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The Journal of Pathology, ISSN 0022-3417, 05/2017, Volume 242, Issue 1, pp. 113 - 125
Journal Article
Oncogene, ISSN 0950-9232, 11/2012, Volume 31, Issue 47, pp. 4898 - 4911
There is enormous interest to target cancer stem cells (CSCs) for clinical treatment because these cells are highly tumorigenic and resistant to chemotherapy.... 
hypoxia | Oct4 | dedifferentiation | melanoma | cancer stem cell | INITIATING CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | PLURIPOTENCY | CELL BIOLOGY | SUBPOPULATION | ONCOLOGY | GENETICS & HEREDITY | GENE-EXPRESSION | GENERATION | MELANOMA-CELLS | DIFFERENTIATION | PROMOTES | Octamer Transcription Factor-3 - physiology | Neoplasm Transplantation | Cell Proliferation | Homeodomain Proteins - metabolism | Humans | Gene Expression Regulation, Neoplastic | Transcriptome | Cell Dedifferentiation | Cell Hypoxia | Octamer Transcription Factor-3 - genetics | Neoplastic Stem Cells - metabolism | Kruppel-Like Transcription Factors - metabolism | Cell Shape | Genes, Reporter | Neoplastic Stem Cells - physiology | Melanoma - metabolism | Promoter Regions, Genetic | Nanog Homeobox Protein | Cell Survival | Spheroids, Cellular - metabolism | Melanoma - pathology | Mice, SCID | Homeodomain Proteins - genetics | Phenotype | Animals | Octamer Transcription Factor-3 - metabolism | Cell Line, Tumor | Mice, Inbred NOD | Mice | Kruppel-Like Transcription Factors - genetics | Cell Movement | Prognosis | Cancer cells | Melanoma | Physiological aspects | Development and progression | Research | Gene expression | Risk factors | Proteins | Genotype & phenotype | Stem cells | Hypoxia | Skin cancer | Tumors | Index Medicus | Transcription factors | Tumor cells | Oct-4 protein | Data processing | Chemotherapy | KLF4 protein | Veins | Xenografts | Differentiation | spheroids | Injuries | Cancer | Cancer stem cell | Dedifferentiation
Journal Article
Journal Article
Journal Article
Journal Article
Cancer Letters, ISSN 0304-3835, 2011, Volume 307, Issue 1, pp. 26 - 36
Abstract Activation of Notch-1 is known to be associated with the development and progression of human malignancies including pancreatic cancer. Emerging... 
Hematology, Oncology and Palliative Medicine | CSC-self renewal | MiRNAs | EMT phenotype | Notch-1 | Genistein | miRNAs | TARGETING NOTCH | DOWN-REGULATION | DRUG-RESISTANCE | BREAST-CANCER | MIR-200 | INHIBITION | ONCOLOGY | SIGNALING PATHWAY | PROSTATE-CANCER | DIFFERENTIATION | EXPRESSION | Cell Adhesion Molecules - genetics | Pancreatic Neoplasms - metabolism | Neoplastic Stem Cells - drug effects | Humans | Wound Healing | Epithelial Cell Adhesion Molecule | Neoplastic Stem Cells - metabolism | Epithelial-Mesenchymal Transition | Hyaluronan Receptors - metabolism | Antigens, Neoplasm - metabolism | Neoplastic Stem Cells - pathology | Tumor Cells, Cultured | Spheroids, Cellular | Anticarcinogenic Agents - pharmacology | Antigens, Neoplasm - genetics | Pancreatic Neoplasms - pathology | RNA, Messenger - genetics | Pancreatic Neoplasms - genetics | Receptor, Notch1 - metabolism | Cell Adhesion - drug effects | Reverse Transcriptase Polymerase Chain Reaction | Cell Adhesion Molecules - metabolism | Hyaluronan Receptors - genetics | Blotting, Western | Cell Movement - drug effects | Genistein - pharmacology | Cell Proliferation - drug effects | MicroRNAs - physiology | Receptor, Notch1 - antagonists & inhibitors | Receptor, Notch1 - genetics | Care and treatment | Pancreatic cancer | Stem cells | Development and progression | Genetic aspects | Drug resistance | Intermediate filament proteins | Cancer | Isoflavones | Metastasis | Cancer therapies | Cell adhesion & migration | Studies | Genotype & phenotype | Cell growth | Medical prognosis | Technological change | Ligands | Pancreas | Acquisitions & mergers | Tumors | Index Medicus | genistein
Journal Article
Journal Article
Nature Cell Biology, ISSN 1465-7392, 08/2015, Volume 17, Issue 8, pp. 1049 - 1061
The TOR (target of rapamycin) kinase limits longevity by poorly understood mechanisms. Rapamycin suppresses the mammalian TORC1 complex, which regulates... 
EPITHELIAL-CELL LINE | GROWTH ARREST | DNA-DAMAGE | IN-VIVO | INFLAMMATORY CYTOKINE SECRETION | HUMAN FIBROBLASTS | TUMOR-CELLS | CANCER-THERAPY | REPLICATIVE SENESCENCE | P53 | CELL BIOLOGY | Up-Regulation | Fibroblasts - enzymology | Cell Proliferation | TOR Serine-Threonine Kinases - metabolism | Humans | Gene Expression Regulation, Neoplastic | Interleukin-1alpha - metabolism | Male | NF-kappa B - metabolism | RNA, Messenger - metabolism | Prostatic Neoplasms - secretion | Prostatic Neoplasms - immunology | Dose-Response Relationship, Drug | TOR Serine-Threonine Kinases - antagonists & inhibitors | Prostatic Neoplasms - genetics | TOR Serine-Threonine Kinases - genetics | Transfection | RNA Interference | Time Factors | Cellular Senescence | Inflammation Mediators - metabolism | Transcription, Genetic | Antineoplastic Agents - pharmacology | Interleukin-1alpha - genetics | Interleukin-6 - metabolism | Prostatic Neoplasms - drug therapy | Prostatic Neoplasms - pathology | Anti-Inflammatory Agents - pharmacology | Interleukin-1alpha - secretion | Sirolimus - metabolism | Tumor Burden | Mice, SCID | Sirolimus - pharmacology | Xenograft Model Antitumor Assays | Phenotype | Animals | Fibroblasts - drug effects | Cell Line, Tumor | Prostatic Neoplasms - enzymology | Mitoxantrone - pharmacology | Interleukins | Aging | Genetic aspects | Properties | Phosphotransferases | Carcinogenesis | Health aspects | Cells | Index Medicus
Journal Article
Journal of Cellular Biochemistry, ISSN 0730-2312, 09/2011, Volume 112, Issue 9, pp. 2296 - 2306
Journal Article