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Nature cell biology, ISSN 1476-4679, 2011, Volume 13, Issue 10, pp. 1202 - 1213
Angiogenesis, the growth of new blood vessels, involves specification of endothelial cells to tip cells and stalk cells, which is controlled by Notch signalling, whereas vascular endothelial growth factor receptor (VEGFR... 
LYMPHATIC ENDOTHELIUM | KINASE-ACTIVITY | VASCULAR NETWORK FORMATION | ENDOTHELIAL-GROWTH-FACTOR | FACTOR-C | MOUSE EMBRYOS | EXTRACELLULAR-MATRIX | MONOCLONAL-ANTIBODY | TUMOR ANGIOGENESIS | FACTOR RECEPTOR-3 | CELL BIOLOGY | Neovascularization, Physiologic - drug effects | Human Umbilical Vein Endothelial Cells - metabolism | Receptors, Notch - metabolism | Humans | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Receptors, Notch - antagonists & inhibitors | Transfection | RNA Interference | Time Factors | Forkhead Transcription Factors - metabolism | Vascular Endothelial Growth Factor D - genetics | Phosphatidylinositol 3-Kinase - metabolism | Vascular Endothelial Growth Factor Receptor-3 - deficiency | Transduction, Genetic | Retinal Vessels - metabolism | Endothelial Cells - metabolism | Rhombencephalon - blood supply | Gene Expression Regulation | Vascular Endothelial Growth Factor Receptor-3 - metabolism | Forkhead Transcription Factors - genetics | Vascular Endothelial Growth Factor C - genetics | Mice, Knockout | Antibodies - pharmacology | Macrophages - metabolism | Vascular Endothelial Growth Factor D - metabolism | Animals | Signal Transduction - drug effects | Vascular Endothelial Growth Factor B - metabolism | Vascular Endothelial Growth Factor C - metabolism | Cell Line, Tumor | Neovascularization, Pathologic - genetics | Vascular Endothelial Growth Factor B - genetics | Mice | Neovascularization, Pathologic - metabolism | Protein Kinase Inhibitors - pharmacology | Vascular Endothelial Growth Factor Receptor-3 - genetics | Endothelial Cells - drug effects | Physiological aspects | Blood vessels | Cellular signal transduction | Growth factor receptors | Research | Vascular endothelial growth factor
Journal Article
Journal Article
Blood, ISSN 0006-4971, 2016, Volume 127, Issue 14, pp. 1780 - 1789
Journal Article
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 0027-8424, 2/1999, Volume 96, Issue 4, pp. 1603 - 1608
... -catenin and, subsequently, to increased transcriptional activity of the β -catenin-T cell-factor/lymphoid-enhancer-factor complex... 
Messenger RNA | Transfection | Tissue samples | RNA | B lymphocytes | Epithelial cells | Genes | Cell lines | Colorectal neoplasms | Genetic mutation | COLON-CANCER | RECEPTOR-BOUND UROKINASE | TRANSCRIPTIONAL ACTIVATION | PROTEIN | PLASMINOGEN-ACTIVATOR | ALPHA-CATENIN | MULTIDISCIPLINARY SCIENCES | XENOPUS EMBRYOS | E-CADHERIN | EXPRESSION | BINDING | Adenocarcinoma | Cell Polarity | Intestinal Mucosa - metabolism | Cytoskeletal Proteins - genetics | Colorectal Neoplasms - genetics | Humans | Gene Expression Regulation, Neoplastic | NF-kappa B - metabolism | Transcription Factor AP-1 - metabolism | Genes, APC | Intestinal Mucosa - cytology | Receptors, Urokinase Plasminogen Activator | Cytoskeletal Proteins - metabolism | Transcription, Genetic | Tumor Cells, Cultured | Colorectal Neoplasms - metabolism | Genes, jun | Recombinant Proteins - metabolism | Cell Line | Promoter Regions, Genetic | Signal Transduction | beta Catenin | Colon - pathology | Membrane Proteins - genetics | Proto-Oncogene Proteins c-jun - genetics | Trans-Activators | Cadherins - physiology | Phosphoproteins - genetics | Zonula Occludens-1 Protein | NF-kappa B - genetics | Urokinase-Type Plasminogen Activator - metabolism | Models, Biological | Proto-Oncogene Proteins c-fos - genetics | Colorectal Neoplasms - pathology | Neoplasm Staging | Colonic Neoplasms | Intestinal Mucosa - pathology | Receptors, Cell Surface - genetics | T cells | Colorectal cancer | Research | Biological Sciences
Journal Article
Journal of cellular and molecular medicine, ISSN 1582-1838, 2013, Volume 17, Issue 1, pp. 30 - 54
Accumulating lines of experimental evidence have revealed that hypoxia‐inducible factors, HIF‐1α and HIF‐2α... 
Hypoxia‐inducible factors | Targeted therapies | Cancer‐initiating cells | Hypoxia | Cancer progression | Metabolic pathways | Cancer stem/progenitor cells | Metastasis‐initiating cells | Metastases | Hypoxia-inducible factors | Cancer-initiating cells | Metastasis-initiating cells | MEDICINE, RESEARCH & EXPERIMENTAL | CLINICALLY RELEVANT SUBTYPES | PANCREATIC-CANCER | Cancer stem | progenitor cells | CHRONIC MYELOID-LEUKEMIA | INHIBITS TUMOR-GROWTH | CELL BIOLOGY | EPITHELIAL-MESENCHYMAL TRANSITION | BREAST-CANCER | PROSTATE-CANCER | GENE-EXPRESSION | FATTY-ACID SYNTHASE | ENDOTHELIAL GROWTH-FACTOR | Neoplasms - metabolism | Hypoxia - drug therapy | Neoplastic Stem Cells - drug effects | Humans | Gene Expression Regulation, Neoplastic | Molecular Targeted Therapy | Hypoxia - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Neoplasm Metastasis | Basic Helix-Loop-Helix Transcription Factors - metabolism | Neoplasms - genetics | Neoplastic Stem Cells - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Neoplastic Stem Cells - pathology | Antineoplastic Agents - pharmacology | Basic Helix-Loop-Helix Transcription Factors - genetics | Signal Transduction | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Intercellular Signaling Peptides and Proteins - genetics | Transcription Factors - genetics | Neoplasms - drug therapy | Transcription Factors - metabolism | Hypoxia - genetics | Hypoxia - pathology | Cell Transformation, Neoplastic - drug effects | Neoplasms - pathology | Apoptosis | Prevention | Glucose metabolism | Physiological aspects | Development and progression | Genetic transcription | Metastasis | Transforming growth factors | Vascular endothelial growth factor | Cancer | Energy metabolism | Transcription factors | Mesenchyme | AKT protein | Insulin-like growth factors | Kinases | Autophagy | Cancer therapies | Neoplasms | Proteins | Homing | Angiogenesis | Signal transduction | Receptors | Epidermal growth factor | Bone marrow | Tumor necrosis factor-TNF | miRNA | Growth factors | Deprivation | Cell survival | Cytokines | Epidermal growth factor receptors | Breast cancer | Rapamycin | Metabolism | Gene expression | Cell differentiation | Insulin | CXCR4 protein | 1-Phosphatidylinositol 3-kinase | Signaling | Pancreatic cancer | Stem cells | Glycolysis | Regulation | Prostate | Cell migration | Tumors | Reviews
Journal Article
The Plant cell, ISSN 1532-298X, 2011, Volume 23, Issue 10, pp. 3824 - 3841
Journal Article