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Kidney International, ISSN 0085-2538, 06/2012, Volume 81, Issue 12, pp. 1226 - 1238
Thrombospondin-1 (TSP-1) is an endogenous activator of transforming growth factor-β (TGF-β), and an anti-angiogenic factor, which may prevent kidney repair.... 
renal fibrosis | TGF-β | renal progression | Renal fibrosis | SUBTOTALLY NEPHRECTOMIZED RATS | DIABETIC-NEPHROPATHY | MATRIX METALLOPROTEINASES | TGF-beta | IMPAIRED ANGIOGENESIS | IN-VIVO | RECEPTOR ANTAGONISM | TUBULOINTERSTITIAL FIBROSIS | UROLOGY & NEPHROLOGY | FIBROTIC RENAL-DISEASE | ENDOTHELIAL GROWTH-FACTOR | Thrombospondin 1 - deficiency | Ureteral Obstruction - complications | Cell Proliferation | Kidney - blood supply | Capillaries - pathology | Kidney - pathology | Nephritis - genetics | Transforming Growth Factor beta1 - metabolism | Collagen Type III - metabolism | Male | Nephritis - prevention & control | Nephritis - pathology | Ureteral Obstruction - metabolism | Vascular Endothelial Growth Factor A - metabolism | Kidney Diseases - genetics | Atrophy | Kidney - metabolism | Nephrectomy | Time Factors | Thrombospondin 1 - genetics | Inflammation Mediators - metabolism | Ureteral Obstruction - genetics | Chemokine CCL2 - metabolism | Capillaries - metabolism | Disease Models, Animal | Kidney Diseases - metabolism | Signal Transduction | Kidney Diseases - pathology | Gene Expression Regulation | Nephritis - etiology | Rats | Rats, Sprague-Dawley | Mice, Knockout | Animals | Ureteral Obstruction - pathology | Fibrosis | Nephritis - metabolism | Mice | Kidney Diseases - etiology | Thrombospondin 1 - metabolism | Chronic Disease | Apoptosis | Kidney Diseases | Vascular Endothelial Growth Factor A | Nephritis | Genomics | Kidney | Life Sciences | Ureteral Obstruction | Capillaries | Inflammation Mediators | Transforming Growth Factor beta1 | Biochemistry, Molecular Biology | Thrombospondin 1 | Collagen Type III | Chemokine CCL2
Journal Article
Circulation Research, ISSN 0009-7330, 07/2015, Volume 117, Issue 2, pp. 129 - 141
Journal Article
Journal of Thrombosis and Haemostasis, ISSN 1538-7933, 04/2018, Volume 16, Issue 4, pp. 791 - 801
Journal Article
Journal Article
Cell, ISSN 0092-8674, 2005, Volume 120, Issue 3, pp. 421 - 433
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 03/2011, Volume 31, Issue 3, pp. 551 - 559
OBJECTIVE—We examined whether plasma levels of angiogenic factors are altered in plasma of patients with peripheral arterial disease (PAD) and whether these... 
arterial thrombosis | angiogenesis | endothelial progenitor cells | LIMB ISCHEMIA | CORD BLOOD | GENE POLYMORPHISM | IN-VITRO | LEG ISCHEMIA | PAR-1 ACTIVATION | PERIPHERAL VASCULAR DISEASE | GROWTH-FACTOR | HEMATOLOGY | UP-REGULATION | INCREASED VASCULAR DENSITY | OUTGROWTH CELLS | Angiogenic Proteins - genetics | Thrombospondin 1 - deficiency | Hindlimb | Cell Proliferation | Peripheral Arterial Disease - surgery | Humans | Endothelial Cells - transplantation | Stem Cells - metabolism | Case-Control Studies | Stem Cell Transplantation | Pregnancy Proteins - blood | RNA Interference | Thrombospondin 1 - genetics | Peripheral Arterial Disease - blood | Disease Models, Animal | Muscle, Skeletal - blood supply | Angiogenic Proteins - deficiency | Angiogenic Proteins - administration & dosage | Endothelial Cells - metabolism | Mice, Inbred C57BL | Cells, Cultured | Vascular Endothelial Growth Factor A - blood | Ischemia - metabolism | Treatment Outcome | Biomarkers - blood | CD47 Antigen - metabolism | Ischemia - physiopathology | Cell Adhesion | Mice, Knockout | Receptors, CXCR4 - metabolism | Peripheral Arterial Disease - physiopathology | Phenotype | Animals | Chemokine CXCL12 - metabolism | Angiogenic Proteins - blood | Thrombospondin 1 - administration & dosage | Placenta Growth Factor | Ischemia - surgery | Mice | Neovascularization, Physiologic | Thrombospondin 1 - blood
Journal Article
Mucosal Immunology, ISSN 1933-0219, 07/2015, Volume 8, Issue 4, pp. 896 - 905
Journal Article
Aging Cell, ISSN 1474-9718, 2011, Volume 10, Issue 5, pp. 769 - 779
Journal Article
Nature Communications, ISSN 2041-1723, 05/2017, Volume 8, Issue 1, p. 15494
Journal Article