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1. Full Text Obesity and cancer - Mechanisms underlying tumour progression and recurrence
by Park, Jiyoung and Morley, Thomas S and Kim, Min and Clegg, Deborah J and Scherer, Philipp E
Nature Reviews Endocrinology, ISSN 1759-5029, 2014, Volume 10, Issue 8, pp. 455 - 465
Over the past several years, the field of cancer research has directed increased interest towards subsets of obesity-associated tumours, which include mammary,... 
BREAST-CANCER | ACTIVATED PROTEIN-KINASE | LINKING OBESITY | COLORECTAL-CANCER | ENDOCRINOLOGY & METABOLISM | IGF-BINDING PROTEIN-3 | BODY-MASS INDEX | COLONIC PRENEOPLASTIC LESIONS | LEPTIN RECEPTOR | GROWTH-FACTOR-I | ADIPOSE-TISSUE | Neoplasms - etiology | Obesity - complications | Humans | Adipose Tissue - physiopathology | Breast Neoplasms - etiology | Male | Disease Progression | Breast Neoplasms - prevention & control | Obesity - metabolism | Animals | Weight Loss | Adipocytes - metabolism | Female | Adipokines - physiology | Ovarian Neoplasms - etiology | Colonic Neoplasms - etiology | Disease Models, Animal | Inflammation - physiopathology | Obesity | Complications and side effects | Relapse | Development and progression | Research | Tumors | Cancer
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2. Full Text Effect of Selumetinib vs Chemotherapy on Progression-Free Survival in Uveal Melanoma: A Randomized Clinical Trial
by Carvajal, Richard D and Sosman, Jeffrey A and Quevedo, Jorge Fernando and Milhem, Mohammed M and Joshua, Anthony M and Kudchadkar, Ragini R and Linette, Gerald P and Gajewski, Thomas F and Lutzky, Jose and Lawson, David H and Lao, Christopher D and Flynn, Patrick J and Albertini, Mark R and Sato, Takami and Lewis, Karl and Doyle, Austin and Ancell, Kristin and Panageas, Katherine S and Bluth, Mark and Hedvat, Cyrus and Erinjeri, Joseph and Ambrosini, Grazia and Marr, Brian and Abramson, David H and Dickson, Mark Andrew and Wolchok, Jedd D and Chapman, Paul B and Schwartz, Gary K
JAMA, ISSN 0098-7484, 06/2014, Volume 311, Issue 23, pp. 2397 - 2405
IMPORTANCE Uveal melanoma is characterized by mutations in GNAQ and GNA11, resulting in mitogen-activated protein kinase pathway activation. OBJECTIVE To... 
METASTATIC-DISEASE | LUNG-CANCER | MEDICINE, GENERAL & INTERNAL | MEK INHIBITOR | MUTANT GNAQ | SOLID TUMORS | CHOROIDAL MELANOMA | COLLABORATIVE OCULAR MELANOMA | GROWTH-FACTOR | MUTATIONS | CELL-DEATH | Dacarbazine - adverse effects | Dacarbazine - therapeutic use | Humans | Middle Aged | Male | Treatment Outcome | Disease Progression | Uveal Neoplasms - drug therapy | Antineoplastic Agents, Alkylating - therapeutic use | Melanoma - drug therapy | Dacarbazine - analogs & derivatives | Survival Analysis | Aged, 80 and over | Adult | Female | Aged | Antineoplastic Agents, Alkylating - adverse effects | Benzimidazoles - adverse effects | Benzimidazoles - therapeutic use | Chemotherapy | Care and treatment | Research | Health aspects | Melanoma | Cancer | Survival analysis | Prescription drugs | Oncology | Clinical outcomes | Index Medicus | Abridged Index Medicus
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3. Full Text Epithelial—mesenchymal and mesenchymal—epithelial transitions in carcinoma progression
by Hugo, Honor and Ackland, M. Leigh and Blick, Tony and Lawrence, Mitchell G and Clements, Judith A and Williams, Elizabeth D and Thompson, Erik W
Journal of Cellular Physiology, ISSN 0021-9541, 11/2007, Volume 213, Issue 2, pp. 374 - 383
Like a set of bookends, cellular, molecular, and genetic changes of the beginnings of life mirror those of one of the most common cause of death—metastatic... 
BREAST-CANCER | NEURAL CREST DEVELOPMENT | PHYSIOLOGY | BASEMENT-MEMBRANE INVASIVENESS | COLORECTAL-CANCER | E-CADHERIN EXPRESSION | PROSTATE-CANCER | GENE-EXPRESSION | GROWTH-FACTOR RECEPTOR-2 | CANCER CELL-LINES | N-CADHERIN | CELL BIOLOGY | Biomarkers - metabolism | Prostatic Neoplasms - pathology | Epithelium - pathology | Epithelial Cells - metabolism | Neoplasm Invasiveness | Humans | Male | Prostatic Neoplasms - physiopathology | Breast Neoplasms | Carcinoma - physiopathology | Disease Progression | Cell Transformation, Neoplastic | Cell Line, Tumor | Mesoderm - physiology | Female | Epithelium - physiology | Carcinoma - pathology | Epithelial Cells - cytology | Mesoderm - pathology
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4. Full Text Microenvironmental regulation of tumor progression and metastasis
by Quail, Daniela F and Joyce, Johanna A
Nature Medicine, ISSN 1078-8956, 11/2013, Volume 19, Issue 11, pp. 1423 - 1437
Cancers develop in complex tissue environments, which they depend on for sustained growth, invasion and metastasis. Unlike tumor cells, stromal cell types... 
PROGENITOR CELLS | M1 MACROPHAGES | MEDICINE, RESEARCH & EXPERIMENTAL | CARCINOMA CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | BONE-MARROW | MYELOID CELLS | MESENCHYMAL STEM-CELLS | CELL BIOLOGY | BREAST-CANCER CELLS | SUPPRESSOR-CELLS | CHRONIC INFLAMMATION | T-CELLS | Macrophages - physiology | Inflammation - pathology | Neoplasms - etiology | Macrophages - pathology | Neoplasm Invasiveness | Stromal Cells - pathology | Humans | Neovascularization, Pathologic | Stromal Cells - immunology | Tumor Microenvironment - physiology | Disease Progression | Tumor Microenvironment - immunology | Inflammation - complications | Cell Cycle Checkpoints | Neoplasm Metastasis - immunology | Models, Biological | Neoplasm Metastasis - pathology | Immunotherapy | Neoplasm Metastasis - physiopathology | Neoplasms - physiopathology | Neoplasms - pathology | Stromal Cells - physiology | Macrophages - immunology | Inflammation - physiopathology | Oncology, Experimental | Cellular control mechanisms | Development and progression | Metastasis | Research | Carcinogenesis | Tumors | Cancer | Cell growth | Drug resistance | Cancer therapies | Medical prognosis
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5. Full Text Gefitinib plus chemotherapy versus placebo plus chemotherapy in EGFR -mutation-positive non-small-cell lung cancer after progression on first-line gefitinib (IMPRESS): a phase 3 randomised trial
by Soria, Jean-Charles, Prof and Wu, Yi-Long, Prof and Nakagawa, Kazuhiko, Prof and Kim, Sang-We, Prof and Yang, Jin-Ji, MD and Ahn, Myung-Ju, Prof and Wang, Jie, MD and Yang, James Chih-Hsin, Prof and Lu, You, Prof and Atagi, Shinji, MD and Ponce, Santiago, MD and Lee, Dae Ho, Prof and Liu, Yunpeng, Prof and Yoh, Kiyotaka, MD and Zhou, Jian-Ying, Prof and Shi, Xiaojin, MD and Webster, Alan, MSc and Jiang, Haiyi, MD and Mok, Tony S K, Prof
Lancet Oncology, The, ISSN 1470-2045, 2015, Volume 16, Issue 8, pp. 990 - 998
Summary Background Optimum management strategies for patients with advanced non-small-cell lung cancer (NSCLC) with acquired resistance to EGFR tyrosine-kinase... 
Hematology, Oncology and Palliative Medicine | TYROSINE KINASE INHIBITORS | MULTICENTER | ONCOLOGY | ADENOCARCINOMA | ACQUIRED-RESISTANCE | OPEN-LABEL | AFATINIB | ERLOTINIB | GEMCITABINE | Lung Neoplasms - drug therapy | Receptor, Epidermal Growth Factor - genetics | Prospective Studies | Guanine - analogs & derivatives | Lung Neoplasms - mortality | Humans | Middle Aged | Antineoplastic Combined Chemotherapy Protocols - adverse effects | Lung Neoplasms - pathology | Male | Cisplatin - administration & dosage | Receptor, Epidermal Growth Factor - metabolism | Time Factors | Adult | Female | Quinazolines - administration & dosage | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Genetic Predisposition to Disease | Lung Neoplasms - enzymology | Drug Administration Schedule | Administration, Oral | Carcinoma, Non-Small-Cell Lung - genetics | Europe | Kaplan-Meier Estimate | Proportional Hazards Models | Treatment Outcome | Carcinoma, Non-Small-Cell Lung - mortality | Pemetrexed | Disease Progression | Disease-Free Survival | Protein Kinase Inhibitors - administration & dosage | Phenotype | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Asia | Glutamates - administration & dosage | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Aged | Biomarkers, Tumor - genetics | Carcinoma, Non-Small-Cell Lung - drug therapy | Carcinoma, Non-Small-Cell Lung - enzymology | Infusions, Intravenous | Mutation | Guanine - administration & dosage | Medical colleges | Chemotherapy | Strategic planning (Business) | Genetic aspects | Product development | Lung cancer, Non-small cell | Gefitinib | Cancer
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6. Full Text BM mesenchymal stromal cell-derived exosomes facilitate multiple myeloma progression
by Roccaro, Aldo M and Sacco, Antonio and Maiso, Patricia and Azab, Abdel Kareem and Tai, Yu-Tzu and Reagan, Michaela and Azab, Feda and Flores, Ludmila M and Campigotto, Federico and Weller, Edie and Anderson, Kenneth C and Scadden, David T and Ghobrial, Irene M
Journal of Clinical Investigation, ISSN 0021-9738, 04/2013, Volume 123, Issue 4, pp. 1542 - 1555
BM mesenchymal stromal cells (BM-MSCs) support multiple myeloma (MM) cell growth, but little is known about the putative mechanisms by which the BM... 
PROMOTE TUMOR-GROWTH | MEDICINE, RESEARCH & EXPERIMENTAL | IN-VITRO | ACTIVATION | MACROPHAGES | MICRORNAS | ACCUMULATION | SECRETION | PROTEOMIC ANALYSIS | RECEPTORS | EXPRESSION | Exosomes - metabolism | Neoplasm Transplantation | Cell Proliferation | Femur - pathology | Humans | Middle Aged | MicroRNAs - metabolism | Tissue Scaffolds | Exosomes - physiology | Female | Tumor Cells, Cultured | Mesenchymal Stromal Cells - physiology | Mice, Inbred C57BL | Mesenchymal Stromal Cells - metabolism | Tumor Burden | Mice, SCID | Multiple Myeloma - metabolism | Bone Marrow Neoplasms - secondary | Disease Progression | Culture Media, Conditioned | Multiple Myeloma - pathology | Animals | Bone Marrow Neoplasms - metabolism | Aged | Mice | MicroRNAs - genetics | Mesenchymal Stem Cell Transplantation | Cell Movement | Epigenetic inheritance | Cell interaction | Multiple myeloma | Stem cells | Development and progression | Genetic aspects | Research
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7. Full Text Autophagy in malignant transformation and cancer progression
by Galluzzi, Lorenzo and Pietrocola, Federico and Bravo‐San Pedro, José Manuel and Amaravadi, Ravi K and Baehrecke, Eric H and Cecconi, Francesco and Codogno, Patrice and Debnath, Jayanta and Gewirtz, David A and Karantza, Vassiliki and Kimmelman, Alec and Kumar, Sharad and Levine, Beth and Maiuri, Maria Chiara and Martin, Seamus J and Penninger, Josef and Piacentini, Mauro and Rubinsztein, David C and Simon, Hans‐Uwe and Simonsen, Anne and Thorburn, Andrew M and Velasco, Guillermo and Ryan, Kevin M and Kroemer, Guido
The EMBO Journal, ISSN 0261-4189, 04/2015, Volume 34, Issue 7, pp. 856 - 880
Autophagy plays a key role in the maintenance of cellular homeostasis. In healthy cells, such a homeostatic activity constitutes a robust barrier against... 
mitophagy | KRAS | adaptive stress responses | inflammation | Beclin 1 | Adaptive stress responses | BCL-X-L | ADVANCED SOLID TUMORS | BIOCHEMISTRY & MOLECULAR BIOLOGY | REGULATES AUTOPHAGY | TUMOR-SUPPRESSOR GENE | DOWN-REGULATION | CELL-DEATH | CELL BIOLOGY | BREAST-CANCER | PHASE-I TRIAL | STRESS-INDUCED AUTOPHAGY | Neoplasms - metabolism | Tumor Escape | Tumor Suppressor Proteins - metabolism | Humans | Autophagy | Cell Transformation, Neoplastic - metabolism | Tumor Suppressor Proteins - immunology | Cell Transformation, Neoplastic - immunology | Neoplasms - therapy | Animals | Neoplasms - immunology | Cell Transformation, Neoplastic - pathology | Neoplasms - pathology | Cellular biology | Cancer therapies | Index Medicus | Reviews
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