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Publication DateProceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2011, Volume 108, Issue 32, pp. 13218 - 13223
Transplantation of human hematopoietic stem cells into severely immunocompromised newborn mice allows the development of a human hematopoietic and immune...
Spleen | Phagocytes | Transgenic animals | Stem cells | Cell lines | Erythrocytes | Mice | Natural killer cells | Hematopoietic stem cells | Blood | cell | CD34 | Xenorejection | signal regulatory proteins | Neonates | Animal models | Translation | Immune response | Cytokines | Severe combined immunodeficiency | Cross-reactivity | Hemopoiesis | Life span | Xenografts | Genetic engineering | Transgenes | Phagocytosis | Biological Sciences | CD34+ cell | xenorejection
Spleen | Phagocytes | Transgenic animals | Stem cells | Cell lines | Erythrocytes | Mice | Natural killer cells | Hematopoietic stem cells | Blood | cell | CD34 | Xenorejection | signal regulatory proteins | Neonates | Animal models | Translation | Immune response | Cytokines | Severe combined immunodeficiency | Cross-reactivity | Hemopoiesis | Life span | Xenografts | Genetic engineering | Transgenes | Phagocytosis | Biological Sciences | CD34+ cell | xenorejection
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Loading Rights2007, 2nd ed., ISBN 9780471471929, xvi, 523
Jacqueline N. Crawley, PhD, is Chief of the Laboratory of Behavioral Neuroscience at the National Institute of Mental Health -- Adjunct Professor of Psychiatry...
Behavior, Animal | Phenotype | Animal models | Neurogenetics | methods | Transgenic mice | Mice, Transgenic | Genetics, Behavioral | Behavior | Disease Models, Animal | Mice, Knockout | Animal models in research
Behavior, Animal | Phenotype | Animal models | Neurogenetics | methods | Transgenic mice | Mice, Transgenic | Genetics, Behavioral | Behavior | Disease Models, Animal | Mice, Knockout | Animal models in research
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2008, 1. Aufl., Current topics in microbiology and immunology, ISBN 3540756477, Volume 324., xiii, 199
Currently not available.
Mice as laboratory animals | Transgenic mice | Human Genetics | Biomedicine | Immunology | Human Physiology
Mice as laboratory animals | Transgenic mice | Human Genetics | Biomedicine | Immunology | Human Physiology
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2003, 3rd ed., ISBN 9780879695743, x, 764
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2004, ISBN 9780691016368, xviii, 299
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1994, 2nd ed., ISBN 9780879693848, xvii, 497
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2000, ISBN 9780471316398, xiii, 329
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Nature, ISSN 0028-0836, 02/2004, Volume 427, Issue 6974, pp. 504 - 504
Mammals cannot naturally produce omega-3 (n-3) fatty acids - beneficial nutrients found mainly in fish oil - from the more abundant omega-6 (n-6) fatty acids...
CELLS | DESATURASE | EXPRESSION | MULTIDISCIPLINARY SCIENCES | Body Composition | Fatty Acid Desaturases - genetics | Disease Susceptibility | Fatty Acids, Omega-3 - metabolism | Caenorhabditis elegans - genetics | Fatty Acid Desaturases - metabolism | Caenorhabditis elegans Proteins - metabolism | Mice, Transgenic | Food Industry - trends | Fatty Acids, Omega-6 - metabolism | Fatty Acids, Omega-6 - administration & dosage | Fatty Acids, Omega-6 - pharmacology | Animals | Dietary Fats - pharmacology | Fatty Acids, Omega-3 - pharmacology | Dietary Fats - administration & dosage | Mice | Caenorhabditis elegans Proteins - genetics | Fatty Acids, Omega-3 - administration & dosage | Food | fat-1 gene | Index Medicus
CELLS | DESATURASE | EXPRESSION | MULTIDISCIPLINARY SCIENCES | Body Composition | Fatty Acid Desaturases - genetics | Disease Susceptibility | Fatty Acids, Omega-3 - metabolism | Caenorhabditis elegans - genetics | Fatty Acid Desaturases - metabolism | Caenorhabditis elegans Proteins - metabolism | Mice, Transgenic | Food Industry - trends | Fatty Acids, Omega-6 - metabolism | Fatty Acids, Omega-6 - administration & dosage | Fatty Acids, Omega-6 - pharmacology | Animals | Dietary Fats - pharmacology | Fatty Acids, Omega-3 - pharmacology | Dietary Fats - administration & dosage | Mice | Caenorhabditis elegans Proteins - genetics | Fatty Acids, Omega-3 - administration & dosage | Food | fat-1 gene | Index Medicus
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Loading Rights2004, ISBN 9780471444602, xi, 474 p., [8] p. of plates
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PLoS Biology, ISSN 1544-9173, 08/2004, Volume 2, Issue 8, pp. 1081 - 1090
Systemic lupus erythematosus (SLE) is a multisystem autoimmune disorder in which complex genetic factors play an important role. Several strains of...
BXSB MICE | SYSTEMIC-LUPUS-ERYTHEMATOSUS | C1Q DEFICIENCY | CHROMATIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | DISEASE | BIOLOGY | NEPHRITIS | SERUM | SUSCEPTIBILITY LOCUS | ZEALAND BLACK MICE | AMYLOID-P-COMPONENT | Gene Targeting | Genetic Predisposition to Disease | Species Specificity | Mice, Inbred C57BL | Autoimmunity - genetics | Genotype | Mice, Transgenic | Chromosome Mapping | Animals | Cell Nucleus - metabolism | Autoimmunity - immunology | Epistasis, Genetic | Lupus Erythematosus, Systemic - genetics | Female | Mice | Models, Genetic | Genome | Quantitative Trait Loci | Microsatellite Repeats | Cohort Studies | Disease Models, Animal | Genetic Linkage | Index Medicus | Mus (Mouse) | Genetics | Immunology | Gene Therapy | Genomics | Lupus | Mutation | Autoimmune diseases | Genes
BXSB MICE | SYSTEMIC-LUPUS-ERYTHEMATOSUS | C1Q DEFICIENCY | CHROMATIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | DISEASE | BIOLOGY | NEPHRITIS | SERUM | SUSCEPTIBILITY LOCUS | ZEALAND BLACK MICE | AMYLOID-P-COMPONENT | Gene Targeting | Genetic Predisposition to Disease | Species Specificity | Mice, Inbred C57BL | Autoimmunity - genetics | Genotype | Mice, Transgenic | Chromosome Mapping | Animals | Cell Nucleus - metabolism | Autoimmunity - immunology | Epistasis, Genetic | Lupus Erythematosus, Systemic - genetics | Female | Mice | Models, Genetic | Genome | Quantitative Trait Loci | Microsatellite Repeats | Cohort Studies | Disease Models, Animal | Genetic Linkage | Index Medicus | Mus (Mouse) | Genetics | Immunology | Gene Therapy | Genomics | Lupus | Mutation | Autoimmune diseases | Genes
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Loading RightsPhotochemistry and Photobiology, ISSN 0031-8655, 07/2017, Volume 93, Issue 4, pp. 1016 - 1024
Naproxen possesses anti‐proliferative and pro‐apoptotic effects besides its known anti‐inflammatory functions. Here, we demonstrate the anticancer effects of...
APOPTOSIS | BIOPHYSICS | NONSTEROIDAL ANTIINFLAMMATORY DRUGS | INDUCED SKIN CARCINOGENESIS | SIGNALING PATHWAY | BIOCHEMISTRY & MOLECULAR BIOLOGY | MUTANT P53 | ENDOPLASMIC-RETICULUM STRESS | NF-KAPPA-B | CANCER | PHOTOCARCINOGENESIS | PROSTAGLANDIN E-2 | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Humans | Neoplasms, Radiation-Induced - etiology | Naproxen - pharmacology | Patched-1 Receptor - genetics | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Skin Neoplasms - prevention & control | Ultraviolet Rays | Carcinoma, Basal Cell - etiology | Epithelial-Mesenchymal Transition | Biomarkers, Tumor - metabolism | Carcinoma, Basal Cell - prevention & control | Female | Neoplasms, Radiation-Induced - prevention & control | Antineoplastic Agents - pharmacology | Neoplasms, Radiation-Induced - pathology | Skin Neoplasms - pathology | Mice, Hairless | Mice, Transgenic | Carcinoma, Basal Cell - pathology | Carcinoma, Squamous Cell - prevention & control | Unfolded Protein Response | Neoplasms, Radiation-Induced - metabolism | Carcinoma, Basal Cell - metabolism | Skin Neoplasms - metabolism | Carcinoma, Squamous Cell - etiology | Animals | Cell Line, Tumor | Skin Neoplasms - etiology | Squamous cell carcinoma | Naproxen | Analysis | Skin cancer | Vimentin | Animal models | Mesenchyme | Cyclin D1 | Carcinogenesis | E-cadherin | Anticancer properties | Carcinogens | N-Cadherin | Protein folding | Proliferating cell nuclear antigen | Inhibition | Invasiveness | Inflammation | Hairless | Ablation | Nitric-oxide synthase | Ultraviolet radiation | Skin | Cyclooxygenase-2 | Tumors | Cancer | Apoptosis | Index Medicus | UVB | skin | photocarcinogenesis | inflammation | UPR signaling
APOPTOSIS | BIOPHYSICS | NONSTEROIDAL ANTIINFLAMMATORY DRUGS | INDUCED SKIN CARCINOGENESIS | SIGNALING PATHWAY | BIOCHEMISTRY & MOLECULAR BIOLOGY | MUTANT P53 | ENDOPLASMIC-RETICULUM STRESS | NF-KAPPA-B | CANCER | PHOTOCARCINOGENESIS | PROSTAGLANDIN E-2 | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Humans | Neoplasms, Radiation-Induced - etiology | Naproxen - pharmacology | Patched-1 Receptor - genetics | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Skin Neoplasms - prevention & control | Ultraviolet Rays | Carcinoma, Basal Cell - etiology | Epithelial-Mesenchymal Transition | Biomarkers, Tumor - metabolism | Carcinoma, Basal Cell - prevention & control | Female | Neoplasms, Radiation-Induced - prevention & control | Antineoplastic Agents - pharmacology | Neoplasms, Radiation-Induced - pathology | Skin Neoplasms - pathology | Mice, Hairless | Mice, Transgenic | Carcinoma, Basal Cell - pathology | Carcinoma, Squamous Cell - prevention & control | Unfolded Protein Response | Neoplasms, Radiation-Induced - metabolism | Carcinoma, Basal Cell - metabolism | Skin Neoplasms - metabolism | Carcinoma, Squamous Cell - etiology | Animals | Cell Line, Tumor | Skin Neoplasms - etiology | Squamous cell carcinoma | Naproxen | Analysis | Skin cancer | Vimentin | Animal models | Mesenchyme | Cyclin D1 | Carcinogenesis | E-cadherin | Anticancer properties | Carcinogens | N-Cadherin | Protein folding | Proliferating cell nuclear antigen | Inhibition | Invasiveness | Inflammation | Hairless | Ablation | Nitric-oxide synthase | Ultraviolet radiation | Skin | Cyclooxygenase-2 | Tumors | Cancer | Apoptosis | Index Medicus | UVB | skin | photocarcinogenesis | inflammation | UPR signaling
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Loading Rights1986, ISBN 9780879691752, ix, 332
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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2004, Volume 101, Issue 36, pp. 13363 - 13367
Neurogenesis continues in the adult brain and is increased in certain pathological states. We reported recently that neurogenesis is enhanced in hippocampus of...
Brain | Biological Sciences | Animal models | Nervous system diseases | Disease models | Neurons | Transgenic animals | Antibodies | Mice | Neurogenesis | Hippocampus | IN-VITRO | AMYLOID BETA-PEPTIDE | MULTIDISCIPLINARY SCIENCES | FOCAL CEREBRAL-ISCHEMIA | RECEPTOR ACTIVATION | CELL-PROLIFERATION | ADULT MICE | SUBVENTRICULAR ZONE | DENTATE GYRUS | HIPPOCAMPAL NEUROGENESIS | MOUSE MODELS | Alzheimer Disease - physiopathology | Amyloid beta-Protein Precursor - genetics | Animals | Mice, Inbred C57BL | Neurons - physiology | Mice, Transgenic | Bromodeoxyuridine - metabolism | Platelet-Derived Growth Factor - genetics | Alzheimer's disease | Research | Proteins | Neurology | Brain research | Mutation | Alzheimers disease | Rodents | Index Medicus
Brain | Biological Sciences | Animal models | Nervous system diseases | Disease models | Neurons | Transgenic animals | Antibodies | Mice | Neurogenesis | Hippocampus | IN-VITRO | AMYLOID BETA-PEPTIDE | MULTIDISCIPLINARY SCIENCES | FOCAL CEREBRAL-ISCHEMIA | RECEPTOR ACTIVATION | CELL-PROLIFERATION | ADULT MICE | SUBVENTRICULAR ZONE | DENTATE GYRUS | HIPPOCAMPAL NEUROGENESIS | MOUSE MODELS | Alzheimer Disease - physiopathology | Amyloid beta-Protein Precursor - genetics | Animals | Mice, Inbred C57BL | Neurons - physiology | Mice, Transgenic | Bromodeoxyuridine - metabolism | Platelet-Derived Growth Factor - genetics | Alzheimer's disease | Research | Proteins | Neurology | Brain research | Mutation | Alzheimers disease | Rodents | Index Medicus
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Loading RightsCirculation, ISSN 0009-7322, 06/2016, Volume 133, Issue 24, pp. 2434 - 2446
BACKGROUND—Altered composition of the gut microbiota is involved in both the onset and progression of obesity and diabetes mellitus. However, the link between...
atherosclerosis | gut microbiota | endotoxemia | CARDIAC & CARDIOVASCULAR SYSTEMS | INTESTINAL MICROBIOTA | HUMAN GUT MICROBIOTA | FECAL MICROBIOTA | DIET-INDUCED OBESITY | BODY-WEIGHT | INSULIN-RESISTANCE | APOE-DEFICIENT MICE | CARDIOVASCULAR-DISEASE | FAT | PERIPHERAL VASCULAR DISEASE | HEALTH | Caco-2 Cells | Atherosclerosis - pathology | Inflammation - microbiology | Endotoxemia - prevention & control | Humans | Male | Mice, Transgenic | Treatment Outcome | Endotoxemia - etiology | Inflammation - etiology | Verrucomicrobia - physiology | Animals | Intestines - microbiology | Atherosclerosis - microbiology | Inflammation - prevention & control | Mice | Atherosclerosis - prevention & control | Disease Models, Animal | Endotoxemia - microbiology | Obesity | Complications and side effects | Care and treatment | Cardiovascular diseases | Atherosclerosis | Index Medicus | Abridged Index Medicus
atherosclerosis | gut microbiota | endotoxemia | CARDIAC & CARDIOVASCULAR SYSTEMS | INTESTINAL MICROBIOTA | HUMAN GUT MICROBIOTA | FECAL MICROBIOTA | DIET-INDUCED OBESITY | BODY-WEIGHT | INSULIN-RESISTANCE | APOE-DEFICIENT MICE | CARDIOVASCULAR-DISEASE | FAT | PERIPHERAL VASCULAR DISEASE | HEALTH | Caco-2 Cells | Atherosclerosis - pathology | Inflammation - microbiology | Endotoxemia - prevention & control | Humans | Male | Mice, Transgenic | Treatment Outcome | Endotoxemia - etiology | Inflammation - etiology | Verrucomicrobia - physiology | Animals | Intestines - microbiology | Atherosclerosis - microbiology | Inflammation - prevention & control | Mice | Atherosclerosis - prevention & control | Disease Models, Animal | Endotoxemia - microbiology | Obesity | Complications and side effects | Care and treatment | Cardiovascular diseases | Atherosclerosis | Index Medicus | Abridged Index Medicus
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Loading RightsAmerican Journal of Pathology, ISSN 0002-9440, 06/2002, Volume 160, Issue 6, pp. 2295 - 2307
Most mouse models of hepatocellular carcinoma have expressed growth factors and oncogenes under the control of a liver-specific promoter. In contrast, we...
BETA-CATENIN GENE | BREAST-CANCER CELLS | PROSTATE-CANCER | TISSUE-SPECIFIC EXPRESSION | C-MYC | LIVER-TUMORS | GLUTAMINE-SYNTHETASE | FACTOR RECEPTOR-3 | PATHOLOGY | FACTOR-ALPHA | HEPATOCARCINOGENESIS | Immunohistochemistry | Receptor, Fibroblast Growth Factor, Type 4 | Humans | Fibroblast Growth Factors - genetics | Molecular Sequence Data | Muscle, Skeletal - metabolism | Fibroblast Growth Factors - biosynthesis | Wnt Proteins | DNA, Neoplasm - chemistry | Cytoskeletal Proteins - metabolism | Liver Neoplasms, Experimental - genetics | Bromodeoxyuridine - metabolism | Liver Neoplasms, Experimental - pathology | Disease Models, Animal | Proto-Oncogene Proteins - metabolism | Recombinant Proteins - metabolism | Amino Acid Sequence | Enzyme-Linked Immunosorbent Assay | beta Catenin | Liver - metabolism | Trans-Activators | Receptors, Fibroblast Growth Factor - biosynthesis | Zebrafish Proteins | Mice, Transgenic | Cytoskeletal Proteins - chemistry | Cell Division - drug effects | Animals | Mice | Mutation | Amino Acid Substitution | Index Medicus | Abridged Index Medicus | Animal Models
BETA-CATENIN GENE | BREAST-CANCER CELLS | PROSTATE-CANCER | TISSUE-SPECIFIC EXPRESSION | C-MYC | LIVER-TUMORS | GLUTAMINE-SYNTHETASE | FACTOR RECEPTOR-3 | PATHOLOGY | FACTOR-ALPHA | HEPATOCARCINOGENESIS | Immunohistochemistry | Receptor, Fibroblast Growth Factor, Type 4 | Humans | Fibroblast Growth Factors - genetics | Molecular Sequence Data | Muscle, Skeletal - metabolism | Fibroblast Growth Factors - biosynthesis | Wnt Proteins | DNA, Neoplasm - chemistry | Cytoskeletal Proteins - metabolism | Liver Neoplasms, Experimental - genetics | Bromodeoxyuridine - metabolism | Liver Neoplasms, Experimental - pathology | Disease Models, Animal | Proto-Oncogene Proteins - metabolism | Recombinant Proteins - metabolism | Amino Acid Sequence | Enzyme-Linked Immunosorbent Assay | beta Catenin | Liver - metabolism | Trans-Activators | Receptors, Fibroblast Growth Factor - biosynthesis | Zebrafish Proteins | Mice, Transgenic | Cytoskeletal Proteins - chemistry | Cell Division - drug effects | Animals | Mice | Mutation | Amino Acid Substitution | Index Medicus | Abridged Index Medicus | Animal Models
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Loading Rights2014, Methods in molecular biology, ISBN 9781493912148, Volume 1194.
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2014, Methods in molecular biology, ISBN 9781493912148, Volume 1194.
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Neurobiology of Aging, ISSN 0197-4580, 2015, Volume 36, Issue 1, pp. 111 - 122
Abstract Anti-amyloid-β (Aβ) immunotherapy is a potential therapeutic strategy to reduce amyloid plaques and amyloid-associated pathologies in Alzheimer's...
Neurology | Internal Medicine | Immune senescence | Alzheimer's disease | Immunotherapy | Bone marrow derived dendritic cell | Immunotherapy - methods | Bone Marrow Cells - cytology | Alzheimer Disease - therapy | Injections, Intraperitoneal | Mice, Transgenic | Spleen - cytology | Brain - metabolism | Aging - immunology | Animals | Microglia - immunology | Alzheimer Disease - prevention & control | Bone Marrow Cells - immunology | Peptide Fragments - immunology | Amyloid beta-Peptides - immunology | Mice | Alzheimer Disease - immunology | Dendritic Cells - transplantation | Alzheimer Disease - psychology | Phagocytosis | Plaque, Amyloid - immunology | Spleen - transplantation | Index Medicus
Neurology | Internal Medicine | Immune senescence | Alzheimer's disease | Immunotherapy | Bone marrow derived dendritic cell | Immunotherapy - methods | Bone Marrow Cells - cytology | Alzheimer Disease - therapy | Injections, Intraperitoneal | Mice, Transgenic | Spleen - cytology | Brain - metabolism | Aging - immunology | Animals | Microglia - immunology | Alzheimer Disease - prevention & control | Bone Marrow Cells - immunology | Peptide Fragments - immunology | Amyloid beta-Peptides - immunology | Mice | Alzheimer Disease - immunology | Dendritic Cells - transplantation | Alzheimer Disease - psychology | Phagocytosis | Plaque, Amyloid - immunology | Spleen - transplantation | Index Medicus
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Loading RightsScience, ISSN 0036-8075, 6/2001, Volume 292, Issue 5522, pp. 1728 - 1731
Glucose homeostasis depends on insulin responsiveness in target tissues, most importantly, muscle and liver. The critical initial steps in insulin action...
Blood glucose | Receptors | Liver | Muscles | Insulin resistance | Reports | Mice | Adipocytes | Type 2 diabetes mellitus | Insulin | T tests | 3T3-L1 ADIPOCYTES | ACTIVATION | HEPATOCYTES | INHIBITION | GLUT4 TRANSLOCATION | MULTIDISCIPLINARY SCIENCES | TRANSCRIPTION | PHOSPHOENOLPYRUVATE CARBOXYKINASE GENE | GLUCOSE-UPTAKE | EXPRESSION | C-LAMBDA | Deoxyglucose - metabolism | Protein-Serine-Threonine Kinases | Homeostasis | Male | Muscle, Skeletal - metabolism | Diabetes Mellitus, Type 2 - metabolism | Insulin - blood | Insulin Resistance - physiology | Islets of Langerhans - cytology | Female | Proto-Oncogene Proteins - metabolism | Gene Targeting | Glucose Tolerance Test | Muscle, Skeletal - enzymology | Islets of Langerhans - physiology | Signal Transduction | Liver - metabolism | Mice, Inbred C57BL | Insulin - administration & dosage | Mice, Transgenic | Proto-Oncogene Proteins - genetics | Proto-Oncogene Proteins c-akt | Insulin - metabolism | Animals | Insulin Resistance - genetics | Glucose - metabolism | Glucose Clamp Technique | Blood Glucose - metabolism | Type 2 diabetes | Research | Analysis | Proteins | Diabetes | Rodents | Genes | Protein kinase B^b | Akt2 protein | Index Medicus
Blood glucose | Receptors | Liver | Muscles | Insulin resistance | Reports | Mice | Adipocytes | Type 2 diabetes mellitus | Insulin | T tests | 3T3-L1 ADIPOCYTES | ACTIVATION | HEPATOCYTES | INHIBITION | GLUT4 TRANSLOCATION | MULTIDISCIPLINARY SCIENCES | TRANSCRIPTION | PHOSPHOENOLPYRUVATE CARBOXYKINASE GENE | GLUCOSE-UPTAKE | EXPRESSION | C-LAMBDA | Deoxyglucose - metabolism | Protein-Serine-Threonine Kinases | Homeostasis | Male | Muscle, Skeletal - metabolism | Diabetes Mellitus, Type 2 - metabolism | Insulin - blood | Insulin Resistance - physiology | Islets of Langerhans - cytology | Female | Proto-Oncogene Proteins - metabolism | Gene Targeting | Glucose Tolerance Test | Muscle, Skeletal - enzymology | Islets of Langerhans - physiology | Signal Transduction | Liver - metabolism | Mice, Inbred C57BL | Insulin - administration & dosage | Mice, Transgenic | Proto-Oncogene Proteins - genetics | Proto-Oncogene Proteins c-akt | Insulin - metabolism | Animals | Insulin Resistance - genetics | Glucose - metabolism | Glucose Clamp Technique | Blood Glucose - metabolism | Type 2 diabetes | Research | Analysis | Proteins | Diabetes | Rodents | Genes | Protein kinase B^b | Akt2 protein | Index Medicus
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