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1. Full Text Transforming growth factor-β1 and diabetic nephropathy
by Chang, Albert S and Hathaway, Catherine K and Smithies, Oliver and Kakoki, Masao
American Journal of Physiology - Renal Physiology, ISSN 0363-6127, 04/2016, Volume 310, Issue 8, pp. F689 - F696
Transforming growth factor-β1 (TGF-β1) is established to be involved in the pathogenesis of diabetic nephropathy. The diabetic milieu enhances oxidative stress... 
Podocyte | Sodium-glucose cotransporter | Proximal tubule | Nephrin | Megalin | Kidney - metabolism | Animals | Sodium-Glucose Transporter 2 - metabolism | Diabetic Nephropathies - metabolism | Humans | Oxidative Stress - physiology | Transforming Growth Factor beta1 - metabolism | Sodium-Glucose Transporter 1 - metabolism | sodium-glucose cotransporter | nephrin | Reviews | proximal tubule | megalin | podocyte
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2. Full Text The extracellular matrix and transforming growth factor-β1: Tale of a strained relationship
by Hinz, Boris
Matrix Biology, ISSN 0945-053X, 09/2015, Volume 47, pp. 54 - 65
Physiological tissue repair aims at restoring the mechano-protective properties of the extracellular matrix. Consequently, redundant regulatory mechanisms are... 
Latent TGF-β1 binding protein (LTBP-1) | Latency associated peptide (LAP) | Fibronectin | Fibrosis | Strain | Integrins - physiology | Mechanotransduction, Cellular | Animals | Extracellular Matrix - physiology | Humans | Cell Adhesion | Transforming Growth Factor beta1 - physiology
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3. Full Text A microRNA circuit mediates transforming growth factor-β1 autoregulation in renal glomerular mesangial cells
by Kato, Mitsuo and Arce, Laura and Wang, Mei and Putta, Sumanth and Lanting, Linda and Natarajan, Rama
Kidney International, ISSN 0085-2538, 08/2011, Volume 80, Issue 4, pp. 358 - 368
Enhanced transforming growth factor-β1 (TGF-β1) expression in renal cells promotes fibrosis and hypertrophy during the progression of diabetic nephropathy. The... 
TGF-β | fibrosis | diabetic nephropathy | gene expression | cell signaling | Up-Regulation | Diabetes Mellitus, Type 2 - genetics | Homeodomain Proteins - metabolism | Transforming Growth Factor beta1 - metabolism | Diabetes Mellitus, Experimental - genetics | Diabetes Mellitus, Type 1 - metabolism | Homeostasis | MicroRNAs - metabolism | Diabetes Mellitus, Type 2 - metabolism | Transfection | Time Factors | Kruppel-Like Transcription Factors - metabolism | Upstream Stimulatory Factors - metabolism | Diabetes Mellitus, Type 1 - chemically induced | Diabetes Mellitus, Experimental - chemically induced | 3' Untranslated Regions | Diabetes Mellitus, Experimental - metabolism | Binding Sites | Collagen Type IV - metabolism | Promoter Regions, Genetic | Diabetic Nephropathies - pathology | Collagen Type I - metabolism | Diabetic Nephropathies - metabolism | Cells, Cultured | Diabetes Mellitus, Type 1 - pathology | Diabetes Mellitus, Type 1 - genetics | Diabetic Nephropathies - genetics | Transforming Growth Factor beta1 - genetics | Mesangial Cells - metabolism | Oligonucleotides - metabolism | Animals | Fibrosis | Diabetes Mellitus, Experimental - pathology | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Mice | Diabetes Mellitus, Type 2 - pathology | Mutation | Zinc Finger E-box-Binding Homeobox 1
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4. Full Text Klotho inhibits transforming growth factor-β1 (TGF-β1) signaling and suppresses renal fibrosis and cancer metastasis in mice
by Doi, Shigehiro and Zou, Yonglong and Togao, Osamu and Pastor, Johanne V and John, George B and Wang, Lei and Shiizaki, Kazuhiro and Gotschall, Russell and Schiavi, Susan and Yorioka, Noriaki and Takahashi, Masaya and Boothman, David A and Kuro-o, Makoto
Journal of Biological Chemistry, ISSN 0021-9258, 03/2011, Volume 286, Issue 10, pp. 8655 - 8665
Fibrosis is a pathological process characterized by infiltration and proliferation of mesenchymal cells in interstitial space. A substantial portion of these... 
Molecular Bases of Disease | Cancer Therapy | NMR | Mouse | Fibrosis | Epithelial-to-Mesenchymal Transition | Metastasis | Transforming Growth Factor β (TGFβ) | Klotho | Kidney
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5. Full Text Regulation of transforming growth factor-β1-driven lung fibrosis by galectin-3
by MacKinnon, Alison C and Gibbons, Michael A and Farnworth, Sarah L and Leffler, Hakon and Nilsson, Ulf J and Delaine, Tamara and Simpson, A. John and Forbes, Stuart J and Hirani, Nik and Gauldie, Jack and Sethi, Tariq
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 03/2012, Volume 185, Issue 5, pp. 537 - 546
Idiopathic pulmonary fibrosis (IPF) is a chronic dysregulated response to alveolar epithelial injury with differentiation of epithelial cells and fibroblasts... 
Fibroblasts | Epithelial cells | Fibrosis | Lung - pathology | Fibroblasts - physiology | Humans | Mice, Inbred C57BL | Transforming Growth Factor beta1 - metabolism | Pulmonary Fibrosis - pathology | Gene Knockout Techniques | Transforming Growth Factor beta1 - physiology | Idiopathic Pulmonary Fibrosis - metabolism | Animals | Galectin 3 - antagonists & inhibitors | Fluorescent Antibody Technique | Pulmonary Fibrosis - physiopathology | Pulmonary Fibrosis - chemically induced | Signal Transduction - physiology | Collagen - biosynthesis | Idiopathic Pulmonary Fibrosis - physiopathology | Mice | Pulmonary Fibrosis - metabolism | Galectin 3 - physiology | Bleomycin - toxicity | Disease Models, Animal | Fibroblasts - metabolism | epithelial cells | fibroblasts | fibrosis
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6. Full Text Transforming growth factor-β1 in children with diabetic nephropathy
by Sawires, Happy and Botrous, Osama and Aboulmagd, Abdelmegeed and Madani, Nadia and Abdelhaleem, Osama
Pediatric Nephrology, ISSN 0931-041X, 1/2019, Volume 34, Issue 1, pp. 81 - 85
Transforming growth factor-β1 (TGF-β1) is a multifunctional cytokine that has numerous biological processes. Earlier research indicates that protein and mRNA... 
Pediatrics | Nephrology | Medicine & Public Health | Type 1 diabetes | Transforming growth factor | Diabetic nephropathy | Children | Urology | Microalbuminuria | Nephropathy | Diabetes mellitus | Transforming growth factor-a | mRNA | Diabetes mellitus (insulin dependent) | Diabetes | Growth factors | Enzyme-linked immunosorbent assay
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7. Full Text Interleukin-10 and transforming growth factor-β1 facilitate long-term potentiation in CA1 region of hippocampus
by Nenov, Miroslav N and Malkov, Anton E and Konakov, Maxim V and Levin, Sergey G
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 10/2019, Volume 518, Issue 3, pp. 486 - 491
It has been shown that pro-inflammatory cytokines preferentially attenuate long-term potentiation (LTP), at the same time the effect of anti-inflammatory... 
Anti-inflammatory cytokines | Interleukine-10 | Transforming growth factor-β1 | Long-term potentiation | Synaptic plasticity
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8. Full Text High serum transforming growth factor-β1 levels predict outcome in hepatocellular carcinoma patients treated with sorafenib
by Lin, Tzu-Hsuan and Shao, Yu-Yun and Chan, Soa-Yu and Huang, Chung-Yi and Hsu, Chih-Hung and Cheng, Ann-Lii
Clinical Cancer Research, ISSN 1078-0432, 08/2015, Volume 21, Issue 16, pp. 3678 - 3684
The TGF-β signaling pathway is crucial in the progression and metastasis of malignancies. We investigated whether the serum TGF-β1 level was related to the... 
Niacinamide - analogs & derivatives | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Liver Neoplasms - drug therapy | Male | Treatment Outcome | Hep G2 Cells | Niacinamide - administration & dosage | Biomarkers, Tumor - blood | Liver Neoplasms - blood | Disease-Free Survival | RNA, Messenger - biosynthesis | Carcinoma, Hepatocellular - drug therapy | Phenylurea Compounds - administration & dosage | Carcinoma, Hepatocellular - pathology | Aged, 80 and over | Adult | Female | Liver Neoplasms - pathology | Aged | Carcinoma, Hepatocellular - blood | Transforming Growth Factor beta1 - blood
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9. Full Text MicroRNA-101 inhibited postinfarct cardiac fibrosis and improved left ventricular compliance via the FBJ osteosarcoma oncogene/transforming growth factor-β1 pathway
by Pan, Zhenwei and Sun, Xuelin and Shan, Hongli and Wang, Ning and Wang, Jinghao and Ren, Jinshuai and Feng, Shuya and Xie, Liangjun and Lu, Chunying and Yuan, Ye and Zhang, Yang and Wang, Ying and Lu, Yanjie and Yang, Baofeng
Circulation, ISSN 0009-7322, 08/2012, Volume 126, Issue 7, pp. 840 - 850
Cardiac interstitial fibrosis is a major cause of the deteriorated performance of the heart in patients with chronic myocardial infarction. MicroRNAs (miRs)... 
microRNAs | heart failure | fibrosis | MicroRNAs - therapeutic use | Proto-Oncogene Proteins c-fos - biosynthesis | Male | Compliance - drug effects | Collagen - biosynthesis | Fibroblasts - metabolism | Animals, Newborn | Vasoconstrictor Agents - pharmacology | Angiotensin II - pharmacology | Echocardiography | Mice, Inbred C57BL | Cells, Cultured | Gene Silencing | Rats | Myocardium - pathology | Treatment Outcome | Rats, Sprague-Dawley | Transforming Growth Factor beta1 - physiology | Compliance - physiology | Animals | Myocardial Infarction - drug therapy | Ventricular Dysfunction, Left - drug therapy | Fibrosis | Cell Proliferation - drug effects | Mice | MicroRNAs - genetics | Hemodynamics | Chronic Disease | Proto-Oncogene Proteins c-fos - physiology
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10. Full Text Transforming growth factor β1 (TGFβ1) in physiology and pathology
by Kajdaniuk, Dariusz and Marek, Bogdan and Borgiel-Marek, Halina and Kos-Kudła, Beata
Endokrynologia Polska, ISSN 0423-104X, 2013, Volume 64, Issue 5, pp. 384 - 396
Neoplasm | Angiogenesis | Pathology | Endocrine gland | TGF beta 1 | TGFβ1 | Pathophysiology | Transforming growth factor beta 1 | Liver | Fibrosis | Physiology | Cancer
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11. Full Text Molecular basis of selective atrial fibrosis due to overexpression of transforming growth factor-β1
by Rahmutula, Dolkun and Marcus, Gregory M and Wilson, Emily E and Ding, Chun-Hua and Xiao, Yuanyuan and Paquet, Agnes C and Barbeau, Rebecca and Barczak, Andrea J and Erle, David J and Olgin, Jeffrey E
Cardiovascular Research, ISSN 0008-6363, 09/2013, Volume 99, Issue 4, pp. 769 - 779
Animal studies show that transforming growth factor-β1 (TGF-β1) is an important mediator of atrial fibrosis and atrial fibrillation (AF). This study... 
Heart failure | Cardiac fibrosis | Gene expression | Atrial fibrillation | Transforming growth factor-beta1 | Heart Atria - pathology | Animals | Receptors, Transforming Growth Factor beta - analysis | Humans | Fibrosis | Atrial Fibrillation - etiology | Mice, Transgenic | Mice | Transforming Growth Factor beta1 - analysis | Mink | Transforming Growth Factor beta1 - physiology | Original
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12. Full Text Sulfated hyaluronan and chondroitin sulfate derivatives interact differently with human transforming growth factor-β1 (TGF-β1)
by Hintze, V and Miron, A and Moeller, S and Schnabelrauch, M and Wiesmann, H.-P and Worch, H and Scharnweber, D
Acta Biomaterialia, ISSN 1742-7061, 07/2012, Volume 8, Issue 6, pp. 2144 - 2152
This study demonstrates that the modification of hyaluronan (hyaluronic acid; Hya) and chondroitin sulfate (CS) with sulfate groups leads to different binding... 
Surface plasmon resonance | Transforming growth factor-β1 (TGF-β1) | Chondroitin sulfate derivatives | Hyaluronic acid/hyaluronan derivatives | ELISA | Surface Plasmon Resonance | Transforming Growth Factor beta1 - chemistry | Enzyme-Linked Immunosorbent Assay | Magnetic Resonance Spectroscopy | Hyaluronic Acid - chemistry | Sulfates - chemistry | Chondroitin Sulfates - chemistry | Spectroscopy, Fourier Transform Infrared
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13. Full Text B cell-derived transforming growth factor-β1 expression limits the induction phase of autoimmune neuroinflammation
by Bjarnadóttir, Kristbjörg and Benkhoucha, Mahdia and Merkler, Doron and Weber, Martin S and Payne, Natalie L and Bernard, Claude C. A and Molnarfi, Nicolas and Lalive, Patrice H
Scientific Reports, ISSN 2045-2322, 10/2016, Volume 6, Issue 1, p. 34594
Studies in experimental autoimmune encephalomyelitis (EAE), a murine model of multiple sclerosis (MS), have shown that regulatory B cells modulate the course... 
Th1 Cells - pathology | Cell Communication - immunology | Dendritic Cells - immunology | Interleukin-17 - immunology | Dendritic Cells - pathology | Encephalomyelitis, Autoimmune, Experimental - immunology | Myelin-Oligodendrocyte Glycoprotein - administration & dosage | Th1 Cells - immunology | B-Lymphocytes, Regulatory - pathology | Transforming Growth Factor beta1 - immunology | Encephalomyelitis, Autoimmune, Experimental - chemically induced | Gene Deletion | Encephalomyelitis, Autoimmune, Experimental - genetics | Interferon-gamma - genetics | Th17 Cells - pathology | Gene Expression | Encephalomyelitis, Autoimmune, Experimental - pathology | Mice, Inbred C57BL | Interleukin-17 - genetics | Transforming Growth Factor beta1 - genetics | B-Lymphocytes, Regulatory - immunology | Mice, Knockout | Animals | Interferon-gamma - immunology | Th17 Cells - immunology | Mice
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14. Full Text Transforming growth factor-β1-mediated renal fibrosis is dependent on the regulation of transforming growth factor receptor 1 expression by let-7b
by Wang, Bo and Jha, Jay C and Hagiwara, Shinji and McClelland, Aaron D and Jandeleit-Dahm, Karin and Thomas, Merlin C and Cooper, Mark E and Kantharidis, Phillip
Kidney International, ISSN 0085-2538, 2014, Volume 85, Issue 2, pp. 352 - 361
Renal fibrosis results from excessive accumulation of extracellular matrix mainly driven by transforming growth factor-β1 (TGF-β1). Certain microRNAs have been... 
microRNAs | chronic kidney disease | TGF-beta | fibrosis | diabetic nephropathy | micro-RNAs | Receptors, Transforming Growth Factor beta - genetics | Apolipoproteins E - deficiency | Kidney - pathology | Humans | Transforming Growth Factor beta1 - metabolism | MicroRNAs - metabolism | Smad3 Protein - metabolism | Renal Insufficiency, Chronic - metabolism | Kidney - metabolism | Transfection | RNA Interference | Time Factors | Renal Insufficiency, Chronic - genetics | Kidney Tubules - pathology | 3' Untranslated Regions | Kidney Tubules - metabolism | Binding Sites | Extracellular Matrix Proteins - metabolism | Protein-Serine-Threonine Kinases - metabolism | Disease Models, Animal | Recombinant Proteins - metabolism | Cell Line | Diabetic Nephropathies - pathology | Signal Transduction | Diabetic Nephropathies - metabolism | Extracellular Matrix Proteins - genetics | Mice, Inbred C57BL | Gene Expression Regulation | Protein-Serine-Threonine Kinases - genetics | Adenine | Rats | Diabetic Nephropathies - genetics | Transforming Growth Factor beta1 - genetics | Mesangial Cells - metabolism | Mice, Knockout | Renal Insufficiency, Chronic - pathology | Animals | Receptors, Transforming Growth Factor beta - metabolism | Apolipoproteins E - genetics | Fibrosis | Mesangial Cells - pathology | Mice | MicroRNAs - genetics | Renal Insufficiency, Chronic - chemically induced
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15. Full Text Histidine and arginine modulate intestinal cell restitution via transforming growth factor-β1
by Matsui, Tatsunobu and Ichikawa, Hiroshi and Fujita, Tomoka and Takemura, Shigekazu and Takagi, Tomohisa and Osada-Oka, Mayuko and Minamiyama, Yukiko
European Journal of Pharmacology, ISSN 0014-2999, 05/2019, Volume 850, pp. 35 - 42
Intestinal wound healing depends on the precise balance of restitution, proliferation, and differentiation of intestinal epithelial cells (IECs). In a previous... 
Histidine | Arginine | Transforming growth factor-β1 | Cell restitution | Intestinal epithelial cell
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16. Full Text Ethanol-induced DNA repair in neural stem cells is transforming growth factor β1-dependent
by Hicks, Steven D and Miller, Michael W
Experimental Neurology, ISSN 0014-4886, 07/2019, Volume 317, pp. 214 - 225
Following neurotoxic damage, cells repair their DNA, and survive or undergo apoptosis. This study tests the hypothesis that ethanol induces a DNA damage... 
DNA damage recognition | Fibroblast growth factor | Reactive oxygen species | Transforming growth factor | Stem cells | Nucleotide excision repair | 9-1-1 complex | Base excision repair | Fetal alcohol spectrum disorder | p53
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