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PLoS ONE, ISSN 1932-6203, 01/2014, Volume 9, Issue 1, pp. e86273 - e86273
Journal Article
JOURNAL OF SURGICAL ONCOLOGY, ISSN 0022-4790, 11/2018, Volume 118, Issue 6, pp. 966 - 974
Background and ObjectivesMethodsTransforming growth factor -induced(TGFBI) protein is a secreted extracellular matrix protein with conflicting roles in cancer,... 
cancer-associated fibroblast | SURGERY | FACTOR-BETA | METASTASIS | gastric cancer | transforming factor -induced | stroma | MATRIX PROTEIN | TGFBI BETA-IG-H3 | OVARIAN-CANCER | ONCOLOGY | SQUAMOUS-CELL CARCINOMA | GENE-EXPRESSION | MYOFIBROBLASTS | STROMAL CELLS | UP-REGULATION
Journal Article
CANCER SCIENCE, ISSN 1349-7006, 04/2018, Volume 109, Issue 4, pp. 980 - 987
Transforming growth factor--induced (TGFBI) is considered to be a vital gene in several carcinomas. In this study we determined the effect of TGFBI on the... 
PROTEIN | proliferation | INSTILLATION | transforming growth factor--induced | IDENTIFICATION | CELL CARCINOMA | BETA | MATRIX METALLOPROTEINASES | urothelial carcinoma | ONCOLOGY | metastasis | BETA-IG-H3 | RECURRENCE | Epirubicin | PROGRESSION | INVASIVE BLADDER-CANCER | Anthracyclines | Carcinoma | RNA | Analysis | Genetic research | Calpain | Bone morphogenetic proteins | Metastasis | Transforming growth factors | Cancer
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2012, Volume 7, Issue 3, pp. e33414 - e33414
The proteolytic activity of Furin responsible for processing full length Notch-1 (p300) plays a critical role in Notch signaling. The amplitude and duration of... 
APOPTOSIS | PATHWAY | PDGF | BIOLOGY | DOWN-REGULATION | RECEPTOR | GOLGI | PROTEINS | CLEAVAGE | Pancreatic Neoplasms - metabolism | Humans | Transplantation, Heterologous | Neoplasms, Experimental - pathology | Furin - genetics | Protein Binding - drug effects | Neoplasms, Experimental - genetics | src-Family Kinases - metabolism | Female | Proto-Oncogene Proteins c-sis - pharmacology | Furin - metabolism | Transforming Growth Factor alpha - pharmacology | Pancreatic Neoplasms - pathology | src-Family Kinases - antagonists & inhibitors | Pancreatic Neoplasms - genetics | Pyrimidines - pharmacology | Receptor, Notch1 - metabolism | Blotting, Western | Microscopy, Confocal | Animals | Tumor Burden - drug effects | Intercellular Signaling Peptides and Proteins - pharmacology | Mice, Nude | Cell Line, Tumor | Mice | Mice, Inbred BALB C | HeLa Cells | Mutation | Neoplasms, Experimental - metabolism | Receptor, Notch1 - genetics | src-Family Kinases - genetics | Tyrosine | Phenols | Platelet-derived growth factor | Transforming growth factors | Pancreatic cancer | Event-related potentials | Activation | Biochemistry | Metastasis | Kinases | Tissues | Carcinogenesis | Proteins | Signal transduction | Carcinogens | Cell growth | Epidermal growth factor | Pathways | Proteolysis | Rodents | Xenografts | Furin | Protein-tyrosine kinase receptors | Localization | Vascular endothelial growth factor | Drug dosages | Protein-tyrosine kinase | Epidermal growth factor receptors | Src protein | Platelet-derived growth factor BB | Signaling | Ligands | Notch protein | Molecular biology | Cancer | Apoptosis | Tumors | Index Medicus
Journal Article
Archives of Dermatological Research, ISSN 0340-3696, 7/2013, Volume 305, Issue 5, pp. 433 - 445
CCN2 plays an important role in the pathogenesis of hypertrophic scars (HTSs). Although CCN2 is involved in many fibroproliferative events, the CCN2 induction... 
c-Jun N-terminal kinase | Scar formation | Medicine & Public Health | Dermatology | Extracellular signal-regulated kinase | Connective tissue growth factor | Hypertrophic scar | FIBROSIS | FACTOR-BETA | COLLAGEN EXPRESSION | KELOID FIBROBLASTS | MAPK | DERMATOLOGY | HYPERTROPHY | PATHWAY | CCN2 | DIFFERENTIATION | SKELETAL DEVELOPMENT | Fibroblasts - enzymology | Phosphorylation | Humans | Transforming Growth Factor beta1 - metabolism | Actins - metabolism | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | JNK Mitogen-Activated Protein Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Wound Healing | Cicatrix, Hypertrophic - pathology | Cicatrix, Hypertrophic - drug therapy | Cicatrix, Hypertrophic - enzymology | Time Factors | Cicatrix, Hypertrophic - genetics | Extracellular Matrix Proteins - metabolism | Skin - pathology | Disease Models, Animal | Recombinant Proteins - metabolism | Rabbits | Collagen Type I - metabolism | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Extracellular Matrix Proteins - genetics | Cells, Cultured | Gene Expression Regulation | Fibroblasts - pathology | Skin - enzymology | Animals | MAP Kinase Signaling System - drug effects | Fibroblasts - drug effects | Protein Kinase Inhibitors - pharmacology | Enzyme Activation | Connective Tissue Growth Factor - metabolism | Skin - drug effects | Architecture | Muscle proteins | Mitogens | Phosphotransferases | Analysis | Collagen | Index Medicus
Journal Article
JOURNAL OF CELLULAR BIOCHEMISTRY, ISSN 0730-2312, 02/2019, Volume 120, Issue 2, pp. 2259 - 2270
Transforming growth factor- (TGF-) induces apoptosis of normal epithelial cells, such as mammary epithelium. Although breast cancer progression associates with... 
FOXA1 | SIGNAL | TGF-BETA | PHOSPHORYLATION | TAMOXIFEN | BIOCHEMISTRY & MOLECULAR BIOLOGY | apoptosis | breast cancer | INDUCTION | CELL BIOLOGY | TGF | TUMOR-SUPPRESSOR | SMAD | EXPRESSION | BINDING | importin7 | REVEALS | RNA sequencing | Estrogen | Development and progression | Breast cancer | Transforming growth factors | Gene expression | Prevention | Analysis | Cancer cells | Bone morphogenetic proteins | Cellular signal transduction | Apoptosis | Cancer
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 0363-6127, 07/2012, Volume 303, Issue 2, pp. F293 - F303
Blaes N, Pecher C, Mehrenberger M, Cellier E, Praddaude F, Chevalier J, Tack I, Couture R, Girolami JP. Bradykinin inhibits high glucose-and growth... 
B2-kinin receptor knockout mice | Transforming growth factor | Fibrosis | Intercellular adhesion molecule-1 | Akt | Renin-angiotensin system | Epithelial growth factor | HUMAN GLOMERULOSCLEROSIS | OXIDATIVE STRESS | PHYSIOLOGY | ACTIVATED PROTEIN-KINASE | ANGIOTENSIN-II | epithelial growth factor | B-2 RECEPTOR | DIABETIC RENAL-DISEASE | TRANSFORMING GROWTH-FACTOR-BETA-1 | UROLOGY & NEPHROLOGY | fibrosis | transforming growth factor | NF-KAPPA-B | UP-REGULATION | renin-angiotensin system | EGF RECEPTOR | intercellular adhesion molecule-1 | Receptor, Bradykinin B2 - genetics | MAP Kinase Signaling System - physiology | Male | Mesangial Cells - drug effects | Dose-Response Relationship, Drug | Diabetes Mellitus, Experimental - chemically induced | Diabetes Mellitus, Experimental - metabolism | Streptozocin - adverse effects | Bradykinin - pharmacology | Disease Models, Animal | Collagen Type IV - metabolism | Collagen Type I - metabolism | Collagen Type I - antagonists & inhibitors | Collagen Type IV - antagonists & inhibitors | Mice, Inbred C57BL | Cells, Cultured | Rats | Glucose - pharmacology | Proto-Oncogene Proteins c-akt - physiology | Mesangial Cells - metabolism | Rats, Sprague-Dawley | Mice, Knockout | Transforming Growth Factor beta - pharmacology | Intercellular Adhesion Molecule-1 - metabolism | Animals | Receptor, Bradykinin B2 - deficiency | Receptor, Bradykinin B2 - metabolism | Signal Transduction - physiology | Mice | Epidermal Growth Factor - pharmacology | Care and treatment | Epidermal growth factor | Bradykinin | Physiological aspects | Development and progression | Genetic aspects | Kidney diseases | Transforming growth factors | Peptides | Collagen | Biosynthesis | Physiology | Glucose | ACE inhibitors | Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology: Journal of the American Heart Association, ISSN 1079-5642, 05/2003, Volume 23, Issue 5, pp. 795 - 801
OBJECTIVE—We investigated the comparative roles of mitogen-activated protein (MAP) kinases, including c-Jun NH2-terminal kinase (JNK), extracellular... 
Vascular smooth muscle cell | Platelet-derived growth factor | Proliferation | Gene transfer | Gene expression | PATHWAYS | FACTOR-BETA | gene transfer | proliferation | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | platelet-derived growth factor | RAT MODEL | PDGF | DISEASE | PERIPHERAL VASCULAR DISEASE | vascular smooth muscle cell | ACTIVATED PROTEIN-KINASES | C-JUN | HEMATOLOGY | SIGNAL-REGULATED KINASE | gene expression | SMOOTH-MUSCLE CELLS | Mitogen-Activated Protein Kinase Kinases - genetics | Hyperplasia | Cyclins - genetics | Mitogen-Activated Protein Kinase Kinases - physiology | Recombinant Fusion Proteins - physiology | Mitogen-Activated Protein Kinase 1 - deficiency | Male | Transforming Growth Factor beta - biosynthesis | Cyclins - biosynthesis | MAP Kinase Kinase 4 | Mitogen-Activated Protein Kinase 1 - genetics | Tumor Suppressor Proteins - genetics | Cell Cycle Proteins - genetics | JNK Mitogen-Activated Protein Kinases | Plasminogen Activator Inhibitor 1 - genetics | p38 Mitogen-Activated Protein Kinases | Carotid Artery Injuries - pathology | Muscle, Smooth, Vascular - drug effects | Transforming Growth Factor beta1 | Transduction, Genetic | Mitogen-Activated Protein Kinase 1 - physiology | Cyclin-Dependent Kinase Inhibitor p21 | Proto-Oncogene Proteins c-sis | Rats | Chemokine CCL2 - genetics | Cell Cycle Proteins - biosynthesis | Mitogen-Activated Protein Kinase Kinases - deficiency | Platelet-Derived Growth Factor - pharmacology | Muscle, Smooth, Vascular - cytology | Rats, Sprague-Dawley | Cell Division - drug effects | Cyclin-Dependent Kinase Inhibitor p27 | Gene Expression Regulation - drug effects | Mitogen-Activated Protein Kinases - deficiency | Cell Movement - drug effects | Plasminogen Activator Inhibitor 1 - biosynthesis | Animals | Transforming Growth Factor beta - genetics | Tunica Intima - pathology | Mitogen-Activated Protein Kinases - physiology | Chemokine CCL2 - biosynthesis | Mitogen-Activated Protein Kinases - genetics | Tumor Suppressor Proteins - biosynthesis | Muscle, Smooth, Vascular - enzymology | Mitogen-Activated Protein Kinase 3 | Index Medicus
Journal Article
Journal of Gastroenterology and Hepatology, ISSN 0815-9319, 03/2012, Volume 27, Issue 3, pp. 566 - 578
Background and Aim:  Cancer invasion and metastasis are characterized by epithelial‐mesenchymal transition (EMT). Hepatocellular carcinoma (HCC) causes... 
chemoprevention | growth factors | epithelial‐mesenchymal transition | cyclooxygenase‐2 | hepatocellular carcinoma | Epithelial-mesenchymal transition | Hepatocellular carcinoma | Cyclooxygenase-2 | Growth factors | Chemoprevention | ACTIVATION | METASTASIS | cyclooxygenase-2 | CELL LINES | CANCER | BETA | INHIBITS APOPTOSIS | INVASION | epithelial-mesenchymal transition | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | STIMULATES PROLIFERATION | Cadherins - metabolism | Vimentin - metabolism | Humans | Fibroblast Growth Factor 2 - pharmacology | Epithelial-Mesenchymal Transition - drug effects | Hepatocyte Growth Factor - pharmacology | Cyclooxygenase 2 - genetics | Oncogene Protein v-akt - genetics | Tumor Cells, Cultured | Cadherins - genetics | Oncogene Protein v-akt - metabolism | Transforming Growth Factor beta1 - pharmacology | Gene Expression | Collagen Type I - metabolism | alpha 1-Antitrypsin - genetics | Albumins - metabolism | Cell Transplantation | Fibronectins - metabolism | Dinoprostone - metabolism | Cell Movement - drug effects | Animals | Signal Transduction - drug effects | Carcinoma, Hepatocellular - pathology | Cyclooxygenase 2 - metabolism | Mice | RNA, Small Interfering | alpha 1-Antitrypsin - metabolism | Epidermal Growth Factor - pharmacology | Albumins - genetics | Carcinoma, Hepatocellular - metabolism | Fibronectins | Carcinoma | Hepatoma | Metastasis | Prevention | Hepatitis | Liver cancer | Prostaglandins | Epidermal growth factor | Analysis | Prostaglandins E | Collagen | Stem cells | Fibroblast growth factors | Intermediate filament proteins | Cancer | Index Medicus
Journal Article
Oral Diseases, ISSN 1354-523X, 09/2013, Volume 19, Issue 6, pp. 604 - 610
Journal Article