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American Journal of Pathology, The, ISSN 0002-9440, 2012, Volume 180, Issue 4, pp. 1663 - 1674
Melanoma is a malignancy characterized by high invasive/metastatic potential, with no efficient therapy after metastasis. Understanding the molecular... 
Pathology | CANCER-CELLS | ADHESION | IN-VITRO | METASTASIS | GENE-EXPRESSION PROFILES | INDUCIBLE GENE | PATHOLOGY | C-JUN | IDENTIFICATION | PROMOTES | BETA-IG-H3 INTERACTS | Neoplasm Transplantation | Up-Regulation | Fibroblasts - physiology | Skin - metabolism | Humans | Neoplasm Proteins - physiology | Gene Expression Regulation, Neoplastic | Extracellular Matrix - metabolism | Neoplasm Proteins - metabolism | Cell Movement - physiology | Gene Knockdown Techniques | Melanoma - genetics | Thymosin - metabolism | Female | Tumor Cells, Cultured | Neoplasm Proteins - genetics | Extracellular Matrix Proteins - metabolism | Fibroblasts - metabolism | Melanoma - metabolism | Actin Cytoskeleton - metabolism | Extracellular Matrix Proteins - genetics | Transforming Growth Factor beta - physiology | Extracellular Matrix Proteins - pharmacology | Extracellular Matrix Proteins - physiology | Melanoma - pathology | Recombinant Proteins - pharmacology | Cell Adhesion - drug effects | Talin - metabolism | Melanoma - secondary | Integrin beta1 - metabolism | Neoplasm Invasiveness - physiopathology | Transforming Growth Factor beta - pharmacology | Animals | Transforming Growth Factor beta - genetics | Cell Adhesion - physiology | Mice, Nude | Fibroblasts - drug effects | Mice | Mice, Inbred BALB C | Genome-Wide Association Study - methods | Transforming Growth Factor beta - metabolism | Index Medicus | Abridged Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2016, Volume 11, Issue 9, pp. e0162698 - e0162698
Journal Article
Autophagy, ISSN 1554-8627, 12/2012, Volume 8, Issue 12, pp. 1782 - 1797
Granular corneal dystrophy type 2 (GCD2) is an autosomal dominant disease characterized by a progressive age-dependent extracellular accumulation of... 
SQSTM1/p62 | TGFBI | defective autophagy | ubiquitination | LC3/MAP1LC3B | rapamycin | Proteins | Binding | Landes | Calcium | Biology | Bioscience | Cell | Cycle | Organogenesis | Cancer | Ubiquitination | Rapamycin | Defective autophagy | APOPTOSIS | KERATO-EPITHELIN MUTATIONS | AGGREGATE-PRONE PROTEINS | NEURODEGENERATION | ALPHA-SYNUCLEIN | CELL-DEATH | MITOCHONDRIAL FISSION | CELL BIOLOGY | BETA-IG-H3 | DISEASE | DEGRADATION | Humans | Middle Aged | Caspase 3 - metabolism | Male | Fibroblasts - ultrastructure | Autophagy - drug effects | Young Adult | Proteolysis - drug effects | Cornea - drug effects | Lysosomes - metabolism | Corneal Dystrophies, Hereditary - pathology | Adult | Female | Cornea - pathology | Cell Death - drug effects | Child | Extracellular Matrix Proteins - metabolism | Membrane Fusion - drug effects | Fibroblasts - metabolism | Phagosomes - drug effects | Biomarkers - metabolism | Lysosomes - drug effects | Cell Survival - drug effects | Intracellular Space - drug effects | Phagosomes - metabolism | Mutant Proteins - metabolism | Corneal Dystrophies, Hereditary - metabolism | Enzyme Activation - drug effects | Fibroblasts - pathology | Sirolimus - pharmacology | Cornea - metabolism | Intracellular Space - metabolism | Kinetics | Transforming Growth Factor beta - metabolism | Index Medicus | MAP1LC3B | Basic Research Paper | LC3 | SQSTM1 | p62
Journal Article
PROTEOMICS – Clinical Applications, ISSN 1862-8346, 04/2014, Volume 8, Issue 3-4, pp. 168 - 177
Journal Article
Journal Article
International Journal of Oncology, ISSN 1019-6439, 10/2011, Volume 39, Issue 4, pp. 1001 - 1009
As an uncommon cancer, mesothelioma is very hard to treat with a low average survival rate owing to its usual late detection and being highly invasive. The... 
Extracellular matrix | Mouse model | Metastasis | TGFBI | Adhesion-mediated signaling | adhesion-mediated signaling | mouse model | BRONCHIAL EPITHELIAL-CELLS | MALIGNANT PLEURAL MESOTHELIOMA | TUMORIGENIC PHENOTYPE | BETAIG-H3 GENE | MTOR | PATHOGENESIS | LUNG-CANCER | ONCOLOGY | metastasis | BETA-IG-H3 | extracellular matrix | ASBESTOS | Asbestos - toxicity | Cyclin D1 - metabolism | RNA, Small Interfering - genetics | Mesothelioma - pathology | Oncogene Proteins v-mos - metabolism | TOR Serine-Threonine Kinases - metabolism | Humans | Phosphatidylinositol 3-Kinases - metabolism | Gene Knockdown Techniques - methods | DNA-Binding Proteins - metabolism | Transforming Growth Factor beta - antagonists & inhibitors | Neoplasms, Mesothelial - pathology | Proto-Oncogene Proteins c-akt - metabolism | Extracellular Matrix Proteins - metabolism | Neoplasms, Mesothelial - genetics | Cell Line | Cell Growth Processes - drug effects | Extracellular Matrix Proteins - antagonists & inhibitors | Extracellular Matrix Proteins - genetics | Epidermal Growth Factor - metabolism | Mesothelioma - genetics | Disease Progression | Transcription Factors - metabolism | Mesothelioma - metabolism | Transforming Growth Factor beta - genetics | Signal Transduction - drug effects | Cell Line, Tumor | Protein Biosynthesis - drug effects | Neoplasms, Mesothelial - metabolism | Mutation | Cell Cycle - drug effects | RNA, Small Interfering - administration & dosage | Transforming Growth Factor beta - metabolism | Mesothelioma - etiology
Journal Article
Molecular Cancer, ISSN 1476-4598, 05/2012, Volume 11, Issue 1
The extracellular matrix (ECM) has a key role in facilitating the progression of ovarian cancer and we have shown recently that the secreted ECM protein TGFBI... 
Antimitotic agents | Fibronectins | Cell death | Cancer cells | Antineoplastic agents | Transforming growth factors | Integrins | Ovarian cancer | Proteins | Medical research | Apoptosis
Journal Article
Oncology Reports, ISSN 1021-335X, 12/2015, Volume 34, Issue 6, pp. 3187 - 3195
Journal Article
British Journal of Ophthalmology, ISSN 0007-1161, 03/2008, Volume 92, Issue 3, pp. 426 - 427
  The authors have also reported the absence of TGBFI mutations in patients with another form of corneal amyloid deposition, polymorphic amyloid degeneration.... 
OPHTHALMOLOGY | GENE | FORM | BIGH3 | Corneal Dystrophies, Hereditary - genetics | Female | Transforming Growth Factor beta1 - genetics | Mutation | Humans | Middle Aged | Case studies | Corneal diseases | Genetic aspects | Gene mutations | Transforming growth factors | Analysis | Patients | Genes | Index Medicus
Journal Article
대한안과학회지, ISSN 0378-6471, 2012, Volume 53, Issue 10, pp. 1397 - 1402
Purpose: To compare long-term outcomes of penetrating keratoplasty in treating macular corneal dystrophy, transforming growth factor beta induced (TGFBI)... 
Transforming growth factor beta induced (TGFBI) dystrophy | Penetrating keratoplasty | Fuchs` dystrophy | Macular dystrophy
Journal Article
Journal of the Korean Ophthalmological Society, ISSN 0378-6471, 10/2012, Volume 53, Issue 10, pp. 1397 - 1402
To compare long-term outcomes of penetrating keratoplasty in treating macular corneal dystrophy, transforming growth factor beta induced (TGFBI) dystrophy, and... 
Transforming growth factor beta induced (TGFBI) dystrophy | Penetrating keratoplasty | Fuchs\' dystrophy | Macular dystrophy
Journal Article
BMC Cancer, ISSN 1471-2407, 06/2012, Volume 12, Issue 1, pp. 239 - 239
Background: Transforming growth factor beta induced (TGFBI) product, an extracellular matrix (ECM) protein, has been implicated as a putative tumor suppressor... 
Mesothelioma | Proliferation | TGFBI | Tumor suppressor | Breast tumor | BRONCHIAL EPITHELIAL-CELLS