysis | Bone marrow | Inhibition | Growth factors | Stability | Gels | Exposure | Gene expression | Mammals | Metabolism | 1-Phosphatidylinositol 3-kinase | Gene silencing | Dilution | Bone mass | Inhibitors | Antigen-presenting cells | Nitric oxide | Stem cells | Protein-serine/threonine kinase | Biotechnology | Phosphorylation | Leukemia | Medical services | Antibodies | Homology | Lymphocytes T | Infections | Activation | Kinases | Experiments | Machinery | Heterogeneity | Biological effects | Control | Lymphocytes | Actin | Plasticity | Nutrients | Immune system | Incubation | Immune response (cell-mediated) | Pharmacology | Inflammation | Ribonucleic acid--RNA | Contamination | Lymphocytes B | Protein kinase | Interferon | Differentiation | Apoptosis
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2007, Volume 119, Issue 3, pp. 529 - 541
Journal Article
Journal Article
International Journal of Biochemistry and Cell Biology, ISSN 1357-2725, 2004, Volume 36, Issue 11, pp. 2120 - 2124
Journal Article
Current opinion in pediatrics, ISSN 1040-8703, 12/2018, Volume 30, Issue 6, pp. 821 - 828
Journal Article
Biochemistry and Cell Biology, ISSN 0829-8211, 12/2009, Volume 87, Issue 6, pp. 825 - 833
Signal transducer and activator of transcription protein 3 (STAT3) is a latent cytosolic transcription factor that is widely recognized as being a master... 
small molecule inhibitors | médicaments anticancéreux | petites molécules inhibitrices | agents peptidomimétiques | signal transducer and activator of transcription protein 3 | interactions protéine-protéine | peptidomimetics | anti-cancer drugs | transducteur de signaux et activateur de la transcription STAT3 | protein-protein interactions | Signal transducer and activator of transcription protein 3 | Small molecule inhibitors | Peptidomimetics | Protein-protein interactions | Anti-cancer drugs | CANCER-CELLS | RESVERATROL | 3,4,5,4'-TETRAMETHOXYSTILBENE DMU-212 | SOLID-PHASE SYNTHESIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | LYMPHOMA KINASE ALK | DIMERIZATION | CELL BIOLOGY | POTENT ANTITUMOR-ACTIVITY | JSI-124 CUCURBITACIN-I | INHIBITOR | MALIGNANT-CELLS | Neoplasms - metabolism | Cytokine Receptor gp130 - metabolism | Peptides - chemistry | Cytokine Receptor gp130 - genetics | Cytokine Receptor gp130 - chemistry | Humans | Transcriptional Activation | Peptides - genetics | Peptide Library | Peptides - metabolism | Neoplasms - genetics | Signal Transduction - physiology | Molecular Structure | Drug Evaluation, Preclinical | STAT3 Transcription Factor - antagonists & inhibitors | Oncogenes | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Physiological aspects | Research | DNA binding proteins | Tumors | Apoptosis | STAT proteins
Journal Article