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Nature, ISSN 0028-0836, 12/2011, Volume 480, Issue 7377, pp. 387 - 390
Journal Article
Journal Article
Journal of the American Chemical Society, ISSN 0002-7863, 04/2014, Volume 136, Issue 17, pp. 6355 - 6361
Journal Article
European Journal of Cancer, ISSN 0959-8049, 2015, Volume 51, Issue 18, pp. 2792 - 2799
Abstract Background Acquired resistance to BRAF inhibitors (BRAFi) is a near-universal phenomenon caused by numerous genetic and non-genetic alterations. In... 
Hematology, Oncology and Palliative Medicine | Resistance | Vemurafenib | NRAS | Splice | Acquired | Dabrafenib | BRAF | MEK1 | MAPK | Meta-analysis | SURVIVAL | RAF INHIBITION | METASTATIC MELANOMA | THERAPY | ONCOLOGY | PATHWAY | COBIMETINIB | B-RAF | MUTATIONS | Skin Neoplasms - drug therapy | United States | Humans | Middle Aged | Melanoma - enzymology | Male | Antineoplastic Agents - therapeutic use | Skin Neoplasms - enzymology | Time Factors | DNA Mutational Analysis | Melanoma - genetics | Skin Neoplasms - mortality | Biomarkers, Tumor - metabolism | Female | Biomarkers, Tumor - antagonists & inhibitors | Proto-Oncogene Proteins B-raf - metabolism | Skin Neoplasms - pathology | Genetic Predisposition to Disease | Europe | Risk Factors | Kaplan-Meier Estimate | Proportional Hazards Models | Treatment Outcome | Melanoma - pathology | Disease Progression | Proto-Oncogene Proteins B-raf - antagonists & inhibitors | Disease-Free Survival | Drug Resistance, Neoplasm - genetics | Phenotype | Signal Transduction - drug effects | Protein Kinase Inhibitors - therapeutic use | Proto-Oncogene Proteins B-raf - genetics | Melanoma - drug therapy | Skin Neoplasms - genetics | Biomarkers, Tumor - genetics | Mutation | Australia | Melanoma - mortality | splice | vemurafenib | meta-analysis | resistance | amplification | spectrum | MEK2 | dabrafenib | acquired
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 04/2012, Volume 287, Issue 16, pp. 13228 - 13238
Journal Article