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Cancer cell, ISSN 1535-6108, 2010, Volume 17, Issue 6, pp. 547 - 559
In mice, Lkb1 deletion and activation of Kras G12D results in lung tumors with a high penetrance of lymph node and distant metastases. We analyzed these... 
CELLCYCLE | SIGNALING | Oncology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Lung Neoplasms - drug therapy | Protein-Serine-Threonine Kinases - deficiency | Protein-Tyrosine Kinases - metabolism | Proto-Oncogene Proteins p21(ras) - genetics | Genomics | Humans | Lung Neoplasms - metabolism | Gene Expression Profiling | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Cell Movement - genetics | Phosphorylation - genetics | RNA Interference | Gene Expression Regulation, Neoplastic - genetics | MAP Kinase Kinase 1 - antagonists & inhibitors | Carcinoma, Non-Small-Cell Lung - metabolism | Signal Transduction - genetics | Enzyme Inhibitors - therapeutic use | Focal Adhesion Protein-Tyrosine Kinases - antagonists & inhibitors | Focal Adhesion Protein-Tyrosine Kinases - genetics | Focal Adhesions - genetics | Signal Transduction - drug effects | Mice, Nude | Cell Line, Tumor | Mice | TOR Serine-Threonine Kinases | src-Family Kinases - genetics | Protein-Tyrosine Kinases - antagonists & inhibitors | ras Proteins - genetics | Lung Neoplasms - pathology | Cell Transdifferentiation - genetics | Protein-Tyrosine Kinases - genetics | Neoplasm Metastasis - drug therapy | Mice, Mutant Strains | Protein-Serine-Threonine Kinases - antagonists & inhibitors | src-Family Kinases - metabolism | Female | Drug Therapy, Combination | Lung Neoplasms - genetics | Cell Adhesion - genetics | Carcinoma, Non-Small-Cell Lung - genetics | Focal Adhesion Protein-Tyrosine Kinases - metabolism | Intracellular Signaling Peptides and Proteins - antagonists & inhibitors | src-Family Kinases - antagonists & inhibitors | Protein-Serine-Threonine Kinases - genetics | Proto-Oncogene Proteins - genetics | Up-Regulation - genetics | Xenograft Model Antitumor Assays | Neoplasm Metastasis - genetics | Animals | MAP Kinase Kinase 2 - antagonists & inhibitors | Protein Kinase Inhibitors - therapeutic use | Focal Adhesions - metabolism | Proteomics | Protein Kinase Inhibitors - pharmacology | Oncology, Experimental | Analysis | Lung cancer | Development and progression | Metastasis | Research | Cancer | Index Medicus
Journal Article
Journal Article
Cell death and differentiation, ISSN 1350-9047, 09/2012, Volume 19, Issue 9, pp. 1459 - 1469
Src, the canonical member of the non-receptor family of tyrosine kinases, is deregulated in numerous cancers, including colon and breast cancers... 
Erk1/2 | proteasome | apoptosis | Bik | Src | Bim | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Neoplasms - metabolism | NIH 3T3 Cells | Humans | Mitochondrial Proteins - genetics | raf Kinases - metabolism | Oncogene Protein p21(ras) - metabolism | MAP Kinase Signaling System | MAP Kinase Kinase 1 - genetics | Neoplasms - genetics | Mitochondrial Proteins - metabolism | Proteolysis | Mitogen-Activated Protein Kinase 1 - genetics | src-Family Kinases - metabolism | Apoptosis Regulatory Proteins - genetics | Membrane Proteins - metabolism | raf Kinases - genetics | MAP Kinase Kinase 2 - genetics | Mitogen-Activated Protein Kinase 3 - genetics | Membrane Proteins - genetics | Enzyme Inhibitors - pharmacology | MAP Kinase Kinase 1 - metabolism | MAP Kinase Kinase 2 - metabolism | Enzyme Activation - drug effects | Apoptosis Regulatory Proteins - metabolism | Drug Resistance, Neoplasm - genetics | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Adaptor Proteins, Signal Transducing - genetics | Cell Line, Tumor | Thapsigargin - pharmacology | Mice | Adaptor Proteins, Signal Transducing - metabolism | Enzyme Activation - genetics | Neoplasms - pathology | src-Family Kinases - genetics | Staurosporine - pharmacology | Drug Resistance, Neoplasm - drug effects | Mitogen-Activated Protein Kinase 1 - metabolism | Oncogene Protein p21(ras) - genetics | Index Medicus | Erk1 | Original Paper
Journal Article
PLoS pathogens, ISSN 1553-7374, 08/2009, Volume 5, Issue 8, pp. e1000559 - e1000559
The intraerythrocytic parasite Plasmodium-the causative agent of malaria-produces an inorganic crystal called hemozoin (Hz) during the heme detoxification... 
Parasitology | Life Sciences & Biomedicine | Microbiology | Science & Technology | Virology | Potassium - metabolism | Reactive Oxygen Species - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Protein-Tyrosine Kinases - metabolism | Humans | Caspase 1 - metabolism | Male | Caspase 1 - immunology | Intracellular Signaling Peptides and Proteins - metabolism | Hemeproteins - immunology | Signal Transduction - immunology | Calcium-Binding Proteins - immunology | Interleukin-1beta - metabolism | src-Family Kinases - metabolism | Cytoskeletal Proteins - metabolism | Female | Interleukin-1beta - biosynthesis | Phosphorylation - immunology | Malaria - metabolism | Syk Kinase | Carrier Proteins - immunology | Disease Models, Animal | Neutrophil Infiltration - immunology | Calcium-Binding Proteins - metabolism | Carrier Proteins - physiology | Apoptosis Regulatory Proteins - immunology | Plasmodium chabaudi - chemistry | Hemeproteins - metabolism | Malaria - immunology | Mice, Inbred C57BL | Cathepsin B - metabolism | Inflammation - immunology | Plasmodium chabaudi - metabolism | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Animals | Carrier Proteins - metabolism | Cytoskeletal Proteins - immunology | CARD Signaling Adaptor Proteins | Malaria - parasitology | Proteomics | HSP90 Heat-Shock Proteins - metabolism | Hemeproteins - physiology | Mice | Phagocytosis | Hostages | Inflammation | Malaria | Phosphotransferases | Index Medicus | Medical research | Salmonella | Phosphorylation | Lipids | Parasites | Experiments | Fever | Tropical diseases
Journal Article
Journal Article
American Journal of Physiology: Cell Physiology, ISSN 1522-1563, 11/2007, Volume 293, Issue 5, pp. C1489 - C1497
.... In a dose- and time-dependent manner, ouabain activated Akt and phosphorylation of its substrates mammalian target of rapamycin and glycogen synthase kinase in neonatal rat cardiac myocytes... 
Heart failure | Growth factor receptor | Sodium pump | ATPase | Cardiac glycosides | Physiology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Protein Kinases - metabolism | Phosphorylation | Phosphatidylinositol Phosphates - metabolism | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Ouabain - toxicity | Phosphatidylinositol 3-Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Dose-Response Relationship, Drug | Myocytes, Cardiac - enzymology | Receptor, Epidermal Growth Factor - metabolism | Time Factors | Enzyme Inhibitors - toxicity | src-Family Kinases - metabolism | MAP Kinase Kinase Kinases - antagonists & inhibitors | Proto-Oncogene Proteins c-akt - metabolism | Animals, Newborn | Sodium-Potassium-Exchanging ATPase - antagonists & inhibitors | Cell Size - drug effects | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | src-Family Kinases - antagonists & inhibitors | Cells, Cultured | Rats | MAP Kinase Kinase Kinases - metabolism | Rats, Sprague-Dawley | Myocytes, Cardiac - pathology | Sodium-Potassium-Exchanging ATPase - metabolism | Animals | Myocytes, Cardiac - drug effects | Mitogen-Activated Protein Kinase 3 - metabolism | Signal Transduction - drug effects | Myocytes, Cardiac - metabolism | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Protein Kinase Inhibitors - pharmacology | TOR Serine-Threonine Kinases | Glycogen Synthase Kinases - metabolism | Hypertrophy | Mitogen-Activated Protein Kinase 1 - metabolism | Index Medicus
Journal Article