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Journal of Clinical Investigation, ISSN 0021-9738, 05/2018, Volume 128, Issue 5, pp. 2064 - 2075
Receptor interacting protein kinase 1 (RIPK1) has important kinase-dependent and kinase-independent scaffolding functions that activate or prevent apoptosis or... 
MEDICINE, RESEARCH & EXPERIMENTAL | TUMOR-NECROSIS-FACTOR | MLKL | PHASE-III | IN-VIVO | SEPTIC SHOCK | DOUBLE-BLIND | PROGRAMMED NECROSIS | TNF-ALPHA | SEVERE SEPSIS | CELL-DEATH | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Amino Acid Chloromethyl Ketones - toxicity | Liver - pathology | Liver - enzymology | Endothelium, Vascular - injuries | Mice, 129 Strain | Endothelium, Vascular - enzymology | Tumor Necrosis Factor-alpha - toxicity | Systemic Inflammatory Response Syndrome - genetics | Necrosis | Receptor-Interacting Protein Serine-Threonine Kinases - genetics | Systemic Inflammatory Response Syndrome - pathology | Animals | Mice, Mutant Strains | Systemic Inflammatory Response Syndrome - enzymology | Endothelium, Vascular - pathology | Mice | Systemic Inflammatory Response Syndrome - chemically induced | Hematopoietic Stem Cells | Care and treatment | Tumor necrosis factor | Physiological aspects | Development and progression | Sepsis | Inflammation | Models | Protein kinases | Health aspects | Apoptosis | Shock | Phosphorylation | Liver | Lethality | Kinases | Clotting | Proteins | Intestine | Rodents | Cell survival | Bacterial infections | Cytokines | Neutrophils | MAP kinase | Permeability | Trauma | Endothelial cells | Hemopoiesis | Endothelium | Protein kinase | Chemokines | Hypothermia
Journal Article
Journal Article
Immunity, ISSN 1074-7613, 2011, Volume 35, Issue 6, pp. 908 - 918
Journal Article
Nature, ISSN 0028-0836, 03/2010, Volume 464, Issue 7285, pp. 104 - 107
Injury causes a systemic inflammatory response syndrome (SIRS) that is clinically much like sepsis. Microbial pathogen-associated molecular patterns (PAMPs)... 
SHOCK | SIRS | OPERATED CALCIUM INFLUX | HUMAN NEUTROPHILS | PEPTIDES | MODELS | MOBILIZATION | CHEMOTAXIS | MULTIDISCIPLINARY SCIENCES | RECEPTORS | TRAUMA | Liver - pathology | Phosphorylation | Humans | Male | Mitochondria - immunology | Liver - injuries | N-Formylmethionine Leucyl-Phenylalanine - metabolism | Liver - immunology | Systemic Inflammatory Response Syndrome - blood | Muscle, Skeletal - immunology | Sepsis - metabolism | Receptors, Formyl Peptide - metabolism | Systemic Inflammatory Response Syndrome - immunology | Neutrophils - metabolism | Calcium Signaling | Femur - injuries | Sepsis - immunology | CpG Islands - immunology | Neutrophils - enzymology | Wounds and Injuries - pathology | Fractures, Bone - immunology | Wounds and Injuries - blood | Cells, Cultured | Neutrophils - immunology | Wounds and Injuries - complications | Rats | Wounds and Injuries - immunology | Acute Lung Injury - pathology | DNA, Mitochondrial - blood | Rats, Sprague-Dawley | DNA, Mitochondrial - immunology | Immunity, Innate - immunology | Systemic Inflammatory Response Syndrome - pathology | Animals | Mitochondria - secretion | Acute Lung Injury - immunology | Fractures, Bone - pathology | Systemic Inflammatory Response Syndrome - complications | Muscle, Skeletal - pathology | Toll-Like Receptor 9 - metabolism | Sepsis - microbiology | Mitogen-Activated Protein Kinases - metabolism | N-Formylmethionine Leucyl-Phenylalanine - immunology | Infection | Peptides | Physiological aspects | Mitochondrial DNA | Inflammation | Research | Risk factors | Fractures | Bacterial proteins | Rheumatoid arthritis | Trauma | Deoxyribonucleic acid--DNA
Journal Article
Trends in Biotechnology, ISSN 0167-7799, 2016, Volume 34, Issue 6, pp. 470 - 482
Journal Article